scholarly article | Q13442814 |
P50 | author | Yon Rojanasakul | Q28036799 |
Sudjit Luanpitpong | Q28050448 | ||
Christian Stehlik | Q28050449 | ||
Neelam Azad | Q28050473 | ||
Liying Wang | Q66414148 | ||
Anand Krishnan V Iyer | Q89472677 | ||
Siera Jo Talbott | Q114455442 | ||
P2860 | cites work | Nitric oxide negatively regulates Fas CD95-induced apoptosis through inhibition of ubiquitin-proteasome-mediated degradation of FLICE inhibitory protein | Q23919035 |
Inhibition of death receptor signals by cellular FLIP | Q24316446 | ||
FLICE-inhibitory proteins: regulators of death receptor-mediated apoptosis | Q24548247 | ||
Increased expression of death receptors 4 and 5 synergizes the apoptosis response to combined treatment with etoposide and TRAIL | Q24554360 | ||
c-IAP1 and c-IAP2 are critical mediators of tumor necrosis factor alpha (TNFalpha)-induced NF-kappaB activation | Q24607621 | ||
Activation of the NF-kappaB pathway by caspase 8 and its homologs | Q28139456 | ||
The caspase-8 inhibitor FLIP promotes activation of NF-kappaB and Erk signaling pathways | Q28145665 | ||
Cellular FLICE-inhibitory protein splice variants inhibit different steps of caspase-8 activation at the CD95 death-inducing signaling complex | Q28209029 | ||
The role of c-FLIP in modulation of CD95-induced apoptosis | Q28293167 | ||
Modulation of life and death by the TNF receptor superfamily | Q28295043 | ||
N-terminal fragment of c-FLIP(L) processed by caspase 8 specifically interacts with TRAF2 and induces activation of the NF-kappaB signaling pathway | Q28629323 | ||
The death domain kinase RIP mediates the TNF-induced NF-kappaB signal | Q29617727 | ||
Cellular IAPs inhibit a cryptic CD95-induced cell death by limiting RIP1 kinase recruitment | Q33589801 | ||
Death receptors couple to both cell proliferation and apoptosis | Q34537833 | ||
The Human Herpes Virus 8-Encoded Viral FLICE-inhibitory Protein Induces Cellular Transformation via NF-κB Activation | Q34542601 | ||
Regulation of lymphocyte proliferation and death by FLIP. | Q34572136 | ||
S-nitrosylation: physiological regulation of NF-kappaB | Q34827002 | ||
4-(4-Chloro-2-methylphenoxy)-N-hydroxybutanamide (CMH) targets mRNA of the c-FLIP variants and induces apoptosis in MCF-7 human breast cancer cells | Q35668904 | ||
Targeting the FLICE Inhibitory Protein (FLIP) in cancer therapy. | Q35679530 | ||
Death receptors in chemotherapy and cancer | Q35743149 | ||
Association of TRAF2 with the short form of cellular FLICE-like inhibitory protein prevents TNFR1-mediated apoptosis | Q36485042 | ||
Regulation and specificity of S-nitrosylation and denitrosylation. | Q36683159 | ||
Regulation of TNFR1 and CD95 signalling by receptor compartmentalization | Q37188507 | ||
TRAIL and other TRAIL receptor agonists as novel cancer therapeutics | Q37598582 | ||
NF-kappaB inducers upregulate cFLIP, a cycloheximide-sensitive inhibitor of death receptor signaling | Q39459534 | ||
Mitotic arrest and JNK-induced proteasomal degradation of FLIP and Mcl-1 are key events in the sensitization of breast tumor cells to TRAIL by antimicrotubule agents | Q39770080 | ||
PKC-mediated phosphorylation regulates c-FLIP ubiquitylation and stability | Q39864593 | ||
The receptor interacting protein 1 inhibits p53 induction through NF-kappaB activation and confers a worse prognosis in glioblastoma | Q39865435 | ||
Combined inhibition of FLIP and XIAP induces Bax-independent apoptosis in type II colorectal cancer cells | Q39935218 | ||
Dynamics within the CD95 death-inducing signaling complex decide life and death of cells | Q40150581 | ||
Cellular FLIP (long form) regulates CD8+ T cell activation through caspase-8-dependent NF-kappa B activation | Q40432682 | ||
Selective knockdown of the long variant of cellular FLICE inhibitory protein augments death receptor-mediated caspase-8 activation and apoptosis. | Q40448034 | ||
Alternative splicing variants of c-FLIP transduce the differential signal through the Raf or TRAF2 in TNF-induced cell proliferation | Q40764047 | ||
Inhibition of NF-kappa B by S-nitrosylation | Q40809466 | ||
Signaling molecules of the NF-kappa B pathway shuttle constitutively between cytoplasm and nucleus. | Q42516012 | ||
The c-FLIP-NH2 terminus (p22-FLIP) induces NF-kappaB activation. | Q42772785 | ||
Bile acids stimulate cFLIP phosphorylation enhancing TRAIL-mediated apoptosis | Q44198653 | ||
Short hairpin RNA targeting c-FLIP sensitizes human cervical adenocarcinoma Hela cells to chemotherapy and radiotherapy | Q45178528 | ||
The distinct roles of TRAF2 and RIP in IKK activation by TNF-R1: TRAF2 recruits IKK to TNF-R1 while RIP mediates IKK activation | Q73745042 | ||
P6195 | funding scheme | extramural research | Q124503498 |
P433 | issue | 12 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | cell biology | Q7141 |
nitric acid | Q83320 | ||
protein | Q8054 | ||
NF-κB | Q411114 | ||
P5008 | on focus list of Wikimedia project | Wikimedia–NIOSH collaboration | Q104416361 |
P304 | page(s) | 1948-57 | |
P577 | publication date | 2014-01-01 | |
P1433 | published in | Cell Cycle | Q1254166 |
P859 | sponsor | NIOSH Health Effects Laboratory Division | Q123344455 |
P1476 | title | S-nitrosylation of FLICE inhibitory protein determines its interaction with RIP1 and activation of NF-kappa B | |
P478 | volume | 13 |
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Q26786132 | S-Nitrosylation in TNF superfamily signaling pathway: Implication in cancer |
Q60584712 | The Emerging Role of Protein S-Nitrosylation in Cancer Metastasis |
Q53067128 | [NO and cancer: itinerary of a double agent]. |
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