| scholarly article | Q13442814 |
| P50 | author | Renato Mantegazza | Q43161279 |
| Marina Mora | Q47504621 | ||
| Francesca Andreetta | Q56507615 | ||
| Laura Cristina Luna Oliva | Q92499073 | ||
| Paolo Confalonieri | Q114413329 | ||
| Ferdinando Cornelio | Q114413349 | ||
| Lucia Morandi | Q117250877 | ||
| P2093 | author name string | Elisabetta Mariani | |
| Patrizia Dassi | |||
| Rossella Lorenzoni | |||
| P2860 | cites work | Cloning of the mouse dysferlin gene and genomic characterization of the SJL-Dysf mutation | Q28203165 |
| Dysferlin, a novel skeletal muscle gene, is mutated in Miyoshi myopathy and limb girdle muscular dystrophy | Q28281738 | ||
| Making sense of the limb-girdle muscular dystrophies | Q33701932 | ||
| The limb-girdle muscular dystrophies-multiple genes, multiple mechanisms | Q33724091 | ||
| Conditional up-regulation of MHC class I in skeletal muscle leads to self-sustaining autoimmune myositis and myositis-specific autoantibodies | Q33912061 | ||
| Expression of immunoreactive major histocompatibility complex products in human skeletal muscles | Q34686933 | ||
| Role of immune cells in animal models for inherited neuropathies: facts and visions | Q34713844 | ||
| Up-regulation of MHC class I expression accompanies but is not required for spontaneous myopathy in dysferlin-deficient SJL/J mice | Q35788717 | ||
| Experimental allergic myositis in SJL/J mouse. Reappraisal of immune reaction based on changes after single immunization | Q38296216 | ||
| The immunobiology of muscle | Q40693166 | ||
| Immunopathogenesis of inflammatory myopathies | Q41270505 | ||
| Multiplex Western blotting system for the analysis of muscular dystrophy proteins | Q42108368 | ||
| A new dysferlin gene mutation in two Japanese families with limb-girdle muscular dystrophy 2B and Miyoshi myopathy | Q43546884 | ||
| Inflammation in dysferlin myopathy: immunohistochemical characterization of 13 patients. | Q43820156 | ||
| Dysferlin deletion in SJL mice (SJL-Dysf) defines a natural model for limb girdle muscular dystrophy 2B | Q57990899 | ||
| Dystrophin-associated protein abnormalities in dystrophin-deficient muscle fibers from symptomatic and asymptomatic Duchenne/Becker muscular dystrophy carriers | Q63457022 | ||
| Monoclonal antibody analysis of mononuclear cells in myopathies. I: Quantitation of subsets according to diagnosis and sites of accumulation and demonstration and counts of muscle fibers invaded by T cells | Q70379552 | ||
| Complement activation in muscle fiber necrosis: demonstration of the membrane attack complex of complement in necrotic fibers | Q71451319 | ||
| Prominent inflammatory changes on muscle biopsy in patients with Miyoshi myopathy | Q73141362 | ||
| Phenotypic variation in a large Japanese family with Miyoshi myopathy with nonsense mutation in exon 19 of dysferlin gene | Q73568744 | ||
| Splicing mutation in dysferlin produces limb-girdle muscular dystrophy with inflammation | Q73680942 | ||
| Regulation of class I MHC expression in skeletal muscle: deleterious effect of aberrant expression on myogenesis | Q77963004 | ||
| P433 | issue | 1-2 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | inflammation | Q101991 |
| muscular dystrophy | Q1137767 | ||
| P304 | page(s) | 130-136 | |
| P577 | publication date | 2003-09-01 | |
| P1433 | published in | Journal of Neuroimmunology | Q15716691 |
| P1476 | title | Muscle inflammation and MHC class I up-regulation in muscular dystrophy with lack of dysferlin: an immunopathological study | |
| P478 | volume | 142 |
| Q54520022 | Absence of T and B lymphocytes modulates dystrophic features in dysferlin deficient animal model. |
| Q51089580 | Annexin A2 links poor myofiber repair with inflammation and adipogenic replacement of the injured muscle. |
| Q83418518 | Anti-TNF therapy using etanercept suppresses degenerative and inflammatory changes in skeletal muscle of older SJL/J mice |
| Q35632288 | CD4+ cells, macrophages, MHC-I and C5b-9 involve the pathogenesis of dysferlinopathy |
| Q28475580 | Characterization of dysferlin deficient SJL/J mice to assess preclinical drug efficacy: fasudil exacerbates muscle disease phenotype |
| Q37372933 | Clinical and laboratory features distinguishing juvenile polymyositis and muscular dystrophy. |
| Q40895111 | Clinico pathological study of adult dermatomyositis: Importance of muscle histology in the diagnosis |
| Q39703713 | Comparative Gene Expression Analysis in the Skeletal Muscles of Dysferlin-deficient SJL/J and A/J Mice |
| Q39751162 | Dexamethasone induces dysferlin in myoblasts and enhances their myogenic differentiation |
| Q36562003 | Differential diagnosis of idiopathic inflammatory myopathies |
| Q37417904 | Differential immunohistological features of inflammatory myopathies and dysferlinopathy |
| Q36085192 | Dysferlin and animal models for dysferlinopathy |
| Q36482213 | Dysferlin deficiency enhances monocyte phagocytosis: a model for the inflammatory onset of limb-girdle muscular dystrophy 2B. |
| Q36943096 | Dysferlin deficiency shows compensatory induction of Rab27A/Slp2a that may contribute to inflammatory onset. |
| Q43280975 | Dysferlin deficiency treated like refractory polymyositis |
| Q38092854 | Dysferlin-deficient muscular dystrophy and innate immune activation. |
| Q34785515 | Effects of nitric oxide on notexin-induced muscle inflammatory responses |
| Q35987272 | Enhanced Muscular Dystrophy from Loss of Dysferlin Is Accompanied by Impaired Annexin A6 Translocation after Sarcolemmal Disruption |
| Q41860710 | Expression levels of TGF-β1 and CTGF are associated with the severity of Duchenne muscular dystrophy |
| Q33655550 | Extensive mononuclear infiltration and myogenesis characterize recovery of dysferlin-null skeletal muscle from contraction-induced injuries |
| Q34360151 | Genetic ablation of complement C3 attenuates muscle pathology in dysferlin-deficient mice |
| Q37413634 | Genetic manipulation of dysferlin expression in skeletal muscle: novel insights into muscular dystrophy |
| Q51725324 | Heterogeneous characteristics of MRI changes of thigh muscles in patients with dysferlinopathy. |
| Q53588069 | Histological and immunohistological changes of the skeletal muscles in older SJL/J mice. |
| Q39106643 | Immune-Mediated Muscle Diseases of the Horse |
| Q91446266 | Immunobiology of Inherited Muscular Dystrophies |
| Q34627294 | Immunotherapy of inflammatory myopathies: practical approach and future prospects |
| Q33882512 | Inflammasome up-regulation and activation in dysferlin-deficient skeletal muscle |
| Q57366892 | Inflammatory and immuno-reactivity in mice induced by intramuscular implants of HSNGLPL peptide grafted-polyurethane |
| Q34094843 | Limb-girdle muscular dystrophy subtypes: First-reported cohort from northeastern China |
| Q38379551 | Major Histocompatibility Complex I and II Expression and Lymphocytic Subtypes in Muscle of Horses with Immune-Mediated Myositis |
| Q34360555 | Membrane repair and immunological danger. |
| Q42209023 | Muscle membrane repair and inflammatory attack in dysferlinopathy. |
| Q30596300 | Myofiber HLA-DR expression is a distinctive biomarker for antisynthetase-associated myopathy |
| Q35669024 | Myofiber damage precedes macrophage infiltration after in vivo injury in dysferlin-deficient A/J mouse skeletal muscle |
| Q57389951 | Myopathy caused by anoctamin 5 mutations and necrotizing vasculitis |
| Q38569863 | Myositis Mimics |
| Q38921880 | Progress and challenges in diagnosis of dysferlinopathy |
| Q37820692 | Review: An update on inflammatory and autoimmune myopathies |
| Q58875758 | The Kinesin Superfamily Motor Protein KIF4 Is Associated With Immune Cell Activation in Idiopathic Inflammatory Myopathies |
| Q35876136 | The composition of cellular infiltrates in anti-HMG-CoA reductase-associated myopathy |
| Q50066457 | Thrombospondin-1 and disease progression in dysferlinopathy. |
| Q35588331 | Ultrastructural changes in dysferlinopathy support defective membrane repair mechanism |
| Q37154934 | Upregulation of MHC class I in transgenic mice results in reduced force-generating capacity in slow-twitch muscle. |
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