scholarly article | Q13442814 |
P2093 | author name string | A. Hirano | |
P2860 | cites work | Altered translation of the matrix genes in Niigata and Yamagata neurovirulent measles virus strains | Q45046458 |
Biased hypermutation and other genetic changes in defective measles viruses in human brain infections | Q45060135 | ||
Development of attenuated measles-virus vaccines. A summary of recentinvestigation | Q45716994 | ||
Defective translation of measles virus matrix protein in a subacute sclerosing panencephalitis cell line | Q45793783 | ||
Persistent infection by a temperature-sensitive mutant isolated from a Sendai virus (HVJ) carrier culture: Its initiation and maintenance without aid of defective interfering particles | Q45800088 | ||
Interference between Virulent and Avirulent Strains of Sendai Virus | Q45801208 | ||
An Intracellular Interaction Between a Temperature-sensitive Mutant and the Original Wild-type HVJ (Sendai Virus) is Responsible for the Establishment and Maintenance of HVJ Persistent Infection | Q45801351 | ||
The measles virus matrix gene and gene product defined by in vitro and in vivo expression | Q45829096 | ||
Accumulated measles virus mutations in a case of subacute sclerosing panencephalitis: interrupted matrix protein reading frame and transcription alteration | Q45829593 | ||
Dominance of temperature-sensitive phenotypes. II. Vesicular stomatitis virus mutants from a persistent infection interfere with shut-off of host protein synthesis by wild-type virus | Q45834722 | ||
Dominance of temperature-sensitive phenotypes. I. Studies of the mechanism of inhibition of the growth of wild-type vesicular stomatitis virus | Q45835861 | ||
Immediate persistent infection by human parainfluenza virus 3: unique fusion properties of the persistently infected cells | Q45848024 | ||
Immediate protection of mice from lethal wild-type Sendai virus (HVJ) infections by a temperature-sensitive mutant, HVJpi, possessing homologous interfering capacity | Q45851325 | ||
Subacute sclerosing panencephalitis (SSPE): isolation of a defective variant of measles virus from brain obtained at autopsy | Q45883170 | ||
Biased hypermutation of viral RNA genomes could be due to unwinding/modification of double-stranded RNA. | Q52868697 | ||
The L protein of a VSV mutant isolated from a persistent infection is responsible for viral interference and dominance over the wild-type | Q69333160 | ||
Mutated and hypermutated genes of persistent measles viruses which caused lethal human brain diseases | Q69401420 | ||
A fusion-defective mutant of the vesicular stomatitis virus glycoprotein | Q27486497 | ||
Complementation analysis of measles virus mutants isolated from persistently infected lymphoblastoid cell lines | Q33916045 | ||
Altered transcription of a defective measles virus genome derived from a diseased human brain | Q33928918 | ||
Temperature-Sensitive Defect of Mutants Isolated from L Cells Persistently Infected with Newcastle Disease Virus | Q34218820 | ||
Persistent infection of BSC-1 cells by defective measles virus derived from subacute sclerosing panencephalitis | Q34506790 | ||
Role of biased hypermutation in evolution of subacute sclerosing panencephalitis virus from progenitor acute measles virus | Q36688348 | ||
Fusion properties of cells persistently infected with human parainfluenza virus type 3: participation of hemagglutinin-neuraminidase in membrane fusion | Q36692466 | ||
Structural defect linked to nonrandom mutations in the matrix gene of biken strain subacute sclerosing panencephalitis virus defined by cDNA cloning and expression of chimeric genes | Q36781555 | ||
Leader sequence distinguishes between translatable and encapsidated measles virus RNAs | Q36799064 | ||
Characterization of human parainfluenza virus type 3 persistent infection in cell culture | Q36809412 | ||
Generalized and localized biased hypermutation affecting the matrix gene of a measles virus strain that causes subacute sclerosing panencephalitis | Q36832492 | ||
Functional interaction between transcriptional elements in the long terminal repeat of reticuloendotheliosis virus: cooperative DNA binding of promoter- and enhancer-specific factors | Q36850201 | ||
Expression of defective measles virus genes in brain tissues of patients with subacute sclerosing panencephalitis | Q36865122 | ||
Multiple viral mutations rather than host factors cause defective measles virus gene expression in a subacute sclerosing panencephalitis cell line | Q36866338 | ||
Identification of a nonproductive, cell-associated form of measles virus by its resistance to inhibition by recombinant human interferon | Q36888866 | ||
Transcription and replication of rhabdoviruses. | Q37063165 | ||
Viral interference-dominance of mutant viruses over wild-type virus in mixed infections | Q37063362 | ||
Tubulin: a factor necessary for the synthesis of both Sendai virus and vesicular stomatitis virus RNAs | Q37391545 | ||
Rapid degradation restricts measles virus matrix protein expression in a subacute sclerosing panencephalitis cell line | Q37404122 | ||
Viral persistence | Q38602842 | ||
Homologous Interference Induced by a Temperature-sensitive Mutant Derived from an HVJ (Sendai Virus) Carrier Culture | Q40027770 | ||
Human parainfluenza virus type 3 transcription in vitro: role of cellular actin in mRNA synthesis | Q40064981 | ||
Measles and SSPE viruses: similarities and differences. | Q40333115 | ||
Development of extremely attenuated live measles virus vaccine (CAM-EX) | Q40916837 | ||
Restricted expression of measles virus proteins in brains from cases of subacute sclerosing panencephalitis | Q41452066 | ||
Nonproductive, cell-associated virus exists before the appearance of antiviral antibodies in experimental measles encephalitis | Q41479535 | ||
Expression of measles virus RNA in peripheral blood mononuclear cells of patients with measles, SSPE, and autoimmune diseases | Q41681109 | ||
Generation of mammalian cells expressing stably measles virus proteins via bicistronic RNA. | Q41681201 | ||
Rescue of a sendai virus DI genome by other parainfluenza viruses: Implications for genome replication | Q41685958 | ||
Host cell proteins required for measles virus reproduction | Q41738157 | ||
High Efficiency DNA-Mediated Transformation of Primate Cells | Q44901923 | ||
P433 | issue | 4 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | subacute sclerosing panencephalitis | Q2475919 |
measles | Q79793 | ||
viral persistence | Q44134905 | ||
P304 | page(s) | 1891-1898 | |
P577 | publication date | 1992-04-01 | |
P1433 | published in | Journal of Virology | Q1251128 |
P1476 | title | Subacute sclerosing panencephalitis virus dominantly interferes with replication of wild-type measles virus in a mixed infection: implication for viral persistence | |
P478 | volume | 66 |
Q36503699 | A review of the possible mechanisms for the persistence of foot-and-mouth disease virus |
Q36643713 | Cell fusion by the envelope glycoproteins of persistent measles viruses which caused lethal human brain disease. |
Q27469529 | Chimeric measles viruses with a foreign envelope |
Q39870131 | Engineered serine protease inhibitor prevents furin-catalyzed activation of the fusion glycoprotein and production of infectious measles virus. |
Q36685898 | Functional analysis of matrix proteins expressed from cloned genes of measles virus variants that cause subacute sclerosing panencephalitis reveals a common defect in nucleocapsid binding |
Q27469706 | Human CD46 enhances nitric oxide production in mouse macrophages in response to measles virus infection in the presence of gamma interferon: dependence on the CD46 cytoplasmic domains |
Q35870503 | Human cell receptor CD46 is down regulated through recognition of a membrane-proximal region of the cytoplasmic domain in persistent measles virus infection |
Q35875948 | Identification of a cytoplasmic Tyr-X-X-Leu motif essential for down regulation of the human cell receptor CD46 in persistent measles virus infection |
Q41331027 | In vivo andin vitro expression of defective hepatitis B virus particles generated by spliced hepatitis B virus RNA |
Q41729122 | Mechanisms involved in natural and experimental neuropathogenicity of influenza viruses: evidence and speculation. |
Q36127607 | Molecular mechanisms of measles virus persistence |
Q41026554 | Persistent infection with an influenza C virus variant is dominantly established in the presence of the parental wild-type virus |
Q30670817 | Subacute sclerosing panencephalitis |
Q37208649 | The matrix proteins of neurovirulent subacute sclerosing panencephalitis virus and its acute measles virus progenitor are functionally different |
Q40970203 | The phosphoprotein genes of measles viruses from subacute sclerosing panencephalitis cases encode functional as well as non-functional proteins and display reduced editing. |
Q36801980 | Vaccine-induced pathogen strain replacement: what are the mechanisms? |
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