| scholarly article | Q13442814 |
| P50 | author | Katherine Huppler Hullsiek | Q90044868 |
| John T. Brooks | Q93069508 | ||
| Howard N Hodis | Q114358595 | ||
| Irini Sereti | Q37383379 | ||
| Eleanor Wilson | Q40899810 | ||
| P2093 | author name string | Jason V Baker | |
| Keith Henry | |||
| Pragna Patel | |||
| Matt Budoff | |||
| Nur Onen | |||
| Amrit Singh | |||
| Erna Kojic | |||
| Ken Lichtenstein | |||
| CDC SUN Study Investigators | |||
| P2860 | cites work | Microbial translocation is a cause of systemic immune activation in chronic HIV infection | Q22251054 |
| HIV infection and the risk of acute myocardial infarction | Q27347231 | ||
| Executive Summary of The Third Report of The National Cholesterol Education Program (NCEP) Expert Panel on Detection, Evaluation, And Treatment of High Blood Cholesterol In Adults (Adult Treatment Panel III) | Q27860959 | ||
| Inflammation, coagulation and cardiovascular disease in HIV-infected individuals | Q28483645 | ||
| CD4+ count-guided interruption of antiretroviral treatment | Q29619489 | ||
| Risk of myocardial infarction in patients with HIV infection exposed to specific individual antiretroviral drugs from the 3 major drug classes: the data collection on adverse events of anti-HIV drugs (D:A:D) study | Q33521080 | ||
| Markers of inflammation, coagulation, and renal function are elevated in adults with HIV infection | Q33857515 | ||
| Traditional risk factors and D-dimer predict incident cardiovascular disease events in chronic HIV infection | Q33907642 | ||
| CX(3)CR1 and vascular adhesion protein-1-dependent recruitment of CD16(+) monocytes across human liver sinusoidal endothelium | Q34056276 | ||
| Nomenclature of monocytes and dendritic cells in blood. | Q34125860 | ||
| Coronary artery calcium score and risk classification for coronary heart disease prediction | Q34552221 | ||
| The CD16+ Monocyte Subset Is More Permissive to Infection and Preferentially Harbors HIV-1 In Vivo | Q58003915 | ||
| Coronary artery calcium area by electron-beam computed tomography and coronary atherosclerotic plaque area. A histopathologic correlative study | Q71730400 | ||
| Elevated CD38 antigen expression on CD8+ T cells is a stronger marker for the risk of chronic HIV disease progression to AIDS and death in the Multicenter AIDS Cohort Study than CD4+ cell count, soluble immune activation markers, or combinations of | Q73848661 | ||
| HIV infection induces age-related changes to monocytes and innate immune activation in young men that persist despite combination antiretroviral therapy | Q83399154 | ||
| The Monocyte in Atherosclerosis — Should I Stay or Should I Go Now? | Q84056093 | ||
| Diversity: A Hallmark of Monocyte Society | Q85092283 | ||
| Coronary artery calcification is an independent stroke predictor in the general population | Q86222605 | ||
| Association of Immunologic and Virologic Factors With Myocardial Infarction Rates in a US Healthcare System | Q34564981 | ||
| Age-associated changes in monocyte and innate immune activation markers occur more rapidly in HIV infected women | Q34568436 | ||
| Assessment of coronary artery disease by cardiac computed tomography: a scientific statement from the American Heart Association Committee on Cardiovascular Imaging and Intervention, Council on Cardiovascular Radiology and Intervention, and Committe | Q34570916 | ||
| The CD14+ CD16+ blood monocytes: their role in infection and inflammation | Q34585882 | ||
| Plasma levels of soluble CD14 independently predict mortality in HIV infection | Q34764002 | ||
| T cell activation and senescence predict subclinical carotid artery disease in HIV-infected women | Q34764361 | ||
| The macrophage: the intersection between HIV infection and atherosclerosis. | Q34898069 | ||
| T cell activation predicts carotid artery stiffness among HIV-infected women | Q35113323 | ||
| Progression of Carotid Intima-Media Thickness in a Contemporary Human Immunodeficiency Virus Cohort | Q35215672 | ||
| Soluble CD163, a novel marker of activated macrophages, is elevated and associated with noncalcified coronary plaque in HIV-infected patients | Q35465943 | ||
| Relationship between T cell activation and CD4+ T cell count in HIV-seropositive individuals with undetectable plasma HIV RNA levels in the absence of therapy | Q36304297 | ||
| Biomarkers of microbial translocation and macrophage activation: association with progression of subclinical atherosclerosis in HIV-1 infection | Q36330954 | ||
| Fractalkine preferentially mediates arrest and migration of CD16+ monocytes | Q36370923 | ||
| HIV status, burden of comorbid disease, and biomarkers of inflammation, altered coagulation, and monocyte activation | Q36383621 | ||
| Shared monocyte subset phenotypes in HIV-1 infection and in uninfected subjects with acute coronary syndrome | Q36440077 | ||
| From vulnerable plaque to vulnerable patient--Part III: Executive summary of the Screening for Heart Attack Prevention and Education (SHAPE) Task Force report | Q36537343 | ||
| Markers of inflammation and CD8 T-cell activation, but not monocyte activation, are associated with subclinical carotid artery disease in HIV-infected individuals. | Q36807389 | ||
| Low CD4+ T-cell count as a major atherosclerosis risk factor in HIV-infected women and men. | Q37057982 | ||
| Association of C-reactive protein and HIV infection with acute myocardial infarction | Q37390630 | ||
| CD14++CD16+ monocytes independently predict cardiovascular events: a cohort study of 951 patients referred for elective coronary angiography | Q39549158 | ||
| Progression of coronary artery calcium predicts all-cause mortality | Q39804804 | ||
| Progression of coronary calcium on serial electron beam tomographic scanning is greater in patients with future myocardial infarction | Q40559178 | ||
| Low CD4+ T cell count is a risk factor for cardiovascular disease events in the HIV outpatient study | Q42992133 | ||
| The study to understand the natural history of HIV and AIDS in the era of effective therapy (SUN Study). | Q43705174 | ||
| The proinflammatory CD14+CD16+DR++ monocytes are a major source of TNF. | Q43925988 | ||
| Risk factors for coronary heart disease in patients treated for human immunodeficiency virus infection compared with the general population | Q44510170 | ||
| Risk factors for the progression of coronary artery calcification in asymptomatic subjects: results from the Multi-Ethnic Study of Atherosclerosis (MESA). | Q50804527 | ||
| An updated coronary risk profile. A statement for health professionals | Q52461335 | ||
| Quantification of coronary artery calcium using ultrafast computed tomography | Q52867038 | ||
| Evaluating changes in coronary artery calcium: an analytic method that accounts for interscan variability | Q52962053 | ||
| Atazanavir is not associated with an increased risk of cardio or cerebrovascular disease events | Q57180859 | ||
| Class of Antiretroviral Drugs and the Risk of Myocardial Infarction | Q57181081 | ||
| Serious Fatal and Nonfatal Non-AIDS-Defining Illnesses in Europe | Q57877234 | ||
| P433 | issue | 6 | |
| P407 | language of work or name | English | Q1860 |
| P304 | page(s) | 831-840 | |
| P577 | publication date | 2014-03-01 | |
| P1433 | published in | AIDS | Q4651863 |
| P1476 | title | Immunologic predictors of coronary artery calcium progression in a contemporary HIV cohort | |
| P478 | volume | 28 |
| Q91790635 | Activated dendritic cells and monocytes in HIV immunological nonresponders: HIV-induced interferon-inducible protein-10 correlates with low future CD4+ recovery |
| Q50001938 | Ageing with HIV. |
| Q35796695 | Altered Monocyte Phenotype in HIV-1 Infection Tends to Normalize with Integrase-Inhibitor-Based Antiretroviral Therapy |
| Q37543675 | Altered Monocyte and Endothelial Cell Adhesion Molecule Expression Is Linked to Vascular Inflammation in Human Immunodeficiency Virus Infection |
| Q46172140 | Antiretroviral therapy: stubborn limitations persist |
| Q35124958 | Assessment of arterial elasticity among HIV-positive participants with high CD4 cell counts: a substudy of the INSIGHT Strategic Timing of AntiRetroviral Treatment (START) trial |
| Q37643812 | Association of Arterial and Lymph Node Inflammation With Distinct Inflammatory Pathways in Human Immunodeficiency Virus Infection |
| Q35171099 | Brief report: Endothelial colony-forming cells and inflammatory monocytes in HIV. |
| Q51779710 | Cardiovascular Disease in the Setting of Human Immunodeficiency Virus Infection |
| Q35064091 | Cardiovascular disease risk in an aging HIV population: not just a question of biology. |
| Q36436569 | Changes in oxidized lipids drive the improvement in monocyte activation and vascular disease after statin therapy in HIV. |
| Q38352065 | Coronary artery disease in patients with HIV infection |
| Q37484834 | Early Antiretroviral Therapy at High CD4 Counts Does Not Improve Arterial Elasticity: A Substudy of the Strategic Timing of AntiRetroviral Treatment (START) Trial |
| Q36220304 | Elevated Soluble CD14 and Lower D-Dimer Are Associated With Cigarette Smoking and Heavy Episodic Alcohol Use in Persons Living With HIV. |
| Q91619134 | Endothelial Colony-Forming Cell Function Is Reduced During HIV Infection |
| Q36971255 | Extra-coronary calcification (aortic valve calcification, mitral annular calcification, aortic valve ring calcification and thoracic aortic calcification) in HIV seropositive and seronegative men: Multicenter AIDS Cohort Study. |
| Q39185675 | HIV Infection and Risk of Cardiovascular Diseases Beyond Coronary Artery Disease |
| Q34494208 | HIV vasculopathy: role of mononuclear cell-associated Krüppel-like factors 2 and 4. |
| Q51748805 | Heavy Cannabis Use Associated With Reduction in Activated and Inflammatory Immune Cell Frequencies in Antiretroviral Therapy-Treated Human Immunodeficiency Virus-Infected Individuals. |
| Q91638984 | How Monocytes Contribute to Increased Risk of Atherosclerosis in Virologically-Suppressed HIV-Positive Individuals Receiving Combination Antiretroviral Therapy |
| Q36556082 | Immune activation and cardiovascular disease in chronic HIV infection |
| Q36957752 | Inflammation, immune activation, and cardiovascular disease in HIV. |
| Q38601647 | Inflammatory monocytes expressing tissue factor drive SIV and HIV coagulopathy. |
| Q26851535 | Influence of immune activation and inflammatory response on cardiovascular risk associated with the human immunodeficiency virus |
| Q35005863 | JAM-A and ALCAM are therapeutic targets to inhibit diapedesis across the BBB of CD14+CD16+ monocytes in HIV-infected individuals |
| Q36024460 | Kidney Dysfunction and Markers of Inflammation in the Multicenter AIDS Cohort Study |
| Q90303159 | Loss of CXCR4 on non-classical monocytes in participants of the Women's Interagency HIV Study (WIHS) with subclinical atherosclerosis |
| Q92021923 | Monocyte Dysfunction, Activation, and Inflammation After Long-Term Antiretroviral Therapy in an African Cohort |
| Q52862162 | Monocyte activation and cardiovascular disease in HIV infection |
| Q34396067 | Monocyte-activation phenotypes are associated with biomarkers of inflammation and coagulation in chronic HIV infection |
| Q37378957 | Monocytes from HIV-infected individuals show impaired cholesterol efflux and increased foam cell formation after transendothelial migration |
| Q35921028 | Non-Classical Monocytes and Monocyte Chemoattractant Protein-1 (MCP-1) Correlate with Coronary Artery Calcium Progression in Chronically HIV-1 Infected Adults on Stable Antiretroviral Therapy |
| Q35655728 | Oxidized LDL Levels Are Increased in HIV Infection and May Drive Monocyte Activation. |
| Q92325712 | PD-1+ and TIGIT+ CD4 T Cells Are Associated With Coronary Artery Calcium Progression in HIV-Infected Treated Adults |
| Q37038775 | Pathophysiology and management of cardiovascular disease in patients with HIV |
| Q40675523 | Plasma IL-6 levels are independently associated with atherosclerosis and mortality in HIV-infected individuals on suppressive antiretroviral therapy |
| Q36087826 | Plasma Selenium Concentrations Are Sufficient and Associated with Protease Inhibitor Use in Treated HIV-Infected Adults |
| Q34288534 | Protease inhibitor monotherapy is associated with a higher level of monocyte activation, bacterial translocation and inflammation |
| Q46306524 | Reply to Cannizzo et al. |
| Q36571165 | Role of immune activation in progression to AIDS. |
| Q35102167 | Rosuvastatin reduces vascular inflammation and T-cell and monocyte activation in HIV-infected subjects on antiretroviral therapy |
| Q46136379 | Soluble CD163 and Clinical Outcomes in Treated HIV Infection: Insights Into Mechanisms |
| Q38985926 | Strategies to limit immune-activation in HIV patients. |
| Q47095325 | Systemic Inflammation, Coagulation, and Clinical Risk in the START Trial |
| Q40504028 | T-cell Activation and E-selectin Are Associated With Coronary Plaque in HIV-infected Young Adults |
| Q35030351 | The CD14 C-260T single nucleotide polymorphism (SNP) modulates monocyte/macrophage activation in treated HIV-infected individuals |
| Q46911047 | The Effect of Rosuvastatin on Vascular Disease Differs by Smoking Status in Treated HIV Infection |
| Q43505499 | The importance of monocytes and macrophages in HIV pathogenesis, treatment, and cure. |
| Q33917580 | Treatment of HIV infection with a raltegravir-based regimen increases LDL levels, but improves HDL cholesterol efflux capacity. |
| Q33626615 | Vitamin D deficiency is associated with IL-6 levels and monocyte activation in HIV-infected persons |
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