Filaggrin deficiency promotes the dissemination of cutaneously inoculated vaccinia virus

scientific article

Filaggrin deficiency promotes the dissemination of cutaneously inoculated vaccinia virus is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1016/J.JACI.2014.12.1923
P932PMC publication ID4461532
P698PubMed publication ID25649082

P50authorYoichiro IwakuraQ56564114
Michiko K OyoshiQ62752735
Christopher N LewisQ83641769
Raif S. GehaQ87942862
P2093author name stringJacqueline Beaupré
Nicholas Venturelli
P2860cites workChildhood atopic dermatitis and warts are associated with increased risk of infection: a US population-based studyQ40232619
Smallpox vaccination: Risk considerations for patients with atopic dermatitisQ44441962
Impaired immune response to vaccinia virus inoculated at the site of cutaneous allergic inflammationQ45399970
Skin inflammation in RelB(-/-) mice leads to defective immunity and impaired clearance of vaccinia virus.Q45406565
IL-17 in atopic eczema: linking allergen-specific adaptive and microbial-triggered innate immune response.Q46214370
IL-17 suppresses TNF-alpha-induced CCL27 production through induction of COX-2 in human keratinocytesQ46794704
Epicutaneous sensitization with superantigen induces allergic skin inflammation.Q54265045
Breaking the (Un)Sound Barrier: Filaggrin Is a Major Gene for Atopic DermatitisQ58859847
Altered stratum corneum barrier and enhanced percutaneous immune responses in filaggrin-null miceQ83627641
Deciphering the complexities of atopic dermatitis: shifting paradigms in treatment approachesQ26852335
Eczema vaccinatumQ27009282
Loss-of-function variations within the filaggrin gene predispose for atopic dermatitis with allergic sensitizationsQ28249806
Interleukin-17 and type 17 helper T cellsQ28256510
Common loss-of-function variants of the epidermal barrier protein filaggrin are a major predisposing factor for atopic dermatitisQ28303063
Tmem79/Matt is the matted mouse gene and is a predisposing gene for atopic dermatitis in human subjectsQ28511427
Two new mutations in linkage group XVI of the house mouse. Flaky tail and varitint-waddler-JQ28586548
Filaggrin deficiency confers a paracellular barrier abnormality that reduces inflammatory thresholds to irritants and haptensQ30494794
Flaky tail mouse denotes human atopic dermatitis in the steady state and by topical application with Dermatophagoides pteronyssinus extract.Q33816405
A homozygous frameshift mutation in the mouse Flg gene facilitates enhanced percutaneous allergen priming.Q33858072
Filaggrin-stratified transcriptomic analysis of pediatric skin identifies mechanistic pathways in patients with atopic dermatitisQ33877517
Filaggrin-deficient mice exhibit TH17-dominated skin inflammation and permissiveness to epicutaneous sensitization with protein antigenQ33915290
Th17 cytokines stimulate CCL20 expression in keratinocytes in vitro and in vivo: implications for psoriasis pathogenesisQ33941199
Requirement of interleukin 17 receptor signaling for lung CXC chemokine and granulocyte colony-stimulating factor expression, neutrophil recruitment, and host defenseQ34087551
Filaggrin mutations associated with skin and allergic diseases.Q34223419
Identification of novel gene signatures in patients with atopic dermatitis complicated by eczema herpeticumQ34294746
Human atopic dermatitis complicated by eczema herpeticum is associated with abnormalities in IFN-γ responseQ34781786
Viral infections in atopic dermatitis Pathogenic aspects and clinical managementQ35562168
IL-10 is critical for Th2 responses in a murine model of allergic dermatitisQ36001280
Epicutaneous antigen exposure induces a Th17 response that drives airway inflammation after inhalation challengeQ36023872
Distinct time effects of vaccination on long-term proliferative and IFN-gamma-producing T cell memory to smallpox in humansQ36399092
The Th17 family: flexibility follows function.Q36713721
TSLP acts on infiltrating effector T cells to drive allergic skin inflammationQ36954478
IL-21R is essential for epicutaneous sensitization and allergic skin inflammation in humans and mice.Q37036021
Toll-like receptor 2 is important for the T(H)1 response to cutaneous sensitizationQ37199491
Vaccinia virus inoculation in sites of allergic skin inflammation elicits a vigorous cutaneous IL-17 responseQ37329533
Epicutaneous sensitization with protein antigen induces localized allergic dermatitis and hyperresponsiveness to methacholine after single exposure to aerosolized antigen in miceQ37380666
Filaggrin mutations that confer risk of atopic dermatitis confer greater risk for eczema herpeticum.Q37406747
Transcutaneous yellow fever vaccination of subjects with or without atopic dermatitisQ37649830
Thymic stromal lymphopoietin and allergic diseaseQ38039733
The cutaneous innate immune response in patients with atopic dermatitis.Q38078473
The multifunctional role of filaggrin in allergic skin diseaseQ38078474
Regulation of Th17 maturation by interleukin 4.Q38149958
Causes of epidermal filaggrin reduction and their role in the pathogenesis of atopic dermatitisQ38234353
Overexpression of IL-1alpha in skin differentially modulates the immune response to scarification with vaccinia virusQ39706186
P433issue6
P407language of work or nameEnglishQ1860
P921main subjectvirologyQ7215
Vaccinia virusQ1986297
P304page(s)1511-8.e6
P577publication date2015-01-30
P1433published inThe Journal of Allergy and Clinical ImmunologyQ7743550
P1476titleFilaggrin deficiency promotes the dissemination of cutaneously inoculated vaccinia virus
P478volume135

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cites work (P2860)
Q38885691Bacterial and Viral Infections in Atopic Dermatitis: a Comprehensive Review
Q38753828Effects of Allergic Sensitization on Antiviral Immunity: Allergen, Virus, and Host Cell Mechanisms
Q92406780Host-microbial dialogues in atopic dermatitis
Q91626434Injury, dysbiosis, and filaggrin deficiency drive skin inflammation through keratinocyte IL-1α release

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