scholarly article | Q13442814 |
P50 | author | Koichiro Kinugawa | Q86697753 |
P2093 | author name string | Charles Vinson | |
Carlin S Long | |||
Mark Y Jeong | |||
P2860 | cites work | c-Jun dimerizes with itself and with c-Fos, forming complexes of different DNA binding affinities | Q28291350 |
c-Jun N-terminal kinase-interacting protein 1 inhibits gene expression in response to hypertrophic agonists in neonatal rat ventricular myocytes | Q28346420 | ||
Chronic phospholamban-sarcoplasmic reticulum calcium ATPase interaction is the critical calcium cycling defect in dilated cardiomyopathy | Q28505358 | ||
The role of Jun, Fos and the AP-1 complex in cell-proliferation and transformation | Q29400446 | ||
Rescue of contractile parameters and myocyte hypertrophy in calsequestrin overexpressing myocardium by phospholamban ablation | Q31685225 | ||
Opposing effects of Jun kinase and p38 mitogen-activated protein kinases on cardiomyocyte hypertrophy | Q33774336 | ||
MEKK1 is essential for cardiac hypertrophy and dysfunction induced by Gq. | Q34020904 | ||
Interleukin-1 beta induces cardiac myocyte growth but inhibits cardiac fibroblast proliferation in culture | Q34223350 | ||
A role for the p38 mitogen-activated protein kinase pathway in myocardial cell growth, sarcomeric organization, and cardiac-specific gene expression | Q34440843 | ||
Cardiac muscle cell hypertrophy and apoptosis induced by distinct members of the p38 mitogen-activated protein kinase family | Q34453631 | ||
Myosin heavy chain messenger RNA and protein isoform transitions during cardiac hypertrophy. Interaction between hemodynamic and thyroid hormone-induced signals | Q34565604 | ||
Adenoviral gene transfer of SERCA2a improves left-ventricular function in aortic-banded rats in transition to heart failure | Q34978011 | ||
Growth factors, growth factor response elements, and the cardiac phenotype | Q35169934 | ||
Thyroid hormone improves function and Ca2+ handling in pressure overload hypertrophy. Association with increased sarcoplasmic reticulum Ca2+-ATPase and alpha-myosin heavy chain in rat hearts | Q37372872 | ||
Localization and regulation of c-fos and c-jun protooncogene induction by systolic wall stress in normal and hypertrophied rat hearts | Q37650751 | ||
Adenoviral delivery of A-FOS, an AP-1 dominant negative, selectively inhibits drug resistance in two human cancer cell lines | Q38290833 | ||
Serum response factor mediates AP-1-dependent induction of the skeletal alpha-actin promoter in ventricular myocytes | Q38357989 | ||
The C-terminal domain of c-fos is required for activation of an AP-1 site specific for jun-fos heterodimers | Q39575770 | ||
In vivo regulation of the beta-myosin heavy chain gene in hypertensive rodent heart | Q40715290 | ||
Growth and hypertrophy of the heart: towards an understanding of cardiac specific and inducible gene expression | Q40786533 | ||
The regulation of AP-1 activity by mitogen-activated protein kinases | Q40998182 | ||
Adrenergic receptor stimulation of the mitogen-activated protein kinase cascade and cardiac hypertrophy | Q42130562 | ||
Transgenic expression of sarcoplasmic reticulum Ca(2+) atpase modifies the transition from hypertrophy to early heart failure | Q43726347 | ||
Regulation of thyroid hormone receptor isoforms in physiological and pathological cardiac hypertrophy | Q43750917 | ||
Dominant negative mutant of c-Jun inhibits cardiomyocyte hypertrophy induced by endothelin 1 and phenylephrine | Q43859251 | ||
Overexpression of the sarcoplasmic reticulum Ca(2+)-ATPase improves myocardial contractility in diabetic cardiomyopathy | Q43935483 | ||
Alpha- and beta-adrenergic stimulation induces distinct patterns of immediate early gene expression in neonatal rat myocardial cells. fos/jun expression is associated with sarcomere assembly; Egr-1 induction is primarily an alpha 1-mediated response | Q44237547 | ||
Sustained activation of nuclear factor kappa B and activator protein 1 in chronic heart failure | Q44346308 | ||
Induction of c-fos gene and protein by growth factors precedes activation of c-myc | Q44887897 | ||
Positive regulation of the skeletal alpha-actin gene by Fos and Jun in cardiac myocytes | Q46099717 | ||
Expression of a constitutively activated mutant of the beta-isozyme of protein kinase C in cardiac myocytes stimulates the promoter of the beta-myosin heavy chain isogene. | Q46312003 | ||
Gq- and ras-dependent pathways mediate hypertrophy of neonatal rat ventricular myocytes following alpha 1-adrenergic stimulation. | Q46652497 | ||
Interleukin-1beta is a negative transcriptional regulator of alpha1-adrenergic induced gene expression in cultured cardiac myocytes | Q46713204 | ||
Expression of cellular oncogenes in the myocardium during the developmental stage and pressure-overloaded hypertrophy of the rat heart. | Q52251620 | ||
A dominant negative to activation protein-1 (AP1) that abolishes DNA binding and inhibits oncogenesis. | Q53441453 | ||
Rapid induction of the expression of proto-oncogene fos during human monocytic differentiation. | Q53547695 | ||
Bone and haematopoietic defects in mice lacking c-fos | Q58326969 | ||
Platelet-derived growth factor induces rapid but transient expression of the c-fos gene and protein | Q59086707 | ||
A null mutation at the c-jun locus causes embryonic lethality and retarded cell growth in culture | Q62555340 | ||
Different requirements for formation of Jun: Jun and Jun: Fos complexes | Q69274554 | ||
Dimerization specificity of the leucine zipper-containing bZIP motif on DNA binding: prediction and rational design | Q70744379 | ||
Thyroid hormone influences beta myosin heavy chain (beta MHC) expression | Q72340101 | ||
Ras activity is required for phenylephrine-induced activation of mitogen-activated protein kinase in cardiac muscle cells | Q72362916 | ||
Stimulation of "stress-regulated" mitogen-activated protein kinases (stress-activated protein kinases/c-Jun N-terminal kinases and p38-mitogen-activated protein kinases) in perfused rat hearts by oxidative and other stresses | Q74358725 | ||
P433 | issue | 13 | |
P407 | language of work or name | English | Q1860 |
P304 | page(s) | 1645-1651 | |
P577 | publication date | 2005-03-28 | |
P1433 | published in | Circulation | Q578091 |
P1476 | title | AFos dissociates cardiac myocyte hypertrophy and expression of the pathological gene program | |
P478 | volume | 111 |
Q64228412 | A Novel Method of Isolating Myofibrils From Primary Cardiomyocyte Culture Suitable for Myofibril Mechanical Study |
Q33917067 | AFos inhibits phenylephrine-mediated contractile dysfunction by altering phospholamban phosphorylation |
Q40785458 | AMP-activated protein kinase α1-sensitive activation of AP-1 in cardiomyocytes |
Q58129769 | Activin Receptor-Like Kinase 4 Haplodeficiency Mitigates Arrhythmogenic Atrial Remodeling and Vulnerability to Atrial Fibrillation in Cardiac Pathological Hypertrophy |
Q34398211 | Cross-talk between angiotensin II and IGF-1-induced connexin 43 expression in human saphenous vein smooth muscle cells |
Q42370598 | Divergence of hypertrophic growth and fetal gene profile: the influence of beta-blockers |
Q37310859 | Gallic acid prevents isoproterenol-induced cardiac hypertrophy and fibrosis through regulation of JNK2 signaling and Smad3 binding activity |
Q37395007 | Involvement of connexin 43 in angiotensin II-induced migration and proliferation of saphenous vein smooth muscle cells via the MAPK-AP-1 signaling pathway |
Q36484609 | Knockout of beta(1)- and beta(2)-adrenoceptors attenuates pressure overload-induced cardiac hypertrophy and fibrosis |
Q36528033 | MAP kinase kinase kinase-2 (MEKK2) regulates hypertrophic remodeling of the right ventricle in hypoxia-induced pulmonary hypertension |
Q92541680 | Molecular mechanisms underlying gallic acid effects against cardiovascular diseases: An update review |
Q56335220 | Network-based predictions of in vivo cardiac hypertrophy |
Q46647997 | Phenylephrine hypertrophy, Ca2+-ATPase (SERCA2), and Ca2+ signaling in neonatal rat cardiac myocytes |
Q35995211 | Propranolol causes a paradoxical enhancement of cardiomyocyte foetal gene response to hypertrophic stimuli |
Q28579994 | Repression of anti-apoptotic genes via AP-1 as a mechanism of apoptosis induction in ventricular cardiomyocytes |
Q51763589 | Restoration of CREB function is linked to completion and stabilization of adaptive cardiac hypertrophy in response to exercise. |
Q35106659 | Sex differences in cardiomyocyte connexin43 expression. |
Q43838536 | T-type Ca²⁺ signalling downregulates MEK1/2 phosphorylation and cross-talk with the RAAS transcriptional response in cardiac myocytes. |
Q33295387 | TrpC3 regulates hypertrophy-associated gene expression without affecting myocyte beating or cell size |
Q46318982 | cAMP-mediated beta-adrenergic signaling negatively regulates Gq-coupled receptor-mediated fetal gene response in cardiomyocytes |
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