scholarly article | Q13442814 |
P50 | author | Harvey J. Alter | Q5677307 |
Barbara Rehermann | Q28052958 | ||
P2093 | author name string | T Jake Liang | |
Stephen M Feinstone | |||
Barbara Rehermann | |||
Michelina Nascimbeni | |||
Michael B Havert | |||
Michael Thomson | |||
Krishna K Murthy | |||
Marian Major | |||
Sophia Gonzales | |||
P2860 | cites work | Viral persistence, antibody to E1 and E2, and hypervariable region 1 sequence stability in hepatitis C virus-inoculated chimpanzees | Q27469342 |
Long-term follow-up of chimpanzees inoculated with the first infectious clone for hepatitis C virus | Q27469355 | ||
Sustained Dysfunction of Antiviral CD8+ T Lymphocytes after Infection with Hepatitis C Virus | Q27469800 | ||
Molecular and immunological significance of chimpanzee major histocompatibility complex haplotypes for hepatitis C virus immune response and vaccination studies | Q27472879 | ||
Stability of a stem-loop involving the initiator AUG controls the efficiency of internal initiation of translation on hepatitis C virus RNA. | Q27472947 | ||
Patr-A and B, the orthologues of HLA-A and B, present hepatitis C virus epitopes to CD8+ cytotoxic T cells from two chronically infected chimpanzees | Q27485117 | ||
Transcripts from a single full-length cDNA clone of hepatitis C virus are infectious when directly transfected into the liver of a chimpanzee | Q27485872 | ||
Prevention of hepatitis C virus infection in chimpanzees by hyperimmune serum against the hypervariable region 1 of the envelope 2 protein | Q27487933 | ||
Transmission of hepatitis C by intrahepatic inoculation with transcribed RNA | Q27860484 | ||
Analysis of successful immune responses in persons infected with hepatitis C virus | Q27860698 | ||
Determinants of viral clearance and persistence during acute hepatitis C virus infection | Q28131757 | ||
Analysis of a successful immune response against hepatitis C virus | Q29619507 | ||
Possible mechanism involving T-lymphocyte response to non-structural protein 3 in viral clearance in acute hepatitis C virus infection | Q29620141 | ||
Cellular immune responses persist and humoral responses decrease two decades after recovery from a single-source outbreak of hepatitis C | Q29620614 | ||
Hepatitis C virus replication in mice with chimeric human livers | Q29620650 | ||
Recovery, persistence, and sequelae in hepatitis C virus infection: a perspective on long-term outcome | Q29620780 | ||
Recurrence of hepatitis C virus after loss of virus-specific CD4(+) T-cell response in acute hepatitis C | Q29620791 | ||
Different clinical behaviors of acute hepatitis C virus infection are associated with different vigor of the anti-viral cell-mediated immune response | Q29620891 | ||
The outcome of hepatitis C virus infection is predicted by escape mutations in epitopes targeted by cytotoxic T lymphocytes | Q30004698 | ||
Noncytolytic control of viral infections by the innate and adaptive immune response | Q34178107 | ||
NK cells cause liver injury and facilitate the induction of T cell-mediated immunity to a viral liver infection | Q41740142 | ||
An infectious molecular clone of a Japanese genotype 1b hepatitis C virus | Q42604623 | ||
Transcripts of a chimeric cDNA clone of hepatitis C virus genotype 1b are infectious in vivo. | Q42677425 | ||
Generation of transmissible hepatitis C virions from a molecular clone in chimpanzees | Q42992662 | ||
Hepatitis C virus: an infectious molecular clone of a second major genotype (2a) and lack of viability of intertypic 1a and 2a chimeras | Q42995261 | ||
Vigorous peripheral blood cytotoxic T cell response during the acute phase of hepatitis C virus infection | Q43000322 | ||
Infectious cDNA clone of the hepatitis C virus genotype 1 prototype sequence | Q43031649 | ||
Emergence of a distinct pattern of viral mutations in chimpanzees infected with a homogeneous inoculum of hepatitis C virus | Q43035114 | ||
CD8+ T lymphocyte responses are induced during acute hepatitis C virus infection but are not sustained | Q45740431 | ||
Vital involvement of a natural killer cell activation receptor in resistance to viral infection. | Q54010687 | ||
Viral Clearance Without Destruction of Infected Cells During Acute HBV Infection | Q56951258 | ||
P433 | issue | 2 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | virology | Q7215 |
hepatitis C | Q154869 | ||
Hepatitis C virus | Q708693 | ||
P304 | page(s) | 862-870 | |
P577 | publication date | 2003-01-01 | |
P1433 | published in | Journal of Virology | Q1251128 |
P1476 | title | The clearance of hepatitis C virus infection in chimpanzees may not necessarily correlate with the appearance of acquired immunity | |
P478 | volume | 77 |
Q33305432 | "Negative vaccination" by specific CD4 T cell tolerisation enhances virus-specific protective antibody responses |
Q35797579 | A critical role for the chimpanzee model in the study of hepatitis C. |
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Q37381208 | Comprehensive analyses of CD8+ T cell responses during longitudinal study of acute human hepatitis C. |
Q34552781 | Control of heterologous hepatitis C virus infection in chimpanzees is associated with the quality of vaccine-induced peripheral T-helper immune response |
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Q40056632 | Extralymphatic virus sanctuaries as a consequence of potent T-cell activation |
Q27480306 | Foxp3+CD4+CD25+ T cells control virus-specific memory T cells in chimpanzees that recovered from hepatitis C |
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Q27487011 | Hepatic Transcriptome Analysis of Hepatitis C Virus Infection in Chimpanzees Defines Unique Gene Expression Patterns Associated with Viral Clearance |
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Q50934385 | Immunopathogenesis of Hepatitis C Virus Infection. |
Q42983326 | Increased frequency of CD56Bright NK-cells, CD3-CD16+CD56- NK-cells and activated CD4+T-cells or B-cells in parallel with CD4+CDC25High T-cells control potentially viremia in blood donors with HCV. |
Q45408928 | Induction of hepatitis C virus (HCV)-specific T cells by needle stick injury in the absence of HCV-viraemia |
Q46753818 | Interferon therapy shifts natural killer subsets among Egyptian patients with chronic hepatitis C |
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Q37417170 | Natural killer cell receptors and their ligands in liver diseases. |
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Q36290084 | Natural killer cells in hepatitis C virus infection: from innate immunity to adaptive immunity |
Q37754682 | Natural killer cells: versatile roles in autoimmune and infectious diseases |
Q33826844 | Novel infectious cDNA clones of hepatitis C virus genotype 3a (strain S52) and 4a (strain ED43): genetic analyses and in vivo pathogenesis studies |
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Q27860899 | Production of infectious hepatitis C virus in tissue culture from a cloned viral genome |
Q35784239 | Prospects for dendritic cell vaccination in persistent infection with hepatitis C virus |
Q24814157 | Replicative homeostasis II: influence of polymerase fidelity on RNA virus quasispecies biology: implications for immune recognition, viral autoimmunity and other "virus receptor" diseases |
Q35817615 | Role of NK and NKT cells in the immunopathogenesis of HCV-induced hepatitis. |
Q27469468 | Scavenger receptor class B type I and hepatitis C virus infection of primary tupaia hepatocytes. |
Q37330751 | Scientific consultation on cell mediated immunity (CMI) in dengue and dengue vaccine development |
Q28484092 | Spontaneous clearance of viral infections by mesoscopic fluctuations |
Q42981653 | Suppression of HCV-specific T cells without differential hierarchy demonstrated ex vivo in persistent HCV infection |
Q45422073 | Sustained E2 antibody response correlates with reduced peak viremia after hepatitis C virus infection in the chimpanzee |
Q50569016 | Sustained response to interferon-alpha plus ribavirin therapy for chronic hepatitis C is closely associated with increased dynamism of intrahepatic natural killer and natural killer T cells. |
Q39474013 | The effects of hepatitis C virus core protein on functional responses in the NK cell line YTS. |
Q43036305 | Vaccine-induced early control of hepatitis C virus infection in chimpanzees fails to impact on hepatic PD-1 and chronicity |
Q39186289 | Virus-induced hepatocellular carcinomas cause antigen-specific local tolerance |
Q80762443 | [Injuries from needles contaminated with hepatitis C virus: how high is the risk of seroconversion for medical personnel really?] |
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