| review article | Q7318358 |
| scholarly article | Q13442814 |
| P6179 | Dimensions Publication ID | 1006159884 |
| P356 | DOI | 10.1186/GB-2004-5-10-244 |
| P932 | PMC publication ID | 545589 |
| P698 | PubMed publication ID | 15461808 |
| P5875 | ResearchGate publication ID | 8253354 |
| P2093 | author name string | Katheleen Gardiner | |
| P2860 | cites work | Global up-regulation of chromosome 21 gene expression in the developing down syndrome brain | Q28198293 |
| Genetic analysis of genome-wide variation in human gene expression | Q29617852 | ||
| Analysis of gene expression in the rat hippocampus using Real Time PCR reveals high inter-individual variation in mRNA expression levels | Q30794274 | ||
| Down's syndrome: a genetic disorder in biobehavioral perspective | Q35204303 | ||
| Down syndrome phenotypes: the consequences of chromosomal imbalance | Q35344729 | ||
| Transcript level alterations reflect gene dosage effects across multiple tissues in a mouse model of down syndrome | Q35574036 | ||
| Gene expression from the aneuploid chromosome in a trisomy mouse model of down syndrome | Q35574050 | ||
| Ts1Cje, a partial trisomy 16 mouse model for Down syndrome, exhibits learning and behavioral abnormalities | Q37394402 | ||
| The neuropsychology of Down syndrome: evidence for hippocampal dysfunction | Q38520897 | ||
| A high proportion of chromosome 21 promoter polymorphisms influence transcriptional activity | Q40544761 | ||
| Segmental trisomy as a mouse model for Down syndrome | Q40718246 | ||
| Mouse models of Down syndrome: how useful can they be? Comparison of the gene content of human chromosome 21 with orthologous mouse genomic regions | Q47415659 | ||
| Dosage-dependent over-expression of genes in the trisomic region of Ts1Cje mouse model for Down syndrome. | Q51940838 | ||
| Allelic Variation in Human Gene Expression | Q57293497 | ||
| Evolutionary breakpoints on human chromosome 21 | Q77165559 | ||
| Natural variation in human gene expression assessed in lymphoblastoid cells | Q78885024 | ||
| P433 | issue | 10 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | Down syndrome | Q47715 |
| genetic variation | Q349856 | ||
| gene dosage | Q1465102 | ||
| animal disease model | Q64732998 | ||
| biomedical investigative technique | Q66648976 | ||
| P304 | page(s) | 244 | |
| P577 | publication date | 2004-09-30 | |
| P1433 | published in | Genome Biology | Q5533480 |
| P1476 | title | Gene-dosage effects in Down syndrome and trisomic mouse models | |
| P478 | volume | 5 |
| Q34229190 | Abnormal microRNA expression in Ts65Dn hippocampus and whole blood: contributions to Down syndrome phenotypes |
| Q33293466 | Altered expression of mitochondrial and extracellular matrix genes in the heart of human fetuses with chromosome 21 trisomy |
| Q37543845 | Aneuploidy: from a physiological mechanism of variance to Down syndrome |
| Q33236637 | Cell type-specific over-expression of chromosome 21 genes in fibroblasts and fetal hearts with trisomy 21. |
| Q27300997 | Down Syndrome Cognitive Phenotypes Modeled in Mice Trisomic for All HSA 21 Homologues |
| Q38715222 | Expression of endogenous mouse APP modulates β-amyloid deposition in hAPP-transgenic mice |
| Q37775217 | GABAB-GIRK2-mediated signaling in Down syndrome. |
| Q34674349 | Genome-wide miR-155 and miR-802 target gene identification in the hippocampus of Ts65Dn Down syndrome mouse model by miRNA sponges |
| Q33324833 | Identification of dysregulated genes in lymphocytes from children with Down syndrome. |
| Q42067838 | Impairment of circulating endothelial progenitors in Down syndrome |
| Q92236496 | Maternal Choline Supplementation Alters Basal Forebrain Cholinergic Neuron Gene Expression in the Ts65Dn Mouse Model of Down Syndrome |
| Q36371958 | Overexpression of amyloid-β protein precursor induces mitochondrial oxidative stress and activates the intrinsic apoptotic cascade |
| Q30367032 | Redox proteomics analysis to decipher the neurobiology of Alzheimer-like neurodegeneration: overlaps in Down's syndrome and Alzheimer's disease brain. |
| Q40880509 | The GIRK2 subunit is involved in IS-like seizures induced by GABA(B) receptor agonists. |
| Q36525183 | The cognitive phenotype of Down syndrome: insights from intracellular network analysis |
| Q38568871 | What can we learn from study of Alzheimer's disease in patients with Down syndrome for early-onset Alzheimer's disease in the general population? |
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