| scholarly article | Q13442814 |
| P2093 | author name string | Akabas MH | |
| P2860 | cites work | Cystic Fibrosis Heterozygote Resistance to Cholera Toxin in the Cystic Fibrosis Mouse Model | Q22299398 |
| A C-terminal motif found in the β 2 -adrenergic receptor, P2Y1 receptor and cystic fibrosis transmembrane conductance regulator determines binding to the Na + /H + exchanger regulatory factor family of PDZ proteins | Q24316901 | ||
| Perturbation of Hsp90 interaction with nascent CFTR prevents its maturation and accelerates its degradation by the proteasome | Q24533430 | ||
| Crystal structure of the ATP-binding subunit of an ABC transporter | Q27766403 | ||
| Identification of the cystic fibrosis gene: cloning and characterization of complementary DNA | Q28119124 | ||
| Regulation of CFTR chloride channels by syntaxin and Munc18 isoforms | Q28255553 | ||
| An Apical PDZ Protein Anchors the Cystic Fibrosis Transmembrane Conductance Regulator to the Cytoskeleton | Q28277503 | ||
| ATPase activity of the cystic fibrosis transmembrane conductance regulator | Q28854569 | ||
| Degradation of CFTR by the ubiquitin-proteasome pathway | Q29616142 | ||
| CFTR channel gating: incremental progress in irreversible steps. | Q33683078 | ||
| Identification of cystic fibrosis transmembrane conductance regulator channel-lining residues in and flanking the M6 membrane-spanning segment. | Q34017913 | ||
| Disulphonic stilbene block of cystic fibrosis transmembrane conductance regulator Cl- channels expressed in a mammalian cell line and its regulation by a critical pore residue | Q34408228 | ||
| Structural analysis of cloned plasma membrane proteins by freeze-fracture electron microscopy | Q36308819 | ||
| Functionally distinct phospho-forms underlie incremental activation of protein kinase-regulated Cl- conductance in mammalian heart | Q36411397 | ||
| Locating the anion-selectivity filter of the cystic fibrosis transmembrane conductance regulator (CFTR) chloride channel. | Q36411911 | ||
| Adenosine Triphosphate–dependent Asymmetry of Anion Permeation in the Cystic Fibrosis Transmembrane Conductance Regulator Chloride Channel | Q36411989 | ||
| Gating of cystic fibrosis transmembrane conductance regulator chloride channels by adenosine triphosphate hydrolysis. Quantitative analysis of a cyclic gating scheme | Q36412100 | ||
| CFTR: the nucleotide binding folds regulate the accessibility and stability of the activated state | Q36415724 | ||
| Halide Permeation in Wild-Type and Mutant Cystic Fibrosis Transmembrane Conductance Regulator Chloride Channels | Q36435908 | ||
| Permeability of Wild-Type and Mutant Cystic Fibrosis Transmembrane Conductance Regulator Chloride Channels to Polyatomic Anions | Q36435912 | ||
| Chloride channels of intracellular organelles. | Q40380567 | ||
| The Cystic Fibrosis Transmembrane Conductance Regulator | Q40486029 | ||
| Toxigenic Diarrheas, Congenital Diarrheas, and Cystic Fibrosis: Disorders of Intestinal Ion Transport | Q40486036 | ||
| Molecular mechanisms of CFTR chloride channel dysfunction in cystic fibrosis | Q40488178 | ||
| The genesis of cystic fibrosis lung disease | Q40848573 | ||
| Cystic fibrosis transmembrane conductance regulator regulates luminal Cl-/HCO3- exchange in mouse submandibular and pancreatic ducts | Q40954140 | ||
| A single conductance pore for chloride ions formed by two cystic fibrosis transmembrane conductance regulator molecules | Q40967502 | ||
| Novel pore-lining residues in CFTR that govern permeation and open-channel block | Q41022138 | ||
| Function of the R Domain in the Cystic Fibrosis Transmembrane Conductance Regulator Chloride Channel | Q41083500 | ||
| Failure of the cystic fibrosis transmembrane conductance regulator to conduct ATP. | Q41213451 | ||
| CFTR as a cAMP-dependent regulator of sodium channels | Q41312779 | ||
| Mutations in the cystic fibrosis gene in men with congenital bilateral absence of the vas deferens | Q41547475 | ||
| Distinct and specific functions of cGMP-dependent protein kinases | Q41576484 | ||
| Defective regulation of outwardly rectifying Cl- channels by protein kinase A corrected by insertion of CFTR | Q41608893 | ||
| Nucleoside triphosphates are required to open the CFTR chloride channel. | Q41769807 | ||
| Purified cystic fibrosis transmembrane conductance regulator (CFTR) does not function as an ATP channel | Q42064297 | ||
| Phosphorylation by protein kinase C is required for acute activation of cystic fibrosis transmembrane conductance regulator by protein kinase A. | Q42529107 | ||
| The two nucleotide-binding domains of cystic fibrosis transmembrane conductance regulator (CFTR) have distinct functions in controlling channel activity | Q42675011 | ||
| Chloride secretion in response to guanylin in colonic epithelial from normal and transgenic cystic fibrosis mice | Q42701115 | ||
| CFTR and outward rectifying chloride channels are distinct proteins with a regulatory relationship | Q42809539 | ||
| Demonstration That CFTR Is a Chloride Channel by Alteration of Its Anion Selectivity | Q43847410 | ||
| Conformational states of CFTR associated with channel gating: the role ATP binding and hydrolysis. | Q46017118 | ||
| Cystic fibrosis transmembrane conductance regulator: expression and helicity of a double membrane‐spanning segment | Q47764074 | ||
| Channel-Lining Residues in the M3 Membrane-Spanning Segment of the Cystic Fibrosis Transmembrane Conductance Regulator | Q48933240 | ||
| Multiple proteolytic systems, including the proteasome, contribute to CFTR processing | Q50337154 | ||
| Cystic fibrosis-associated mutations at arginine 347 alter the pore architecture of CFTR. Evidence for disruption of a salt bridge | Q52533943 | ||
| Structure of the multidrug resistance P-glycoprotein to 2.5 nm resolution determined by electron microscopy and image analysis | Q53970603 | ||
| Stimulation of CFTR activity by its phosphorylated R domain | Q54558597 | ||
| Correction of Defective Protein Kinesis of Human P-glycoprotein Mutants by Substrates and Modulators | Q57976939 | ||
| Disease-Associated Mutations in Cytoplasmic Loops 1 and 2 of Cystic Fibrosis Transmembrane Conductance Regulator Impede Processing or Opening of the Channel† | Q57976941 | ||
| Multi-ion pore behaviour in the CFTR chloride channel | Q59073236 | ||
| P433 | issue | 6 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | cystic fibrosis | Q178194 |
| transmembrane protein | Q424204 | ||
| P304 | page(s) | 3729-3732 | |
| P577 | publication date | 2000-02-01 | |
| P13046 | publication type of scholarly work | review article | Q7318358 |
| P1433 | published in | Journal of Biological Chemistry | Q867727 |
| P1476 | title | Cystic fibrosis transmembrane conductance regulator. Structure and function of an epithelial chloride channel | |
| P478 | volume | 275 |
| Q33327200 | 4'-Methyl-4,5'-bithiazole-based correctors of defective delta F508-CFTR cellular processing |
| Q36173784 | ATP release via anion channels |
| Q35108320 | Abnormal passive chloride absorption in cystic fibrosis jejunum functionally opposes the classic chloride secretory defect |
| Q43724548 | Activity of fucosyltransferases and altered glycosylation in cystic fibrosis airway epithelial cells |
| Q33286071 | Additive effect of multiple pharmacological chaperones on maturation of CFTR processing mutants |
| Q48888998 | Analysis of ABCC6 (MRP6) in normal human tissues |
| Q41944862 | Analysis of c.3499+200TA(7_56) and D7S523 Microsatellites Linked to Cystic Fibrosis Transmembrane Regulator |
| Q37441497 | Animal Enterotoxigenic Escherichia coli |
| Q57375039 | Anion binding and transport properties of cyclic 2,6-bis(1,2,3-triazol-1-yl)pyridines |
| Q34245855 | Binding and Selectivity of Halides with Macrocyclic polyamines |
| Q40605887 | Binding of sulfonylurea by AtMRP5, an Arabidopsis multidrug resistance-related protein that functions in salt tolerance |
| Q50665220 | CFTR fails to inhibit the epithelial sodium channel ENaC expressed in Xenopus laevis oocytes |
| Q85895434 | CFTR-mutation specific applications of CFTR-directed monoclonal antibodies |
| Q34225532 | Chloride channel in vanadocytes of a vanadium-rich ascidian Ascidia sydneiensis samea |
| Q52596420 | Comparison of differentially expressed genes in the salivary glands of male ticks, Amblyomma americanum and Dermacentor andersoni |
| Q39939464 | Control of cellular GADD34 levels by the 26S proteasome |
| Q92697837 | Cystic Fibrosis-Related Diabetes: Pathophysiology and Therapeutic Challenges |
| Q41819157 | Cystic fibrosis transmembrane conductance regulator: the NBF1+R (nucleotide-binding fold 1 and regulatory domain) segment acting alone catalyses a Co2+/Mn2+/Mg2+-ATPase activity markedly inhibited by both Cd2+ and the transition-state analogue ortho |
| Q45146747 | Destabilization of the transmembrane domain induces misfolding in a phenotypic mutant of cystic fibrosis transmembrane conductance regulator |
| Q44529210 | Disassembly and degradation of photosystem I in an in vitro system are multievent, metal-dependent processes |
| Q35895699 | Divergent CFTR orthologs respond differently to the channel inhibitors CFTRinh-172, glibenclamide, and GlyH-101 |
| Q34982463 | Experimental and theoretical studies on halide binding with a p-xylyl-based azamacrocycle |
| Q38171539 | Functional architecture of the CFTR chloride channel |
| Q37858330 | Golgi Bypass: Skirting Around the Heart of Classical Secretion |
| Q21284513 | Hematopoietic and mesenchymal stem cells for the treatment of chronic respiratory diseases: role of plasticity and heterogeneity |
| Q41168425 | Hematopoietic stem/progenitor cells express functional mitochondrial energy-dependent cystic fibrosis transmembrane conductance regulator |
| Q43737971 | Identification of a region of strong discrimination in the pore of CFTR. |
| Q41574129 | Increased Expression of Plasma-Induced ABCC1 mRNA in Cystic Fibrosis. |
| Q40724271 | Introduction of the most common cystic fibrosis mutation (Delta F508) into human P-glycoprotein disrupts packing of the transmembrane segments. |
| Q40850802 | Localization of sequences within the C-terminal domain of the cystic fibrosis transmembrane conductance regulator which impact maturation and stability |
| Q21284420 | Mechanisms of the noxious inflammatory cycle in cystic fibrosis |
| Q42992980 | Molecular dynamics simulation of the transmembrane subunit of BtuCD in the lipid bilayer |
| Q36037679 | Molecular pathology of the CFTR locus in male infertility |
| Q40537131 | Processing mutations located throughout the human multidrug resistance P-glycoprotein disrupt interactions between the nucleotide binding domains. |
| Q44973198 | Purification and crystallization of the cystic fibrosis transmembrane conductance regulator (CFTR). |
| Q36224105 | Regulation of calcium-activated chloride channels in smooth muscle cells: a complex picture is emerging |
| Q33903142 | Regulation of volume-activated chloride channels by P-glycoprotein: phosphorylation has the final say! |
| Q52718918 | Revisiting sweat chloride test results based on recent guidelines for diagnosis of cystic fibrosis |
| Q35235024 | Small molecule inhibition of the Na(+)/H(+) exchange regulatory factor 1 and parathyroid hormone 1 receptor interaction. |
| Q37754873 | Stem cell therapy for cystic fibrosis: current status and future prospects |
| Q27631209 | Structural basis of the Na+/H+ exchanger regulatory factor PDZ1 interaction with the carboxyl-terminal region of the cystic fibrosis transmembrane conductance regulator |
| Q38132251 | The application of Ussing chambers for determining the impact of microbes and probiotics on intestinal ion transport |
| Q48278905 | The distribution of Abcc6 in normal mouse tissues suggests multiple functions for this ABC transporter |
| Q28649323 | The elements of life and medicines |
| Q35206694 | The phenotypic consequences of CFTR mutations |
| Q39140318 | The plant multidrug resistance ABC transporter AtMRP5 is involved in guard cell hormonal signalling and water use. |
| Q52072748 | Topology studies of the chloroplast protein import channel Toc75. |
| Q51760773 | Use of a 4-week up-titration regimen of roflumilast in patients with severe COPD. |
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