| scholarly article | Q13442814 |
| P50 | author | Thomas E. Van Dyke | Q55029890 |
| P2093 | author name string | L Shapira | |
| S Amar | |||
| C Champagne | |||
| P2860 | cites work | Factors in virulence expression and their role in periodontal disease pathogenesis | Q34015505 |
| Effects of a Porphyromonas gingivalis infection on inflammatory mediator response and pregnancy outcome in hamsters | Q34540639 | ||
| Protection against endotoxic shock and lipopolysaccharide-induced local inflammation by tetracycline: correlation with inhibition of cytokine secretion. | Q35473213 | ||
| The bacterial etiology of destructive periodontal disease: current concepts | Q36056602 | ||
| Detection of interleukin 8 and tumor necrosis factor in normal humans after intravenous endotoxin: the effect of antiinflammatory agents | Q36361007 | ||
| Lipopolysaccharide-induced selective priming effects on tumor necrosis factor alpha and nitric oxide production in mouse peritoneal macrophages | Q36361247 | ||
| A novel mouse model to study the virulence of and host response to Porphyromonas (Bacteroides) gingivalis | Q36962002 | ||
| Modulation of bone metabolism by two chemically distinct lipopolysaccharide fractions from Bacteroides gingivalis | Q37030896 | ||
| Biology of asaccharolytic black-pigmented Bacteroides species | Q37054903 | ||
| Inflammatory mediators and the destruction of bone | Q37101523 | ||
| Ability of bacteria associated with chronic inflammatory disease to stimulate E-selectin expression and promote neutrophil adhesion | Q39821679 | ||
| Porphyromonas gingivalis lipopolysaccharide is poorly recognized by molecular components of innate host defense in a mouse model of early inflammation | Q39823660 | ||
| Animal chamber models for study of host-parasite interactions | Q40687389 | ||
| Response of man to endotoxin | Q40825144 | ||
| The role of the host response in periodontal disease progression: implications for future treatment strategies | Q40864530 | ||
| The use of crevicular fluid prostaglandin E2 levels as a predictor of periodontal attachment loss | Q41358401 | ||
| Role of tumor necrosis factor-alpha in lipopolysaccharide-induced pathologic alterations. | Q42173056 | ||
| Serum levels of tumor necrosis factor determine the fatal or non-fatal course of endotoxic shock | Q43875895 | ||
| Time course of various inflammatory mediators during recurrent endotoxemia. | Q43893118 | ||
| The Presence and Biologic Activity of Cementum-Bound Endotoxin | Q43936715 | ||
| Fractions of lipopolysaccharide from Escherichia coli O111:B4 prepared by two extraction procedures | Q44588578 | ||
| Tissue levels of bone resorptive cytokines in periodontal disease | Q44714371 | ||
| Levels of interleukin 1 beta in tissue from sites of active periodontal disease | Q45210347 | ||
| Immunobiological activities of chemically defined lipid A from lipopolysaccharides of Porphyromonas gingivalis | Q50149141 | ||
| Immunobiological properties of chemically defined lipid A from lipopolysaccharide of Porphyromonas (Bacteroides) gingivalis | Q50152000 | ||
| Lipopolysaccharide-stimulated PGE2 release from human monocytes. Comparison of lipopolysaccharides prepared from suspected periodontal pathogens | Q50197282 | ||
| Immunomodulatory and superantigen activities of bacteria associated with adult periodontitis. | Q52521916 | ||
| The cardiovascular response of normal humans to the administration of endotoxin. | Q54346884 | ||
| Porphyromonas gingivalis lipopolysaccharide stimulation of human monocytes: dependence on serum and CD14 receptor. | Q54637219 | ||
| The secretion of PGE2, IL-1 beta, IL-6, and TNF alpha by adherent mononuclear cells from early onset periodontitis patients. | Q54641596 | ||
| Detection of circulating tumor necrosis factor after endotoxin administration. | Q54747335 | ||
| Lipopolysaccharide isolated from Porphyromonas gingivalis grown in hemin-limited chemostat conditions has a reduced capacity for human neutrophil priming. | Q64930188 | ||
| Endotoxin levels in periodontally healthy and diseased sites: correlation with levels of gram-negative bacteria | Q67594970 | ||
| Identity of limulus amoebocyte lysate-active root surface materials from periodontally involved teeth | Q68740284 | ||
| The distribution of bacterial lipopolysaccharide (endotoxin) in relation to periodontally involved root surfaces | Q68740286 | ||
| Lipopolysaccharide (endotoxin) from individual periodontally involved teeth | Q68773627 | ||
| Chemical characterization and biologic properties of lipopolysaccharide from Bacteroides gingivalis strains W50, W83, and ATCC 33277 | Q69476281 | ||
| Cytokine appearance in human endotoxemia and primate bacteremia | Q70371866 | ||
| Release of reactive nitrogen intermediates and reactive oxygen intermediates from mouse peritoneal macrophages. Comparison of activating cytokines and evidence for independent production | Q70405593 | ||
| Preliminary characterization of material eluted from the roots of periodontally diseased teeth | Q70526623 | ||
| Involvement of protein kinase C and protein tyrosine kinase in lipopolysaccharide-induced TNF-alpha and IL-1 beta production by human monocytes | Q71636818 | ||
| Stimulation of macromolecular synthesis by endotoxin-treated 3T6 fibroblasts | Q72852940 | ||
| Tumor necrosis factor (TNF)-induced cutaneous necrosis is mediated by TNF receptor 1 | Q73325200 | ||
| Increased interleukin-1 beta (IL-1 beta) concentration in gingival tissue from periodontitis patients | Q93529018 | ||
| P433 | issue | 6 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | lipopolysaccharide | Q421804 |
| Porphyromonas gingivalis | Q3214147 | ||
| P304 | page(s) | 2736-2742 | |
| P577 | publication date | 1998-06-01 | |
| P1433 | published in | Infection and Immunity | Q6029193 |
| P1476 | title | Strain-dependent activation of monocytes and inflammatory macrophages by lipopolysaccharide of Porphyromonas gingivalis | |
| P478 | volume | 66 |
| Q22009378 | A novel lipopolysaccharide-induced transcription factor regulating tumor necrosis factor alpha gene expression: molecular cloning, sequencing, characterization, and chromosomal assignment |
| Q95652048 | Alterations of peripheral gene expression in response to lipopolysaccharide-induced synovitis as a model for inflammation in horses |
| Q39061369 | DHA suppresses Prevotella intermedia lipopolysaccharide-induced production of proinflammatory mediators in murine macrophages |
| Q39852234 | Differential expression of immunoregulatory genes in monocytes in response to Porphyromonas gingivalis and Escherichia coli lipopolysaccharide. |
| Q39521213 | Evidence of recombination in Porphyromonas gingivalis and random distribution of putative virulence markers |
| Q41148300 | Immune response of macrophages from young and aged mice to the oral pathogenic bacterium Porphyromonas gingivalis |
| Q40003727 | In vitro expression of tumor necrosis factor-alpha, interleukin 1beta, and interleukin 8 mRNA by bovine macrophages following exposure to Porphyromonas levii. |
| Q37204278 | Interaction between periodontitis and liver diseases |
| Q96769046 | Is periodontal disease a risk factor for severe COVID-19 illness? |
| Q34010067 | Life below the gum line: pathogenic mechanisms of Porphyromonas gingivalis |
| Q37033446 | Metabolic network model of a human oral pathogen. |
| Q28590692 | Mice lacking inducible nitric oxide synthase demonstrate impaired killing of Porphyromonas gingivalis |
| Q34132296 | Modulation of an interleukin-12 and gamma interferon synergistic feedback regulatory cycle of T-cell and monocyte cocultures by Porphyromonas gingivalis lipopolysaccharide in the absence or presence of cysteine proteinases |
| Q36726966 | Movement-evoked hyperalgesia induced by lipopolysaccharides is not suppressed by glucocorticoids |
| Q37611976 | NADPH Oxidase Contributes to Resistance against Aggregatibacter actinomycetemcomitans-Induced Periodontitis in Mice |
| Q37704915 | Periodontitis as a risk factor of atherosclerosis |
| Q37099426 | Porphyromonas gingivalis RgpA-Kgp proteinase-adhesin complexes penetrate gingival tissue and induce proinflammatory cytokines or apoptosis in a concentration-dependent manner |
| Q37546378 | Porphyromonas gingivalis lipid A phosphatase activity is critical for colonization and increasing the commensal load in the rabbit ligature model |
| Q39812114 | SOCS-3 Regulates Alveolar Bone Loss in Experimental Periodontitis |
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