scholarly article | Q13442814 |
P2093 | author name string | Jing Tan | |
D. Jean White | |||
David L. Maass | |||
Jureta W. Horton | |||
P2860 | cites work | Protein kinase C isoform expression and regulation in the developing rat heart | Q28258963 |
A protein kinase C translocation inhibitor as an isozyme-selective antagonist of cardiac function | Q28289468 | ||
Cardiotrophic Effects of Protein Kinase C : Analysis by In Vivo Modulation of PKC Translocation | Q29393083 | ||
Myocardial inflammatory responses to sepsis complicated by previous burn injury | Q33199249 | ||
Myocardial dysfunction in septic shock | Q33893312 | ||
Interleukin 1 and tumor necrosis factor inhibit cardiac myocyte beta-adrenergic responsiveness | Q34301819 | ||
Intracellular transport mechanisms of signal transducers | Q34516202 | ||
A circulating myocardial depressant substance in humans with septic shock. Septic shock patients with a reduced ejection fraction have a circulating factor that depresses in vitro myocardial cell performance | Q34560402 | ||
Hypoxia alters the subcellular distribution of protein kinase C isoforms in neonatal rat ventricular myocytes | Q37362277 | ||
Molecular analysis of the interactions between protein kinase C-epsilon and filamentous actin | Q38332864 | ||
Activation of NF-kappaB by antineoplastic agents. Role of protein kinase C. | Q38345137 | ||
Isoform-specific induction of PKC-epsilon by high glucose protects heart-derived H9c2 cells against hypoxic injury | Q38519666 | ||
Endogenous calcitonin gene-related peptide protects human alveolar epithelial cells through protein kinase Cepsilon and heat shock protein | Q40443576 | ||
Protein kinase C, interleukin-1 beta, and corticosteroids regulate shedding of the type I, 55 kDa TNF receptor from human airway epithelial cells | Q41220470 | ||
Effects of fluid resuscitation on cardiac dysfunction following thermal injury | Q41437750 | ||
Decreased contractility and compliance of the left ventricle as complications of thermal trauma | Q41567432 | ||
The time course of cardiac NF-kappaB activation and TNF-alpha secretion by cardiac myocytes after burn injury: contribution to burn-related cardiac contractile dysfunction | Q43960162 | ||
I kappa B overexpression in cardiomyocytes prevents NF-kappa B translocation and provides cardioprotection in trauma | Q44310024 | ||
Inhibition of delta-protein kinase C protects against reperfusion injury of the ischemic heart in vivo | Q44642004 | ||
Conventional protein kinase C and atypical protein kinase Czeta differentially regulate macrophage production of tumour necrosis factor-alpha and interleukin-10. | Q44850861 | ||
Selective decontamination of the digestive tract attenuated the myocardial inflammation and dysfunction that occur with burn injury | Q44980688 | ||
Preservation of base-line hemodynamic function and loss of inducible cardioprotection in adult mice lacking protein kinase C epsilon | Q46673227 | ||
Inhibition of tumor necrosis factor prevents myocardial dysfunction during burn shock. | Q51629796 | ||
P433 | issue | 2 | |
P304 | page(s) | R887–896 | |
P577 | publication date | 2007-02-01 | |
P1433 | published in | American Journal of Physiology - Regulatory, Integrative and Comparative Physiology | Q2201819 |
P1476 | title | Effects of burn injury on myocardial signaling and cytokine secretion: Possible role of PKC | |
P478 | volume | 292 |
Q33917121 | Burn serum causes a CD14-dependent mitochondrial damage in primary cardiomyocytes |
Q43748846 | Burn-induced apoptosis of cardiomyocytes is survivin dependent and regulated by PI3K/Akt, p38 MAPK and ERK pathways |
Q26771250 | Cardiovascular Dysfunction Following Burn Injury: What We Have Learned from Rat and Mouse Models |
Q37414220 | Estrogen treatment following severe burn injury reduces brain inflammation and apoptotic signaling |
Q54567860 | Insulin-mediated inhibition of p38 mitogen-activated protein kinase protects cardiomyocytes in severe burns. |
Q99549050 | Pathological Responses of Cardiac Mitochondria to Burn Trauma |
Q48171562 | Role of Small Interfering RNA Silencing Protein Kinase C-α Gene on the Occurrence of Ultrafiltration Failure in Peritoneal Dialysis Rats. |
Q37595301 | α₁ adrenoceptor activation by norepinephrine inhibits LPS-induced cardiomyocyte TNF-α production via modulating ERK1/2 and NF-κB pathway |
Search more.