Motor neurons in Cu/Zn superoxide dismutase-deficient mice develop normally but exhibit enhanced cell death after axonal injury

scientific journal article

Motor neurons in Cu/Zn superoxide dismutase-deficient mice develop normally but exhibit enhanced cell death after axonal injury is …
instance of (P31):
scholarly articleQ13442814

External links are
P6179Dimensions Publication ID1047868408
P356DOI10.1038/NG0596-43
P3181OpenCitations bibliographic resource ID144126
P698PubMed publication ID8673102

P50authorM. Flint BealQ67409129
P2093author name stringA. G. Reaume
D. F. Siwek
D. G. Flood
E. K. Hoffman
H. M. Wilcox
J. L. Elliott
N. W. Kowall
R. H. Brown
R. J. Ferrante
R. W. Scott
W. D. Snider
P2860cites workAmyotrophic lateral sclerosis and structural defects in Cu,Zn superoxide dismutaseQ27731976
Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosisQ28131805
Dilated cardiomyopathy and neonatal lethality in mutant mice lacking manganese superoxide dismutaseQ28284593
Disruption of the proto-oncogene int-2 in mouse embryo-derived stem cells: a general strategy for targeting mutations to non-selectable genesQ29547355
Motor neuron degeneration in mice that express a human Cu,Zn superoxide dismutase mutationQ29547561
Null mutation of copper/zinc superoxide dismutase in Drosophila confers hypersensitivity to paraquat and reduced longevityQ33850583
Superoxide dismutase 1 with mutations linked to familial amyotrophic lateral sclerosis possesses significant activityQ35693516
Reactions of nitric oxide, superoxide and peroxynitrite with superoxide dismutase in neurodegenerationQ40610035
Human disease, free radicals, and the oxidant/antioxidant balanceQ40810054
Altered reactivity of superoxide dismutase in familial amyotrophic lateral sclerosisQ41237348
Cardiac malformation in neonatal mice lacking connexin43.Q41358695
Extracellular superoxide dismutase and other superoxide dismutase isoenzymes in tissues from nine mammalian speciesQ42848083
Brain-derived neurotrophic factor rescues spinal motor neurons from axotomy-induced cell deathQ46535566
Carbonyl assays for determination of oxidatively modified proteinsQ54644873
An adverse property of a familial ALS-linked SOD1 mutation causes motor neuron disease characterized by vacuolar degeneration of mitochondriaQ54965159
Determination of glutathione and glutathione disulfide using glutathione reductase and 2-vinylpyridineQ56879285
Non-injection methods for the production of embryonic stem cell-embryo chimaerasQ59092151
A sensitive spectrophotometric method for the determination of superoxide dismutase activity in tissue extractsQ63473138
Is hydroxyl radical generated by the Fenton reaction in vivo?Q70095449
Superoxide dismutase activity, oxidative damage, and mitochondrial energy metabolism in familial and sporadic amyotrophic lateral sclerosisQ72621641
P433issue1
P407language of work or nameEnglishQ1860
P921main subjectcell deathQ2383867
Superoxide dismutase 1, solubleQ15328173
P304page(s)43–47
P577publication date1996-05-01
P1433published inNature GeneticsQ976454
P1476titleMotor neurons in Cu/Zn superoxide dismutase-deficient mice develop normally but exhibit enhanced cell death after axonal injury
P478volume13