scholarly article | Q13442814 |
review article | Q7318358 |
P50 | author | Peter Wenner | Q56027758 |
P2093 | author name string | Peter Wenner | |
P2860 | cites work | Two developmental switches in GABAergic signalling: the K+-Cl- cotransporter KCC2 and carbonic anhydrase CAVII | Q24676766 |
GABA: a pioneer transmitter that excites immature neurons and generates primitive oscillations | Q28252805 | ||
Activity-dependent scaling of quantal amplitude in neocortical neurons | Q28264163 | ||
Reduction of KCC2 expression and GABAA receptor-mediated excitation after in vivo axonal injury | Q28571012 | ||
BDNF from microglia causes the shift in neuronal anion gradient underlying neuropathic pain | Q29616106 | ||
Activity-dependent scaling of GABAergic synapse strength is regulated by brain-derived neurotrophic factor | Q30436721 | ||
Narp regulates homeostatic scaling of excitatory synapses on parvalbumin-expressing interneurons | Q30496836 | ||
Homeostatic plasticity studied using in vivo hippocampal activity-blockade: synaptic scaling, intrinsic plasticity and age-dependence | Q33293516 | ||
Reduction in endocannabinoid tone is a homeostatic mechanism for specific inhibitory synapses | Q33814760 | ||
Excitation-transcription coupling via calcium/calmodulin-dependent protein kinase/ERK1/2 signaling mediates the coordinate induction of VGLUT2 and Narp triggered by a prolonged increase in glutamatergic synaptic activity | Q33824919 | ||
Chloride regulation in the pain pathway | Q33991375 | ||
Critical periods for experience-dependent synaptic scaling in visual cortex | Q34134898 | ||
GABAergic synaptic scaling in embryonic motoneurons is mediated by a shift in the chloride reversal potential. | Q34175358 | ||
Trans-synaptic shift in anion gradient in spinal lamina I neurons as a mechanism of neuropathic pain | Q34224419 | ||
Mechanism of activity-dependent downregulation of the neuron-specific K-Cl cotransporter KCC2. | Q34320496 | ||
Spontaneous network activity in the embryonic spinal cord regulates AMPAergic and GABAergic synaptic strength | Q34494212 | ||
Cation-chloride cotransporters and neuronal function | Q34969477 | ||
BDNF regulates the intrinsic excitability of cortical neurons | Q35039394 | ||
Using the structure of inhibitory networks to unravel mechanisms of spatiotemporal patterning | Q35148250 | ||
Brain injury impairs dentate gyrus inhibitory efficacy | Q35242333 | ||
Aspects of the homeostaic plasticity of GABAA receptor-mediated inhibition | Q35940390 | ||
Febrile seizures in the developing brain result in persistent modification of neuronal excitability in limbic circuits | Q36057730 | ||
Homeostatic control of neural activity: from phenomenology to molecular design | Q36507590 | ||
Activity differentially regulates the surface expression of synaptic AMPA and NMDA glutamate receptors | Q36509364 | ||
Altered chloride homeostasis in neurological disorders: a new target | Q36689467 | ||
Sensing and expressing homeostatic synaptic plasticity. | Q36724238 | ||
GABAA transmission is a critical step in the process of triggering homeostatic increases in quantal amplitude | Q36837523 | ||
Epilepsy, E/I Balance and GABA(A) Receptor Plasticity. | Q36852883 | ||
Differential activity-dependent, homeostatic plasticity of two neocortical inhibitory circuits | Q36956872 | ||
The age of plasticity: developmental regulation of synaptic plasticity in neocortical microcircuits | Q37131136 | ||
NKCC1 and AE3 appear to accumulate chloride in embryonic motoneurons. | Q37131831 | ||
Compensatory changes in cellular excitability, not synaptic scaling, contribute to homeostatic recovery of embryonic network activity | Q37167640 | ||
Unraveling mechanisms of homeostatic synaptic plasticity | Q37752834 | ||
Too many cooks? Intrinsic and synaptic homeostatic mechanisms in cortical circuit refinement | Q37857840 | ||
Temporal structure in spatially organized neuronal ensembles: a role for interneuronal networks. | Q40375750 | ||
Persistently modified h-channels after complex febrile seizures convert the seizure-induced enhancement of inhibition to hyperexcitability | Q42214350 | ||
Activity deprivation reduces miniature IPSC amplitude by decreasing the number of postsynaptic GABA(A) receptors clustered at neocortical synapses. | Q42670226 | ||
Postsynaptic spiking homeostatically induces cell-autonomous regulation of inhibitory inputs via retrograde signaling. | Q42788404 | ||
Down-regulation of the potassium-chloride cotransporter KCC2 contributes to spasticity after spinal cord injury | Q43146807 | ||
Postsynaptic depolarization scales quantal amplitude in cortical pyramidal neurons. | Q43744896 | ||
alpha- and betaCaMKII. Inverse regulation by neuronal activity and opposing effects on synaptic strength | Q44258612 | ||
Inhibition of Na(+)-K(+)-Cl(-) cotransporter during focal cerebral ischemia decreases edema and neuronal damage | Q44283379 | ||
GABAA receptor subunit immunoreactivity in primate visual cortex: distribution in macaques and humans and regulation by visual input in adulthood. | Q44381491 | ||
Different forms of homeostatic plasticity are engaged with distinct temporal profiles | Q44984996 | ||
Selective reconfiguration of layer 4 visual cortical circuitry by visual deprivation | Q46229886 | ||
Postsynaptic expression of homeostatic plasticity at neocortical synapses. | Q46384452 | ||
The chloride transporter Na(+)-K(+)-Cl- cotransporter isoform-1 contributes to intracellular chloride increases after in vitro ischemia. | Q46923567 | ||
Activity-dependent changes in the intrinsic properties of cultured neurons | Q47925605 | ||
Reduction in number of immunostained GABAergic neurones in deprived-eye dominance columns of monkey area 17. | Q48353707 | ||
BDNF has opposite effects on the quantal amplitude of pyramidal neuron and interneuron excitatory synapses | Q48377879 | ||
Multiple forms of synaptic plasticity triggered by selective suppression of activity in individual neurons. | Q48435334 | ||
Potentiation of cortical inhibition by visual deprivation. | Q48438986 | ||
Activity-dependent regulation of inhibitory synaptic transmission in hippocampal neurons. | Q48595994 | ||
Brain-derived neurotrophic factor mediates the activity-dependent regulation of inhibition in neocortical cultures. | Q48683647 | ||
P275 | copyright license | Creative Commons Attribution 3.0 Unported | Q14947546 |
P6216 | copyright status | copyrighted | Q50423863 |
P407 | language of work or name | English | Q1860 |
P921 | main subject | cell | Q7868 |
amino acid | Q8066 | ||
nervous system | Q9404 | ||
carboxylic acid | Q134856 | ||
neuroplasticity | Q849491 | ||
butyrate | Q1018145 | ||
physiological phenomenon | Q66615932 | ||
P304 | page(s) | 489470 | |
P577 | publication date | 2011-08-17 | |
P1433 | published in | Neural Plasticity | Q15716605 |
P1476 | title | Mechanisms of GABAergic homeostatic plasticity | |
P478 | volume | 2011 |
Q35147839 | Activity blockade and GABAA receptor blockade produce synaptic scaling through chloride accumulation in embryonic spinal motoneurons and interneurons |
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Q34471254 | Alterations in cortical network oscillations and parvalbumin neurons in schizophrenia |
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Q37094054 | Bilateral transient changes in thalamic nucleus ventroposterior lateralis after thoracic hemisection in the rat |
Q21129486 | Gephyrin, the enigmatic organizer at GABAergic synapses |
Q37094059 | Hearing loss differentially affects thalamic drive to two cortical interneuron subtypes |
Q48550236 | Homeostatic plasticity induced by brief activity deprivation enhances long-term potentiation in the mature rat hippocampus. |
Q27024114 | Homeostatic synaptic plasticity in developing spinal networks driven by excitatory GABAergic currents |
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Q36850754 | Inactivity-induced phrenic and hypoglossal motor facilitation are differentially expressed following intermittent vs. sustained neural apnea |
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Q58766522 | Reducing inhibition: A promising new strategy for the treatment of schizophrenia |
Q48147001 | Regional Specificity of GABAergic Regulation of Cross-Modal Plasticity in Mouse Visual Cortex after Unilateral Enucleation. |
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Q27321496 | Rescue of inhibitory synapse strength following developmental hearing loss |
Q40010489 | The Anaphase-Promoting Complex (APC) ubiquitin ligase regulates GABA transmission at the C. elegans neuromuscular junction |
Q50481138 | The Cav3-Kv4 complex acts as a calcium sensor to maintain inhibitory charge transfer during extracellular calcium fluctuations. |
Q38060556 | Time scales of memory, learning, and plasticity |
Q47650436 | Tinnitus: Prospects for Pharmacological Interventions With a Seesaw Model |
Q37699493 | Transcriptional and epigenetic regulation of Hebbian and non-Hebbian plasticity |
Q35560224 | Visual experience prevents dysregulation of GABAB receptor-dependent short-term depression in adult superior colliculus |
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