| scholarly article | Q13442814 |
| P50 | author | Andrea L McGinley | Q87419147 |
| P2093 | author name string | Q Tian Wang | |
| Zane Deliu | |||
| Yanyang Li | |||
| P2860 | cites work | ASXL1 mutations promote myeloid transformation through loss of PRC2-mediated gene repression | Q24296658 |
| Histone H2A deubiquitinase activity of the Polycomb repressive complex PR-DUB | Q24309403 | ||
| SWI/SNF complexes containing Brahma or Brahma-related gene 1 play distinct roles in smooth muscle development | Q24634169 | ||
| Epigenetic mechanisms in cardiac development and disease | Q26853290 | ||
| Trials with 'epigenetic' drugs: an update | Q27000542 | ||
| Tnni3k modifies disease progression in murine models of cardiomyopathy | Q27349094 | ||
| A human homolog of Additional sex combs, ADDITIONAL SEX COMBS-LIKE 1, maps to chromosome 20q11 | Q28186334 | ||
| Functional conservation of Asxl2, a murine homolog for the Drosophila enhancer of trithorax and polycomb group gene Asx | Q28504707 | ||
| Thyroid transcription factor-1, hepatocyte nuclear factor-3beta, surfactant protein B, C, and Clara cell secretory protein in developing mouse lung | Q28506850 | ||
| Spectrum of heart disease associated with murine and human GATA4 mutation | Q28509800 | ||
| Maintenance of adult cardiac function requires the chromatin factor Asxl2 | Q28511761 | ||
| Chronic activation of the prostaglandin receptor EP4 promotes hyaluronan-mediated neointimal formation in the ductus arteriosus | Q28581731 | ||
| Loss-of-function Additional sex combs like 1 mutations disrupt hematopoiesis but do not cause severe myelodysplasia or leukemia | Q28585869 | ||
| T1alpha, a lung type I cell differentiation gene, is required for normal lung cell proliferation and alveolus formation at birth | Q28587220 | ||
| A murine model of Holt-Oram syndrome defines roles of the T-box transcription factor Tbx5 in cardiogenesis and disease | Q28594121 | ||
| Mesenchymal nuclear factor I B regulates cell proliferation and epithelial differentiation during lung maturation | Q28594779 | ||
| Cardiac hypertrophy: the good, the bad, and the ugly | Q29615247 | ||
| Epigenetics: a landscape takes shape | Q29616623 | ||
| Transcription factor pathways and congenital heart disease. | Q37997218 | ||
| The prevalence of adult congenital heart disease, results from a systematic review and evidence based calculation | Q38052187 | ||
| Multiple binding of methyl-CpG and polycomb proteins in long-term gene silencing events | Q40203326 | ||
| A prospective study to assess the frequency of familial clustering of congenital bicuspid aortic valve | Q40874790 | ||
| Polycomb repressive complex 2 regulates normal development of the mouse heart | Q41259507 | ||
| Genetics of congenital heart disease | Q41451951 | ||
| Epigenetic repression of cardiac progenitor gene expression by Ezh2 is required for postnatal cardiac homeostasis. | Q41811528 | ||
| Combined mutation screening of NKX2-5, GATA4, and TBX5 in congenital heart disease: multiple heterozygosity and novel mutations | Q41892668 | ||
| Heart malformation: what are the chances it could happen again? | Q42874167 | ||
| Cardiac fibroblasts regulate myocardial proliferation through beta1 integrin signaling | Q43152071 | ||
| Patterns of recurrence of congenital heart disease: an analysis of 6,640 consecutive pregnancies evaluated by detailed fetal echocardiography | Q47644393 | ||
| Mutations of polycomb-associated gene ASXL1 in myelodysplastic syndromes and chronic myelomonocytic leukaemia. | Q47956567 | ||
| Recurrence risks in offspring of adults with major heart defects: results from first cohort of British collaborative study | Q50878628 | ||
| Fetal programming of adult disease: implications for prenatal care. | Q51878301 | ||
| Molecular markers of cardiac endocardial cushion development. | Q52096947 | ||
| Formation of the atrioventricular septal structures in the normal mouse. | Q52188928 | ||
| Two novel patients with Bohring-Opitz syndrome caused by de novo ASXL1 mutations. | Q55055822 | ||
| De novo nonsense mutations in ASXL1 cause Bohring-Opitz syndrome | Q55671561 | ||
| Recurrence of Congenital Heart Defects in Families | Q63353105 | ||
| Kinetics of pulmonary epithelial proliferation during prenatal growth of the mouse lung | Q67347673 | ||
| jumonji downregulates cardiac cell proliferation by repressing cyclin D1 expression | Q73642456 | ||
| Ventricular septal defects | Q79354602 | ||
| The spectrum of cardiovascular anomalies in CHF1/Hey2 deficient mice reveals roles in endocardial cushion, myocardial and vascular maturation | Q81387718 | ||
| An optimized procedure for whole-mount in situ hybridization on mouse embryos and embryoid bodies | Q81577763 | ||
| ??? | Q28304085 | ||
| Deletion of Asxl1 results in myelodysplasia and severe developmental defects in vivo | Q30413034 | ||
| Platelets contribute to postnatal occlusion of the ductus arteriosus | Q33387350 | ||
| Clinical effect of point mutations in myelodysplastic syndromes | Q33395996 | ||
| Identification of a cardiac disease modifier gene using forward genetics in the mouse | Q33504526 | ||
| Lunatic Fringe-mediated Notch signaling is required for lung alveogenesis | Q33589433 | ||
| Additional sex combs-like 1 belongs to the enhancer of trithorax and polycomb group and genetically interacts with Cbx2 in mice | Q33595603 | ||
| Failure of postnatal ductus arteriosus closure in prostaglandin transporter-deficient mice | Q33720481 | ||
| The molecular basis of lung morphogenesis | Q33855377 | ||
| Genetic dissection of cardiac growth control pathways | Q33938724 | ||
| Congenital heart disease and genetic syndromes: specific correlation between cardiac phenotype and genotype. | Q34123316 | ||
| Bohring-Opitz (Oberklaid-Danks) syndrome: clinical study, review of the literature, and discussion of possible pathogenesis | Q34168200 | ||
| Heterogeneity of genetic modifiers ensures normal cardiac development | Q34193363 | ||
| Epigenetic modifications of stem cells: a paradigm for the control of cardiac progenitor cells. | Q34224125 | ||
| Modifier genes and the plasticity of genetic networks in mice | Q34236664 | ||
| De novo truncating mutations in ASXL3 are associated with a novel clinical phenotype with similarities to Bohring-Opitz syndrome | Q34326370 | ||
| De novo frameshift mutation in ASXL3 in a patient with global developmental delay, microcephaly, and craniofacial anomalies | Q34371843 | ||
| Epigenetics and cardiovascular disease | Q34788746 | ||
| ACC/AHA 2008 guidelines for the management of adults with congenital heart disease: a report of the American College of Cardiology/American Heart Association Task Force on Practice Guidelines (Writing Committee to Develop Guidelines on the Managemen | Q34889917 | ||
| Additional sex combs-like 2 is required for polycomb repressive complex 2 binding at select targets | Q34989584 | ||
| Cyclooxygenase-1 and cyclooxygenase-2 in the mouse ductus arteriosus: individual activity and functional coupling with nitric oxide synthase | Q35045603 | ||
| Mutational spectrum analysis of chronic myelomonocytic leukemia includes genes associated with epigenetic regulation: UTX, EZH2, and DNMT3A. | Q35346433 | ||
| Transcription factor cascades in congenital heart malformation. | Q35601724 | ||
| Ductus arteriosus blood flow during first 48 hours of life | Q35604230 | ||
| Critical congenital heart disease--utility of routine screening for chromosomal and other extracardiac malformations | Q35784358 | ||
| Epigenetic regulation of cardiac development and function by polycomb group and trithorax group proteins | Q36185227 | ||
| A chemical method for fast and sensitive detection of DNA synthesis in vivo | Q36497275 | ||
| The molecular basis of congenital heart disease | Q36993761 | ||
| Functional and cancer genomics of ASXL family members | Q37040682 | ||
| Genetic insights into normal and abnormal heart development | Q37045291 | ||
| Interpreting neonatal lethal phenotypes in mouse mutants: insights into gene function and human diseases | Q37363610 | ||
| Loss of Asxl1 leads to myelodysplastic syndrome-like disease in mice | Q37512080 | ||
| Loss of the tumor suppressor BAP1 causes myeloid transformation | Q37545704 | ||
| Epigenetic modifications as therapeutic targets | Q37800011 | ||
| Epigenetic factors and cardiac development | Q37878713 | ||
| Epigenetics and cardiovascular development. | Q37950482 | ||
| P433 | issue | 7 | |
| P921 | main subject | circulatory system | Q11068 |
| ASXL transcriptional regulator 1 | Q21499098 | ||
| ASXL transcriptional regulator 2 | Q21499108 | ||
| P304 | page(s) | 671-686 | |
| P577 | publication date | 2014-07-01 | |
| P1433 | published in | Genesis | Q5532784 |
| P1476 | title | Additional sex combs-like family genes are required for normal cardiovascular development | |
| P478 | volume | 52 |
| Q59797105 | ASXL2 regulates hematopoiesis in mice and its deficiency promotes myeloid expansion |
| Q58615781 | Asxl1 exerts an antiproliferative effect on mouse lung maturation via epigenetic repression of the E2f1-Nmyc axis |
| Q34469971 | Functional proteomics of the epigenetic regulators ASXL1, ASXL2 and ASXL3: a convergence of proteomics and epigenetics for translational medicine |
| Q33560062 | Nkx2-5 and Sarcospan genetically interact in the development of the muscular ventricular septum of the heart |
| Q38928350 | The Role of Additional Sex Combs-Like Proteins in Cancer |
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