scholarly article | Q13442814 |
P2093 | author name string | Masaki Sakurai | |
Noboru Yoshioka | |||
Tsutomu Kamiyama | |||
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P433 | issue | 4 | |
P407 | language of work or name | English | Q1860 |
P304 | page(s) | 2304-2313 | |
P577 | publication date | 2005-11-02 | |
P1433 | published in | Journal of Neurophysiology | Q1709863 |
P1476 | title | Synapse elimination in the corticospinal projection during the early postnatal period | |
P478 | volume | 95 |
Q50255105 | A retrograde tracing study of compensatory corticospinal projections in rats with neonatal hemidecortication |
Q36864156 | Activity- and use-dependent plasticity of the developing corticospinal system |
Q82029129 | Chapter 3 Development of the corticospinal system and spinal motor circuits |
Q34834606 | Comprehensive analysis of neonatal versus adult unilateral decortication in a mouse model using behavioral, neuroanatomical, and DNA microarray approaches |
Q41184269 | Corticospinal axons make direct synaptic connections with spinal motoneurons innervating forearm muscles early during postnatal development in the rat. |
Q48233641 | Corticospinal tract development and spinal cord innervation differ between cervical and lumbar targets. |
Q64100324 | Glial Contribution to Excitatory and Inhibitory Synapse Loss in Neurodegeneration |
Q35187485 | Motor cortex maturation is associated with reductions in recurrent connectivity among functional subpopulations and increases in intrinsic excitability |
Q30416960 | Postnatal development of chorda tympani axons in the rat nucleus of the solitary tract |
Q46172975 | Regressive events in rat corticospinal axons during development in in vitro slice cocultures: retraction, amputation, and degeneration |
Q28749514 | Specific involvement of postsynaptic GluN2B-containing NMDA receptors in the developmental elimination of corticospinal synapses |
Q35782942 | The Cell Death Pathway Regulates Synapse Elimination through Cleavage of Gelsolin in Caenorhabditis elegans Neurons |
Q37291446 | The decline in synaptic GluN2B and rise in inhibitory neurotransmission determine the end of a critical period |
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