| scholarly article | Q13442814 |
| P819 | ADS bibcode | 2009PLoSO...4.4291M |
| P356 | DOI | 10.1371/JOURNAL.PONE.0004291 |
| P932 | PMC publication ID | 2627930 |
| P698 | PubMed publication ID | 19172181 |
| P5875 | ResearchGate publication ID | 23951320 |
| P50 | author | Rachel Wevrick | Q46130607 |
| P2093 | author name string | Rebecca E Mercer | |
| P2860 | cites work | Deletion of the MBII-85 snoRNA gene cluster in mice results in postnatal growth retardation | Q21092489 |
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| Expression and imprinting of MAGEL2 suggest a role in Prader-willi syndrome and the homologous murine imprinting phenotype | Q47841909 | ||
| The human MAGEL2 gene and its mouse homologue are paternally expressed and mapped to the Prader-Willi region | Q47912961 | ||
| Prader-Willi syndrome transcripts are expressed in phenotypically significant regions of the developing mouse brain | Q48199393 | ||
| Sexual incentive motivation, olfactory preference, and activation of the vomeronasal projection pathway by sexually relevant cues in non-copulating and naive male rats | Q48527826 | ||
| Disruption of the mouse Necdin gene results in hypothalamic and behavioral alterations reminiscent of the human Prader-Willi syndrome. | Q49155367 | ||
| The imprinted gene Magel2 regulates normal circadian output. | Q51976832 | ||
| Developmental and reproductive performance in circadian mutant mice. | Q54510589 | ||
| Inactivation of the mouse Magel2 gene results in growth abnormalities similar to Prader-Willi syndrome | Q55880070 | ||
| Determining the Stage of the Estrous Cycle in the Mouse by the Appearance of the Vagina | Q69580554 | ||
| Imminent oocyte exhaustion and reduced follicular recruitment mark the transition to acyclicity in aging C57BL/6J mice | Q71702598 | ||
| Follicle stimulating and leutinizing hormones, estradiol and testosterone in Prader-Willi syndrome | Q80609343 | ||
| P275 | copyright license | Creative Commons Attribution 4.0 International | Q20007257 |
| P6216 | copyright status | copyrighted | Q50423863 |
| P433 | issue | 1 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | Prader–Willi syndrome | Q594013 |
| P304 | page(s) | e4291 | |
| P577 | publication date | 2009-01-27 | |
| P1433 | published in | PLOS One | Q564954 |
| P1476 | title | Loss of magel2, a candidate gene for features of Prader-Willi syndrome, impairs reproductive function in mice | |
| P478 | volume | 4 |
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| Q44879185 | Dysfunctional oleoylethanolamide signaling in a mouse model of Prader-Willi syndrome |
| Q99402564 | Emerging Roles of the MAGE Protein Family in Stress Response Pathways |
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| Q28074219 | Genetics of Prader-Willi syndrome and Prader-Will-Like syndrome |
| Q36684836 | Genome-wide coexpression of steroid receptors in the mouse brain: Identifying signaling pathways and functionally coordinated regions |
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| Q35416510 | Impaired hypothalamic regulation of endocrine function and delayed counterregulatory response to hypoglycemia in Magel2-null mice. |
| Q42603373 | Imprinting analysis of porcine MAGEL2 gene in two fetal stages and association analysis with carcass traits |
| Q37909236 | Is there a genomically imprinted social brain? |
| Q37590281 | Leptin signaling defects in a mouse model of Prader-Willi syndrome: An orphan genetic obesity syndrome no more? |
| Q48418722 | Loss of Maged1 results in obesity, deficits of social interactions, impaired sexual behavior and severe alteration of mature oxytocin production in the hypothalamus |
| Q28485151 | Magel2 is required for leptin-mediated depolarization of POMC neurons in the hypothalamic arcuate nucleus in mice |
| Q47357118 | Magel2 knockout mice manifest altered social phenotypes and a deficit in preference for social novelty. |
| Q28506430 | Magel2, a Prader-Willi syndrome candidate gene, modulates the activities of circadian rhythm proteins in cultured cells |
| Q51629774 | Muscle dysfunction caused by loss of Magel2 in a mouse model of Prader-Willi and Schaaf-Yang syndromes. |
| Q90028444 | Mutations in MAGEL2 and L1CAM Are Associated With Congenital Hypopituitarism and Arthrogryposis |
| Q37803582 | Neurobehavioral phenotype in Prader–Willi syndrome |
| Q37445958 | New Perspectives on Genomic Imprinting, an Essential and Multifaceted Mode of Epigenetic Control in the Developing and Adult Brain |
| Q37725825 | Parent-of-origin and trans-generational germline influences on behavioral development: the interacting roles of mothers, fathers, and grandparents |
| Q42058938 | Prader-Willi Syndrome and Schaaf-Yang Syndrome: Neurodevelopmental Diseases Intersecting at the MAGEL2 Gene |
| Q48201315 | Progressive postnatal decline in leptin sensitivity of arcuate hypothalamic neurons in the Magel2-null mouse model of Prader-Willi syndrome |
| Q36625658 | RNAs of the human chromosome 15q11-q13 imprinted region |
| Q44803556 | Recommendations for the investigation of animal models of Prader-Willi syndrome |
| Q47290627 | Sleeve gastrectomy leads to weight loss in the Magel2 knockout mouse |
| Q47706180 | The Prader-Willi syndrome proteins MAGEL2 and necdin regulate leptin receptor cell surface abundance through ubiquitination pathways |
| Q35680716 | The neurobiology of mouse models syntenic to human chromosome 15q. |
| Q89747418 | Translational outcomes in a full gene deletion of ubiquitin protein ligase E3A rat model of Angelman syndrome |
| Q37284354 | Truncating mutations of MAGEL2 cause Prader-Willi phenotypes and autism |
| Q28607464 | USP7 Acts as a Molecular Rheostat to Promote WASH-Dependent Endosomal Protein Recycling and Is Mutated in a Human Neurodevelopmental Disorder |
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