scholarly article | Q13442814 |
P356 | DOI | 10.1084/JEM.20022190 |
P953 | full work available at URL | https://rupress.org/jem/article-pdf/197/10/1377/1142145/jem197101377.pdf |
https://europepmc.org/articles/PMC2193780 | ||
https://europepmc.org/articles/PMC2193780?pdf=render | ||
https://rupress.org/jem/article-pdf/197/10/1377/1707974/jem197101377.pdf | ||
P932 | PMC publication ID | 2193780 |
P698 | PubMed publication ID | 12743174 |
P5875 | ResearchGate publication ID | 10763646 |
P50 | author | Carol E. Schrader | Q41870482 |
P2093 | author name string | Janet Stavnezer | |
Joycelyn Vardo | |||
P2860 | cites work | Cancer Research | Q326097 |
Activation-induced cytidine deaminase (AID) deficiency causes the autosomal recessive form of the Hyper-IgM syndrome (HIGM2) | Q24290325 | ||
Structure and function of the N-terminal 40 kDa fragment of human PMS2: a monomeric GHL ATPase | Q27635053 | ||
High affinity cooperative DNA binding by the yeast Mlh1-Pms1 heterodimer | Q27936605 | ||
Eukaryotic DNA mismatch repair | Q27939116 | ||
AID mutates E. coli suggesting a DNA deamination mechanism for antibody diversification | Q28208979 | ||
Involvement of mouse Mlh1 in DNA mismatch repair and meiotic crossing over | Q28282791 | ||
AID is required to initiate Nbs1/gamma-H2AX focus formation and mutations at sites of class switching | Q28366125 | ||
Hot spot focusing of somatic hypermutation in MSH2-deficient mice suggests two stages of mutational targeting | Q28506440 | ||
Role for mismatch repair proteins Msh2, Mlh1, and Pms2 in immunoglobulin class switching shown by sequence analysis of recombination junctions | Q28513507 | ||
Reduced isotype switching in splenic B cells from mice deficient in mismatch repair enzymes | Q28585862 | ||
Different mismatch repair deficiencies all have the same effects on somatic hypermutation: intact primary mechanism accompanied by secondary modifications | Q28586687 | ||
Class switch recombination and hypermutation require activation-induced cytidine deaminase (AID), a potential RNA editing enzyme | Q29547201 | ||
Switch junction sequences in PMS2-deficient mice reveal a microhomology-mediated mechanism of Ig class switch recombination. | Q33952028 | ||
The many faces of mismatch repair in meiosis | Q33984289 | ||
Roles for mismatch repair factors in regulating genetic recombination | Q34057743 | ||
MSH2 deficiency contributes to accelerated APC-mediated intestinal tumorigenesis | Q34386079 | ||
DNA sequences at immunoglobulin switch region recombination sites | Q34963864 | ||
Immunoglobulin genes: generating diversity with AID and UNG. | Q34990524 | ||
DNA-dependent protein kinase activity is not required for immunoglobulin class switching | Q36371923 | ||
Ku70 is required for late B cell development and immunoglobulin heavy chain class switching | Q36401127 | ||
Two new isotype-specific switching activities detected for Ig class switching | Q36709872 | ||
Mutations, duplication, and deletion of recombined switch regions suggest a role for DNA replication in the immunoglobulin heavy-chain switch | Q36776415 | ||
Mlh1 is unique among mismatch repair proteins in its ability to promote crossing-over during meiosis | Q38344945 | ||
Two pathways for removal of nonhomologous DNA ends during double-strand break repair in Saccharomyces cerevisiae | Q40023936 | ||
Antibody class switching | Q41134290 | ||
Ku80 is required for immunoglobulin isotype switching | Q41821763 | ||
Deficiency in Msh2 affects the efficiency and local sequence specificity of immunoglobulin class-switch recombination: parallels with somatic hypermutation. | Q42676262 | ||
The AID enzyme induces class switch recombination in fibroblasts | Q42818062 | ||
Immunoglobulin isotype switching is inhibited and somatic hypermutation perturbed in UNG-deficient mice | Q44194766 | ||
IgH class switch recombination to IgG1 in DNA-PKcs-deficient B cells | Q77995587 | ||
Alternative end joining during switch recombination in patients with ataxia-telangiectasia | Q78022313 | ||
P433 | issue | 10 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | antibody | Q79460 |
immunology | Q101929 | ||
genetic recombination | Q211675 | ||
MutS homolog 2 | Q14911693 | ||
MutL homolog 1 | Q14911713 | ||
P304 | page(s) | 1377–1383 | |
P577 | publication date | 2003-05-12 | |
2003-05-19 | |||
P1433 | published in | Journal of Experimental Medicine | Q3186912 |
P1476 | title | Mlh1 can function in antibody class switch recombination independently of Msh2 | |
Mlh1 Can Function in Antibody Class Switch Recombination Independently of Msh2 | |||
P478 | volume | 197 |
Q36211150 | A role for the MutL mismatch repair Mlh3 protein in immunoglobulin class switch DNA recombination and somatic hypermutation |
Q34118000 | AID and somatic hypermutation. |
Q36173942 | AIDing antibody diversity by error-prone mismatch repair |
Q36954874 | Activities of human exonuclease 1 that promote cleavage of transcribed immunoglobulin switch regions |
Q28513971 | Altered somatic hypermutation and reduced class-switch recombination in exonuclease 1-mutant mice |
Q36231123 | Antibody class switch recombination: roles for switch sequences and mismatch repair proteins |
Q36540495 | Antibody diversification caused by disrupted mismatch repair and promiscuous DNA polymerases |
Q28269740 | Class-switch recombination: interplay of transcription, DNA deamination and DNA repair |
Q36316915 | Combinatorial mechanisms regulating AID-dependent DNA deamination: interacting proteins and post-translational modifications |
Q36015019 | DNA deamination in immunity |
Q36229513 | DNA polymerase beta is able to repair breaks in switch regions and plays an inhibitory role during immunoglobulin class switch recombination |
Q34595025 | DNA polymerase eta is the sole contributor of A/T modifications during immunoglobulin gene hypermutation in the mouse |
Q28508643 | Deletion of the nucleotide excision repair gene Ercc1 reduces immunoglobulin class switching and alters mutations near switch recombination junctions |
Q28589828 | Examination of Msh6- and Msh3-deficient mice in class switching reveals overlapping and distinct roles of MutS homologues in antibody diversification |
Q35671715 | Human models of inherited immunoglobulin class switch recombination and somatic hypermutation defects (hyper-IgM syndromes). |
Q38735124 | MBD4 Facilitates Immunoglobulin Class Switch Recombination |
Q36403873 | Mapping of a functional recombination motif that defines isotype specificity for mu-->gamma3 switch recombination implicates NF-kappaB p50 as the isotype-specific switching factor |
Q36037736 | Mechanism and control of V(D)J recombination versus class switch recombination: similarities and differences |
Q34773522 | Mismatch repair proteins MSH2, MLH1, and EXO1 are important for class-switch recombination events occurring in B cells that lack nonhomologous end joining |
Q35605313 | Mismatch-mediated error prone repair at the immunoglobulin genes |
Q28586272 | Msh2 ATPase activity is essential for somatic hypermutation at a-T basepairs and for efficient class switch recombination |
Q44600123 | Mutations in AID and UNG extend the function of AID |
Q40240123 | Mutations occur in the Ig Smu region but rarely in Sgamma regions prior to class switch recombination |
Q35576430 | Nucleic acid structures and enzymes in the immunoglobulin class switch recombination mechanism |
Q34068165 | PMS2 endonuclease activity has distinct biological functions and is essential for genome maintenance |
Q37106800 | Pol zeta ablation in B cells impairs the germinal center reaction, class switch recombination, DNA break repair, and genome stability |
Q34115234 | Regulation of immunoglobulin class-switch recombination: choreography of noncoding transcription, targeted DNA deamination, and long-range DNA repair |
Q53589401 | Requirement of non-canonical activity of uracil DNA glycosylase for class switch recombination. |
Q35830410 | The role of the non-homologous end-joining pathway in lymphocyte development |
Q36945144 | Ubiquitylated PCNA plays a role in somatic hypermutation and class-switch recombination and is required for meiotic progression |
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