| scholarly article | Q13442814 |
| P2093 | author name string | W C Hop | |
| A Eerenberg | |||
| C E Hack | |||
| E van der Voort | |||
| G van Mierlo | |||
| J A Hazelzet | |||
| K F Joosten | |||
| M H Suur | |||
| R de Groot | |||
| P2860 | cites work | Neutrophil-mediated injury to endothelial cells. Enhancement by endotoxin and essential role of neutrophil elastase | Q28361648 |
| Pediatric risk of mortality (PRISM) score | Q34164023 | ||
| Initial studies on the administration of C1-esterase inhibitor to patients with septic shock or with a vascular leak syndrome induced by interleukin-2 therapy. | Q34662427 | ||
| Interplay of complement and cytokines in the pathogenesis of septic shock | Q35636407 | ||
| Inhibitors of complement and neutrophils: a critical evaluation of their role in the treatment of sepsis | Q36170123 | ||
| Interaction of complement with Neisseria meningitidis and Neisseria gonorrhoeae | Q36633889 | ||
| Neutrophil-mediated vascular injury in shock and multiple organ failure. | Q38636315 | ||
| Activation of the complement system in baboons challenged with live Escherichia coli: correlation with mortality and evidence for a biphasic activation pattern | Q40269037 | ||
| Endothelial cell-to-cell junctions. Structural characteristics and functional role in the regulation of vascular permeability and leukocyte extravasation | Q40668538 | ||
| Influence of C1-inhibitor on inflammation, edema and shock | Q40747685 | ||
| Complement deficiency states and meningococcal disease | Q40804745 | ||
| The role of endothelial cells in the systemic inflammatory response syndrome and multiple system organ failure | Q40941906 | ||
| Proteolytic inactivation of plasma C1- inhibitor in sepsis | Q41076639 | ||
| C4b-binding protein exacerbates the host response to Escherichia coli | Q41152573 | ||
| TNF-alpha suppresses IL-1 alpha and IL-6 upregulation of C4b-binding protein in HepG-2 hepatoma cells | Q41230598 | ||
| A rate nephelometer for measuring specific proteins by immunoprecipitin reactions | Q41269427 | ||
| Group II phospholipase A2 in serum in critically ill surgical patients | Q44699906 | ||
| Elevated plasma levels of the anaphylatoxins C3a and C4a are associated with a fatal outcome in sepsis | Q45254421 | ||
| Age-related differences in outcome and severity of DIC in children with septic shock and purpura. | Q53836364 | ||
| Complement abnormalities during an epidemic of group B meningococcal infection in children. | Q53945338 | ||
| Hyperphospholipasemia A2 in human volunteers challenged with intravenous endotoxin | Q67577330 | ||
| Plasma elastase alpha 1-antitrypsin and lactoferrin in sepsis: evidence for neutrophils as mediators in fatal sepsis | Q67755145 | ||
| Protein C, protein S and C4b-binding protein in severe infection and septic shock | Q67900271 | ||
| A modified competitive inhibition radioimmunoassay for the detection of C3a. Use of 125I-C3 instead of 125I-C3a | Q68092050 | ||
| Complement activation and endotoxin levels in systemic meningococcal disease | Q69075766 | ||
| Low prevalence of complement deficiencies among patients with meningococcal disease in Norway | Q70652427 | ||
| Therapy with interleukin-2 induces the systemic release of phospholipase-A2 | Q70823137 | ||
| The excessive complement activation in fulminant meningococcal septicemia is predominantly caused by alternative pathway activation | Q71071247 | ||
| CRP-mediated activation of complement in vivo: assessment by measuring circulating complement-C-reactive protein complexes | Q71197109 | ||
| A highly variable region in members of the streptococcal M protein family binds the human complement regulator C4BP | Q71555615 | ||
| Binding of complement proteins C1q and C4bp to serum amyloid P component (SAP) in solid contra liquid phase | Q71623447 | ||
| Application of a monoclonal antibody against a neoepitope on activated C4 in an ELISA for the quantification of complement activation via the classical pathway | Q72088344 | ||
| C1 esterase inhibitor concentrate for capillary leakage syndrome following bone marrow transplantation | Q73877807 | ||
| P433 | issue | 11 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | sepsis | Q183134 |
| septic shock | Q1765564 | ||
| P304 | page(s) | 5350-5356 | |
| P577 | publication date | 1998-11-01 | |
| P1433 | published in | Infection and Immunity | Q6029193 |
| P1476 | title | Complement activation in relation to capillary leakage in children with septic shock and purpura | |
| P478 | volume | 66 |
| Q26784268 | Activated Complement Factors as Disease Markers for Sepsis |
| Q35357709 | Adjuvant treatment of severe acute pancreatitis with C1 esterase inhibitor concentrate after haematopoietic stem cell transplantation |
| Q35140175 | Administration of C1 inhibitor reduces neutrophil activation in patients with sepsis |
| Q48502611 | Auxiliary activation of the complement system and its importance for the pathophysiology of clinical conditions |
| Q34464961 | C1 inhibitor: molecular and clinical aspects |
| Q98771397 | Childhood Obesity, Endothelial Cell Activation, and Critical Illness |
| Q40883151 | Complement activation and complement-dependent inflammation by Neisseria meningitidis are independent of lipopolysaccharide |
| Q43593887 | Diffuse non-hydrostatic, non-hypooncotic edema in autoimmune lupus-like disorders |
| Q43579775 | Elastase and granzymes during meningococcal disease in children: correlation to disease severity |
| Q34644525 | Epidemic meningitis, meningococcaemia, and Neisseria meningitidis |
| Q57456315 | High levels of circulating GM-CSFCD4 T cells are predictive of poor outcomes in sepsis patients: a prospective cohort study |
| Q35211292 | Host genetic determinants of Neisseria meningitidis infections. |
| Q27472957 | Increased production of interleukin-8 in primary human monocytes and in human epithelial and endothelial cell lines after dengue virus challenge |
| Q36241437 | Meningococcal infections at the start of the 21st century. |
| Q33587385 | Neisseria meningitidis Opc invasin binds to the sulphated tyrosines of activated vitronectin to attach to and invade human brain endothelial cells |
| Q36845133 | Stages of meningococcal sepsis simulated in vitro, with emphasis on complement and Toll-like receptor activation |
| Q35144175 | The immunopathogenesis of meningococcal disease |
| Q27027974 | The role of complement system in septic shock |
| Q33812435 | Update on meningococcal disease with emphasis on pathogenesis and clinical management |
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