scholarly article | Q13442814 |
review article | Q7318358 |
P2093 | author name string | Johnson WG | |
P2860 | cites work | Synphilin-1 associates with alpha-synuclein and promotes the formation of cytosolic inclusions | Q22009563 |
Mutation in the alpha-synuclein gene identified in families with Parkinson's disease | Q27860459 | ||
Inhibition of caspase-1 slows disease progression in a mouse model of Huntington's disease | Q28114818 | ||
Association of missense and 5'-splice-site mutations in tau with the inherited dementia FTDP-17 | Q28274687 | ||
NACP/alpha-synuclein immunoreactivity in fibrillary components of neuronal and oligodendroglial cytoplasmic inclusions in the pontine nuclei in multiple system atrophy | Q28289929 | ||
Mapping of a gene for Parkinson's disease to chromosome 4q21-q23 | Q28295005 | ||
Ala30Pro mutation in the gene encoding alpha-synuclein in Parkinson's disease | Q29547175 | ||
Stabilization of alpha-synuclein secondary structure upon binding to synthetic membranes | Q29617606 | ||
Structure/function in neuroprotection and apoptosis. | Q30431508 | ||
Abnormal accumulation of NACP/alpha-synuclein in neurodegenerative disorders | Q32121511 | ||
From computer simulations to human disease: emerging themes in protein folding | Q33630957 | ||
Neuropathologic overlap of progressive supranuclear palsy, Pick's disease and corticobasal degeneration | Q34371240 | ||
Alternative conformations of amyloidogenic proteins govern their behavior | Q34387876 | ||
Familial progressive supranuclear palsy. Description of a pedigree and review of the literature | Q34719147 | ||
Autopsy-proven progressive supranuclear palsy in two siblings | Q34734443 | ||
Lewy bodies contain altered alpha-synuclein in brains of many familial Alzheimer's disease patients with mutations in presenilin and amyloid precursor protein genes | Q35754459 | ||
The environmental dependency of protein folding best explains prion and amyloid diseases | Q36054435 | ||
Caspase-8 is required for cell death induced by expanded polyglutamine repeats. | Q40961806 | ||
Both familial Parkinson's disease mutations accelerate alpha-synuclein aggregation | Q40964871 | ||
Steroid hormones block amyloid fibril-induced 3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazolium bromide (MTT) formazan exocytosis: relationship to neurotoxicity. | Q40992053 | ||
Neurodegenerative disorders with extensive tau pathology: a comparative study and review | Q41084709 | ||
Normal and pathological Tau proteins as factors for microtubule assembly. | Q41379250 | ||
Harm's way: polyglutamine repeats and the activation of an apoptotic pathway. | Q41644857 | ||
The alternative conformations of amyloidogenic proteins and their multi-step assembly pathways | Q41728317 | ||
Polyglutamine-expanded human huntingtin transgenes induce degeneration of Drosophila photoreceptor neurons. | Q45296540 | ||
Human recombinant NACP/alpha-synuclein is aggregated and fibrillated in vitro: relevance for Lewy body disease | Q46084408 | ||
Abnormal distribution of the non-Abeta component of Alzheimer's disease amyloid precursor/alpha-synuclein in Lewy body disease as revealed by proteinase K and formic acid pretreatment | Q46198761 | ||
Alpha-synuclein immunoreactivity in glial cytoplasmic inclusions in multiple system atrophy | Q47772222 | ||
The ubiquitin pathway in Parkinson's disease | Q48015939 | ||
Accumulation of alpha-synuclein/NACP is a cytopathological feature common to Lewy body disease and multiple system atrophy. | Q48342277 | ||
Accelerated in vitro fibril formation by a mutant alpha-synuclein linked to early-onset Parkinson disease. | Q48353748 | ||
Synthetic filaments assembled from C-terminally truncated alpha-synuclein. | Q48360396 | ||
Huntingtin acts in the nucleus to induce apoptosis but death does not correlate with the formation of intranuclear inclusions. | Q48373564 | ||
Binding of alpha-synuclein to brain vesicles is abolished by familial Parkinson's disease mutation. | Q48384247 | ||
Glial cytoplasmic inclusions in white matter oligodendrocytes of multiple system atrophy brains contain insoluble alpha-synuclein. | Q48389356 | ||
Filamentous alpha-synuclein inclusions link multiple system atrophy with Parkinson's disease and dementia with Lewy bodies. | Q48401512 | ||
Multiple-system atrophy: a new alpha-synuclein disease? | Q48406192 | ||
Changes in presynaptic protein NACP/alpha-synuclein in an ischemic gerbil hippocampus. | Q48466817 | ||
Reversible conversion of monomeric human prion protein between native and fibrilogenic conformations. | Q54102352 | ||
Synchrotron X-ray studies suggest that the core of the transthyretin amyloid fibril is a continuous β-sheet helix | Q57945110 | ||
Extensive axonal Lewy neurites in Parkinson's disease: a novel pathological feature revealed by alpha-synuclein immunocytochemistry | Q64861825 | ||
Aggregates from mutant and wild-type alpha-synuclein proteins and NAC peptide induce apoptotic cell death in human neuroblastoma cells by formation of beta-sheet and amyloid-like filaments | Q64866811 | ||
P433 | issue | 4 | |
P921 | main subject | neurodegeneration | Q1755122 |
P304 | page(s) | 609-616 | |
P577 | publication date | 2000-05-01 | |
P1433 | published in | Journal of Anatomy | Q2108124 |
P1476 | title | Late-onset neurodegenerative diseases--the role of protein insolubility | |
P478 | volume | 196 ( Pt 4) |
Q34340531 | Alpha synuclein aggregation: is it the toxic gain of function responsible for neurodegeneration in Parkinson's disease? |
Q37253310 | Alzheimer's disease and blood-brain barrier function-Why have anti-beta-amyloid therapies failed to prevent dementia progression? |
Q43256880 | Characterization of intrinsically disordered proteins with electrospray ionization mass spectrometry: conformational heterogeneity of alpha-synuclein |
Q38609997 | Cognitive impairment in humanized APP×PS1 mice is linked to Aβ(1-42) and NOX activation. |
Q28367378 | Conformational characterization of oligomeric intermediates and aggregates in beta-lactoglobulin heat aggregation |
Q36772901 | Decreased blood-brain barrier P-glycoprotein function in the progression of Parkinson's disease, PSP and MSA. |
Q33371063 | Differential regulation of wild-type and mutant alpha-synuclein binding to synaptic membranes by cytosolic factors |
Q24544265 | Disulfide cross-linked protein represents a significant fraction of ALS-associated Cu, Zn-superoxide dismutase aggregates in spinal cords of model mice |
Q24685507 | Dorfin localizes to the ubiquitylated inclusions in Parkinson's disease, dementia with Lewy bodies, multiple system atrophy, and amyotrophic lateral sclerosis |
Q41969333 | Effects of urea and acetic acid on the heme axial ligation structure of ferric myoglobin at very acidic pH. |
Q27027455 | Evaluation of traditional medicines for neurodegenerative diseases using Drosophila models |
Q37351380 | Impaired mitochondrial energy production and ABC transporter function-A crucial interconnection in dementing proteopathies of the brain |
Q34741685 | Learning cell biology as a team: a project-based approach to upper-division cell biology |
Q50764411 | Mutant protein kinase Cgamma found in spinocerebellar ataxia type 14 is susceptible to aggregation and causes cell death. |
Q43265626 | Neuroprotection in Parkinson's disease: love story or mission impossible? |
Q42208600 | One-year follow-up of transgene expression by integrase-defective lentiviral vectors and their therapeutic potential in spinocerebellar ataxia model mice |
Q34831541 | Programmed cell death in amyotrophic lateral sclerosis |
Q34286115 | Protein misfolding and disease; protein refolding and therapy |
Q35036503 | Recent advances in amyotrophic lateral sclerosis research |
Q35577679 | The Hunt for a Cure for Parkinson’s Disease |
Q45884689 | The emerging utility of animal models of chronic neurodegenerative diseases |
Q24299624 | The novel cytosolic RING finger protein dactylidin is up-regulated in brains of patients with Alzheimer's disease |
Q30503511 | Two-dimensional infrared correlation spectroscopy study of sequential events in the heat-induced unfolding and aggregation process of myoglobin |
Q41141295 | α -Synuclein Modification in an ALS Animal Model |
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