| scholarly article | Q13442814 |
| P2093 | author name string | Christopher D Link | |
| Susan Wang | |||
| Shin Murakami | |||
| Hana Murakami | |||
| Kevin Machino | |||
| P2860 | cites work | Amyloid beta: multiple mechanisms of toxicity and only some protective effects? | Q27008826 |
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| Alzheimer's disease: genes, proteins, and therapy | Q28131806 | ||
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| Thermotaxis is a robust mechanism for thermoregulation in Caenorhabditis elegans nematodes | Q30566713 | ||
| Expression of human beta-amyloid peptide in transgenic Caenorhabditis elegans | Q33902367 | ||
| Cerebral amyloid angiopathies: a pathologic, biochemical, and genetic view | Q34268430 | ||
| Using mice to model Alzheimer's dementia: an overview of the clinical disease and the preclinical behavioral changes in 10 mouse models | Q34418781 | ||
| Drosophila melanogaster as a model organism for Alzheimer's disease | Q34464181 | ||
| β-Amyloid carrying the Dutch mutation has diverse effects on calpain-mediated toxicity in hippocampal neurons | Q35872520 | ||
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| Utility of an improved model of amyloid-beta (Aβ₁₋₄₂) toxicity in Caenorhabditis elegans for drug screening for Alzheimer's disease | Q36460071 | ||
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| Chronic high levels of the RCAN1-1 protein may promote neurodegeneration and Alzheimer disease | Q38078146 | ||
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| Alzheimer's disease drug discovery: in vivo screening using Caenorhabditis elegans as a model for β-amyloid peptide-induced toxicity | Q38139529 | ||
| Immunotherapy for the treatment of Alzheimer's disease: amyloid-β or tau, which is the right target? | Q38912261 | ||
| Thermotaxis in Caenorhabditis elegans analyzed by measuring responses to defined Thermal stimuli. | Q43030766 | ||
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| Amyloid fibril protein nomenclature: 2012 recommendations from the Nomenclature Committee of the International Society of Amyloidosis | Q46569493 | ||
| Aging-dependent and -independent modulation of associative learning behavior by insulin/insulin-like growth factor-1 signal in Caenorhabditis elegans. | Q48942620 | ||
| Association between frailty and dementia: a population-based study. | Q50694550 | ||
| Manipulation of serotonin signal suppresses early phase of behavioral aging in Caenorhabditis elegans. | Q51992813 | ||
| P921 | main subject | Caenorhabditis elegans | Q91703 |
| overexpression | Q61643320 | ||
| P304 | page(s) | 202 | |
| P577 | publication date | 2014-07-04 | |
| P1433 | published in | Frontiers in Genetics | Q2499875 |
| P1476 | title | A semi-automated motion-tracking analysis of locomotion speed in the C. elegans transgenics overexpressing beta-amyloid in neurons | |
| P478 | volume | 5 |
| Q34010229 | Alzheimer's patient feedback to complement research using model systems for cognitive aging and dementia |
| Q58775193 | D-Track-A semi-automatic 3D video-tracking technique to analyse movements and routines of aquatic animals with application to captive dolphins |
| Q59808737 | Frailty and as a Benchtop Animal Model for Screening Drugs Including Natural Herbs |
| Q41497184 | In vitro aggregating β-lactamase-polyQ chimeras do not induce toxic effects in an in vivo Caenorhabditis elegans model. |
| Q51533022 | Metformin Attenuates Aβ Pathology Mediated Through Levamisole Sensitive Nicotinic Acetylcholine Receptors in a C. elegans Model of Alzheimer's Disease. |
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