scholarly article | Q13442814 |
P6179 | Dimensions Publication ID | 1046937871 |
P356 | DOI | 10.1038/SJ.CDD.4402014 |
P2880 | NIOSHTIC-2 ID | 20031102 |
P698 | PubMed publication ID | 16858426 |
P5875 | ResearchGate publication ID | 6928382 |
P50 | author | Vincent Castranova | Q24747188 |
P2093 | author name string | F Chen | |
Q Chang | |||
D Bhatia | |||
P2860 | cites work | Signal-induced ubiquitination of IkappaBalpha by the F-box protein Slimb/beta-TrCP | Q21666110 |
Endogenous inhibitors of nuclear factor-kB, an opportunity for cancer control | Q23912838 | ||
TAK1 is a ubiquitin-dependent kinase of MKK and IKK | Q24291454 | ||
Sequential modification of NEMO/IKKgamma by SUMO-1 and ubiquitin mediates NF-kappaB activation by genotoxic stress | Q24300945 | ||
Bcl10 activates the NF-kappaB pathway through ubiquitination of NEMO | Q24303248 | ||
A corepressor/coactivator exchange complex required for transcriptional activation by nuclear receptors and other regulated transcription factors | Q28246411 | ||
Site-specific phosphorylation of IkappaBalpha by a novel ubiquitination-dependent protein kinase activity | Q28276063 | ||
Oscillations in NF-kappaB signaling control the dynamics of gene expression | Q28289121 | ||
Signal-induced site-specific phosphorylation targets I kappa B alpha to the ubiquitin-proteasome pathway | Q28615706 | ||
Proteasomal regulation of nuclear receptor corepressor-mediated repression | Q35202493 | ||
Molecular linkage between the kinase ATM and NF-kappaB signaling in response to genotoxic stimuli | Q40312088 | ||
IKKα limits macrophage NF-κB activation and contributes to the resolution of inflammation | Q59054264 | ||
P433 | issue | 11 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | cell biology | Q7141 |
gene | Q7187 | ||
protein | Q8054 | ||
carcinogenesis | Q1637543 | ||
P5008 | on focus list of Wikimedia project | Wikimedia–NIOSH collaboration | Q104416361 |
P304 | page(s) | 1835-1838 | |
P577 | publication date | 2006-07-21 | |
P1433 | published in | Cell Death & Differentiation | Q2943974 |
P859 | sponsor | NIOSH Health Effects Laboratory Division | Q123344455 |
P1476 | title | Finding NEMO by K63-linked polyubiquitin chain | |
P478 | volume | 13 |
Q37107525 | Genetic susceptibility to lupus: the biological basis of genetic risk found in B cell signaling pathways. |
Q41945976 | Human parainfluenza virus type 2 V protein inhibits TRAF6-mediated ubiquitination of IRF7 to prevent TLR7- and TLR9-dependent interferon induction |
Q26860007 | Interferon induction by RNA viruses and antagonism by viral pathogens |
Q24315943 | Involvement of linear polyubiquitylation of NEMO in NF-kappaB activation |
Q39177871 | Linear polyubiquitin chains: a new modifier involved in NFκB activation and chronic inflammation, including dermatitis. |
Q37526370 | Linear polyubiquitination: a new regulator of NF-kappaB activation. |
Q37453504 | Pathogen recognition in the innate immune response. |
Q34115691 | Polyubiquitin conjugation to NEMO by triparite motif protein 23 (TRIM23) is critical in antiviral defense |
Q39627012 | Regulation of IκB kinase by GβL through recruitment of the protein phosphatases |
Q27012739 | Roles for the ubiquitin-proteasome pathway in protein quality control and signaling in the retina: implications in the pathogenesis of age-related macular degeneration |
Q26864035 | Roles of NOD1 (NLRC1) and NOD2 (NLRC2) in innate immunity and inflammatory diseases |
Q27674024 | Specific recognition of linear polyubiquitin by A20 zinc finger 7 is involved in NF-κB regulation |
Q37951705 | Subversion of innate immune responses by bacterial hindrance of NF-κB pathway |
Q37208221 | Sustained oxidative stress inhibits NF-kappaB activation partially via inactivating the proteasome |
Q36749651 | TWEAK-FN14 signaling induces lysosomal degradation of a cIAP1-TRAF2 complex to sensitize tumor cells to TNFalpha. |
Q37295365 | The zinc finger domain of IKKγ (NEMO) protein in health and disease |
Q34548586 | Toll-like receptors in Alzheimer's disease |
Q23912835 | Tumor promoting or tumor suppressing of NF-kB, a matter of cell context dependency |
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