Cloning, expression and localization of human BM88 shows that it maps to chromosome 11p15.5, a region implicated in Beckwith-Wiedemann syndrome and tumorigenesis

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Cloning, expression and localization of human BM88 shows that it maps to chromosome 11p15.5, a region implicated in Beckwith-Wiedemann syndrome and tumorigenesis is …
instance of (P31):
scholarly articleQ13442814

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P932PMC publication ID1221787
P698PubMed publication ID11311134

P50authorFelice TironeQ37374268
P2093author name stringMatsas R
Mamalaki A
Georgopoulou N
Pappa C
Gaitanou M
Buanne P
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The BM88 antigen, a novel neuron-specific molecule, enhances the differentiation of mouse neuroblastoma cells.Q48072903
P433issuePt 3
P407language of work or nameEnglishQ1860
P921main subjectCell cycle exit and neuronal differentiation 1Q21131293
Beckwith-Wiedemann syndromeQ521863
P304page(s)715-724
P577publication date2001-05-01
P1433published inBiochemical JournalQ864221
P1476titleCloning, expression and localization of human BM88 shows that it maps to chromosome 11p15.5, a region implicated in Beckwith-Wiedemann syndrome and tumorigenesis
P478volume355

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cites work (P2860)
Q36140588BM88/CEND1 coordinates cell cycle exit and differentiation of neuronal precursors
Q66680403Cend1, a Story with Many Tales: From Regulation of Cell Cycle Progression/Exit of Neural Stem Cells to Brain Structure and Function
Q35060879Functional Interactions between BM88/Cend1, Ran-binding protein M and Dyrk1B kinase affect cyclin D1 levels and cell cycle progression/exit in mouse neuroblastoma cells
Q34017774The 4C5 cell-impermeable anti-HSP90 antibody with anti-cancer activity, is composed of a single light chain dimer

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