| scholarly article | Q13442814 |
| P5530 | Altmetric DOI | 10.1038/JID.2014.532 |
| P6409 | CORE ID | 82299961 |
| P6179 | Dimensions Publication ID | 1033323389 |
| P356 | DOI | 10.1038/JID.2014.532 |
| P953 | full work available at URL | http://eprints.whiterose.ac.uk/85850/ |
| P3181 | OpenCitations bibliographic resource ID | 2152728 |
| P698 | PubMed publication ID | 25525775 |
| P1154 | Scopus EID | 2-s2.0-84925381286 |
| P50 | author | Joerg Wenzel | Q47726648 |
| P2093 | author name string | Thomas Bieber | |
| Angelo Massimiliano D'Erme | |||
| Andreas Bosio | |||
| Michael Hölzel | |||
| Dagmar Wilsmann-Theis | |||
| Bettina Peters | |||
| Miriam Wittmann | |||
| Sandra Ferring-Schmitt | |||
| Julia Wagenpfeil | |||
| Sonja Sternberg | |||
| P2860 | cites work | Anti-IL-36R antibodies, potentially useful for the treatment of psoriasis: a patent evaluation of WO2013074569 | Q87152167 |
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| Enhanced type I interferon signalling promotes Th1-biased inflammation in cutaneous lupus erythematosus | Q81349191 | ||
| Inter-regulation of Th17 cytokines and the IL-36 cytokines in vitro and in vivo: implications in psoriasis pathogenesis | Q84865125 | ||
| P433 | issue | 4 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | biomarker | Q864574 |
| P304 | page(s) | 1025-32 | |
| P577 | publication date | 2015-04-01 | |
| P1433 | published in | Journal of Investigative Dermatology | Q3186921 |
| P1476 | title | IL-36γ (IL-1F9) is a biomarker for psoriasis skin lesions | |
| P478 | volume | 135 |
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| Q46547722 | Autocrine and Paracrine Regulation of Keratinocyte Proliferation through a Novel Nrf2-IL-36γ Pathway |
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| Q26781378 | Biomarkers of An Autoimmune Skin Disease--Psoriasis |
| Q96769267 | CARD14E138A signalling in keratinocytes induces TNF-dependent skin and systemic inflammation |
| Q64238214 | Cathepsin G cleaves and activates IL-36γ and promotes the inflammation of psoriasis |
| Q34553191 | Cathepsin S is the major activator of the psoriasis-associated proinflammatory cytokine IL-36γ. |
| Q49185989 | Commensal microbiota modulate gene expression in the skin. |
| Q38840151 | Cross-Disease Transcriptomics: Unique IL-17A Signaling in Psoriasis Lesions and an Autoimmune PBMC Signature |
| Q28255491 | Cytokines in psoriasis |
| Q56591790 | Detection of IL-36γ through noninvasive tape stripping reliably discriminates psoriasis from atopic eczema |
| Q62660641 | Disrupting the IL-36 and IL-23/IL-17 loop underlies the efficacy of calcipotriol and corticosteroid therapy for psoriasis |
| Q97526290 | Diterpenoids and Triterpenoids From Frankincense Are Excellent Anti-psoriatic Agents: An in silico Approach |
| Q96135358 | Dithranol targets keratinocytes, their crosstalk with neutrophils and inhibits the IL-36 inflammatory loop in psoriasis |
| Q52886990 | Elevation of serum squamous cell carcinoma antigen 2 in patients with psoriasis: associations with disease severity and response to the treatment. |
| Q46242608 | Epidermal cornification is preceded by the expression of a keratinocyte-specific set of pyroptosis-related genes |
| Q92191072 | Function and Regulation of IL-36 Signaling in Inflammatory Diseases and Cancer Development |
| Q64080513 | IL-36 Cytokines: Regulators of Inflammatory Responses and Their Emerging Role in Immunology of Reproduction |
| Q94451656 | IL-36 signaling in keratinocytes controls early IL-23 production in psoriasis-like dermatitis |
| Q52802857 | IL-36γ Is a Strong Inducer of IL-23 in Psoriatic Cells and Activates Angiogenesis. |
| Q40526106 | IL-36γ has proinflammatory effects on human endothelial cells |
| Q41035494 | IL-36γ inhibits differentiation and induces inflammation of keratinocyte via Wnt signaling pathway in psoriasis |
| Q58093804 | IL-38 has an anti-inflammatory action in psoriasis and its expression correlates with disease severity and therapeutic response to anti-IL-17A treatment |
| Q88872584 | Ichthyosis molecular fingerprinting shows profound TH17 skewing and a unique barrier genomic signature |
| Q55111930 | Identification of small-molecule elastase inhibitors as antagonists of IL-36 cytokine activation. |
| Q48295806 | Increased expression of the interleukin-36 cytokines in lesions of hidradenitis suppurativa. |
| Q48288323 | Increased interleukin-36γ expression in skin and sera of patients with atopic dermatitis and mycosis fungoides/Sézary syndrome. |
| Q90322928 | Inhibition of Neutrophil Elastase and Cathepsin G As a New Approach to the Treatment of Psoriasis: From Fundamental Biology to Development of New Target-Specific Drugs |
| Q90126307 | Interleukin-36 (IL-36) system in the 1-fluoro-2,4-dinitrobenzene (DNFB) mouse model of allergic contact dermatitis |
| Q64892752 | Interleukin-36 in Infectious and Inflammatory Skin Diseases. |
| Q41825896 | Interleukin-36γ is expressed by neutrophils and can activate microglia, but has no role in experimental autoimmune encephalomyelitis. |
| Q57050102 | IκBζ is a key transcriptional regulator of IL-36-driven psoriasis-related gene expression in keratinocytes |
| Q40252612 | Juvenile generalized pustular psoriasis with IL36RN mutation treated with short-term infliximab |
| Q88115668 | Landscape of long non-coding RNAs in Trichophyton mentagrophytes-induced rabbit dermatophytosis lesional skin and normal skin |
| Q90482659 | Metformin inhibits IL-1β secretion via impairment of NLRP3 inflammasome in keratinocytes: implications for preventing the development of psoriasis |
| Q37601487 | Mutations in IL36RN are associated with geographic tongue |
| Q40473700 | Neutrophil Elastase-mediated proteolysis activates the anti-inflammatory cytokine IL-36 Receptor antagonist. |
| Q64071657 | Neutrophil Extracellular Traps Promote Inflammatory Responses in Psoriasis via Activating Epidermal TLR4/IL-36R Crosstalk |
| Q51072843 | Neutrophil extracellular traps can serve as platforms for processing and activation of IL-1 family cytokines. |
| Q93186368 | Non-invasive Approaches for the Diagnosis of Autoimmune/Autoinflammatory Skin Diseases-A Focus on Psoriasis and Lupus erythematosus |
| Q58102982 | Patients with Congenital Ichthyosis and TGM1 Mutations Overexpress Other ARCI Genes in the Skin: Part of a Barrier Repair Response? |
| Q92262103 | Personalized medicine-concepts, technologies, and applications in inflammatory skin diseases |
| Q35922976 | Proteogenomic analysis of psoriasis reveals discordant and concordant changes in mRNA and protein abundance. |
| Q26744032 | Psoriasis, psoriatic arthritis, and rheumatoid arthritis: Is all inflammation the same? |
| Q47358944 | Quantitative ligand and receptor binding studies reveal the mechanism of interleukin-36 (IL-36) pathway activation |
| Q50046027 | RNA-Seq Analysis of IL-1B and IL-36 Responses in Epidermal Keratinocytes Identifies a Shared MyD88-Dependent Gene Signature |
| Q64925719 | RNA-seq and flow-cytometry of conventional, scalp, and palmoplantar psoriasis reveal shared and distinct molecular pathways. |
| Q91735227 | Scoping review of biological treatment of deficiency of interleukin-36 receptor antagonist (DITRA) in children and adolescents |
| Q92999995 | Small Molecule IL-36γ Antagonist as a Novel Therapeutic Approach for Plaque Psoriasis |
| Q33713754 | Spotlight on dupilumab in the treatment of atopic dermatitis: design, development, and potential place in therapy |
| Q50094846 | Staphylococcus aureus Virulent PSMα Peptides Induce Keratinocyte Alarmin Release to Orchestrate IL-17-Dependent Skin Inflammation |
| Q91715185 | The Significance of IL-36 Hyperactivation and IL-36R Targeting in Psoriasis |
| Q46320645 | The psoriasis-associated IL-17A induces and cooperates with IL-36 cytokines to control keratinocyte differentiation and function |
| Q57465642 | The small molecule rhodomyrtone suppresses TNF-α and IL-17A-induced keratinocyte inflammatory responses: A potential new therapeutic for psoriasis |
| Q35519210 | Transcriptional landscape of psoriasis identifies the involvement of IL36 and IL36RN. |
| Q90164292 | Transcriptome analysis of HPV-induced warts and healthy skin in humans |
| Q48282383 | Tumour necrosis factor-α-inhibitor-induced neutrophilic folliculitis presenting with strong lesional expression of interleukin-36γ. |
| Q36289222 | Unprocessed Interleukin-36α Regulates Psoriasis-Like Skin Inflammation in Cooperation With Interleukin-1 |
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