Duplication mutation as an SOS response in Escherichia coli: enhanced duplication formation by a constitutively activated RecA.

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Duplication mutation as an SOS response in Escherichia coli: enhanced duplication formation by a constitutively activated RecA. is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1093/GENETICS/123.2.255
P932PMC publication ID1203797
P698PubMed publication ID2684744

P2093author name stringH Echols
J Dimpfl
P2860cites workSOS Repair Hypothesis: Phenomenology of an Inducible DNA Repair Which is Accompanied by MutagenesisQ28141658
The origin of mutantsQ28288915
The significance of responses of the genome to challengeQ28913697
Properties of the translocatable tetracycline-resistance element Tn10 in Escherichia coli and bacteriophage lambdaQ33993160
Ultraviolet mutagenesis and inducible DNA repair in Escherichia coliQ34072546
Spontaneous tandem genetic duplications in Salmonella typhimurium arise by unequal recombination between rRNA (rrn) cistronsQ35364956
Capacity of RecA protein to bind preferentially to UV lesions and inhibit the editing subunit (epsilon) of DNA polymerase III: a possible mechanism for SOS-induced targeted mutagenesisQ35588095
Inducibility of a gene product required for UV and chemical mutagenesis in Escherichia coliQ36369577
Mutagenesis and inducible responses to deoxyribonucleic acid damage in Escherichia coliQ37060983
Involvement of the activated form of RecA protein in SOS mutagenesis and stable DNA replication in Escherichia coliQ37579186
A mutant of Escherichia coli showing constitutive expression of the lysogenic induction and error-prone DNA repair pathwaysQ37598966
Recombination between repeated genes in microorganismsQ39569901
Enzymes of general recombinationQ39664928
Tandem genetic duplications in phage and bacteriaQ39707701
Inducible DNA repair systemsQ39826678
Recombination deficient mutants of E. coli and other bacteriaQ39880456
Is there SOS induction in mammalian cells?Q40217915
Mutation rate: some biological and biochemical considerationsQ40266425
SOS functions, cancer and inducible evolutionQ40313397
The SOS regulatory system of Escherichia coliQ40335741
Isolation and characterization of mutants of Escherichia coli deficient in induction of mutations by ultraviolet lightQ40850491
Uvm mutants of Escherichia coli K12 deficient in UV mutagenesis. I. Isolation of uvm mutants and their phenotypical characterization in DNA repair and mutagenesisQ40958827
Preferential unequal recombination in the glyS region of the Escherichia coli chromosomeQ41008325
Adaptive evolution that requires multiple spontaneous mutations. I. Mutations involving an insertion sequence.Q42960926
Prophage induction and cell division in E. coli. III. Mutations sfiA and sfiB restore division in tif and lon strains and permit the expression of mutator properties of tifQ44110038
RecA protein and SOS. Correlation of mutagenesis phenotype with binding of mutant RecA proteins to duplex DNA and LexA cleavageQ44834679
Effects of DNA-repair processes on the induction of genetic duplications in bacteria by ultraviolet lightQ50205403
A repetitive DNA sequence, rhs, responsible for duplications within the Escherichia coli K-12 chromosomeQ50207908
Tandem duplications of the histidine operon observed following generalized transduction in Salmonella typhimuriumQ50227159
Genetic duplications induced at very high frequency by ultraviolet irradiation in Escherichia coli.Q54139746
Repair of DNA double-strand breaks in Escherichia coli K12 requires a functional recN productQ54485834
Duplication of the structural gene for glycyl-transfer RNA synthetase in Escherichia coliQ69938790
Damage to DNA induces expression of the ruv gene of Escherichia coliQ70241303
The selective advantage of cancer cells: a consequence of genome mobilization in the course of the induction of DNA repair processes? (Model studies on yeast)Q70378923
The SOS regulatory system: control of its state by the level of RecA proteaseQ70442106
The steric effect in lysogenization by bacteriophage lambda. I. Lysogenization of a partially diploid strain of Escherichia coli K-12Q71165589
Constitutive expression of SOS functions and modulation of mutagenesis resulting from resolution of genetic instability at or near the recA locus of Escherichia coliQ72756712
P433issue2
P407language of work or nameEnglishQ1860
P921main subjectEscherichia coliQ25419
P1104number of pages6
P304page(s)255-260
P577publication date1989-10-01
P1433published inGeneticsQ3100575
P1476titleDuplication mutation as an SOS response in Escherichia coli: enhanced duplication formation by a constitutively activated RecA.
Duplication mutation as an SOS response in Escherichia coli: enhanced duplication formation by a constitutively activated RecA
P478volume123

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cites work (P2860)
Q24596056Adaptive mutation: the uses of adversity
Q35148298Bacterial assays for recombinagens
Q34617418Conjugational Hyperrecombination Achieved by Derepressing the LexA Regulon, Altering the Properties of RecA Protein and Inactivating Mismatch Repair in Escherichia coli K-12
Q33727996Conjugative DNA transfer induces the bacterial SOS response and promotes antibiotic resistance development through integron activation.
Q42962535Control of large chromosomal duplications in Escherichia coli by the mismatch repair system
Q35917475Directed mutation: between unicorns and goats
Q35165984Dynamics of gene duplication in the genomes of chlorophyll d-producing cyanobacteria: implications for the ecological niche
Q24672935High fidelity of RecA-catalyzed recombination: a watchdog of genetic diversity
Q41110210Mechanisms of directed mutation
Q54244726Participation of the SOS system in producing deletions in E. coli plasmids
Q33743436Sister chromatid exchange frequencies in Escherichia coli analyzed by recombination at the dif resolvase site.
Q34872344Starvation-associated genome restructuring can lead to reproductive isolation in yeast
Q34504117Stress-induced evolution and the biosafety of genetically modified microorganisms released into the environment
Q33719467cAMP-dependent SOS induction and mutagenesis in resting bacterial populations.