scholarly article | Q13442814 |
P2093 | author name string | Emilio R Garrido-Sanabria | |
Luis F Pacheco Otalora | |||
Massoud F Arshadmansab | |||
Frank Skinner | |||
Hans-Günther Knaus | |||
Boris S Ermolinsky | |||
Carlos F Mello | |||
Ileana Garcia | |||
Mauro S Oliveira | |||
P2860 | cites work | Modulators of Small- and Intermediate-Conductance Calcium-Activated Potassium Channels and their Therapeutic Indications | Q22305804 |
Small-conductance, calcium-activated potassium channels from mammalian brain | Q24318679 | ||
Selective positive modulation of the SK3 and SK2 subtypes of small conductance Ca2+-activated K+ channels | Q24564966 | ||
A human intermediate conductance calcium-activated potassium channel | Q24654684 | ||
Small conductance Ca2+-activated K+ channels and calmodulin | Q24678202 | ||
Small conductance Ca2+-activated K+ channels modulate synaptic plasticity and memory encoding | Q28216064 | ||
Modification of seizure activity by electrical stimulation. II. Motor seizure | Q28236678 | ||
Functions of SK channels in central neurons | Q28241766 | ||
Activation of human IK and SK Ca2+ -activated K+ channels by NS309 (6,7-dichloro-1H-indole-2,3-dione 3-oxime) | Q28286744 | ||
Pharmacological characterization of small-conductance Ca(2+)-activated K(+) channels stably expressed in HEK 293 cells | Q28343978 | ||
Compounds that block both intermediate-conductance (IK(Ca)) and small-conductance (SK(Ca)) calcium-activated potassium channels | Q28344086 | ||
Initiation of network bursts by Ca2+-dependent intrinsic bursting in the rat pilocarpine model of temporal lobe epilepsy | Q28361757 | ||
Temporal lobe epilepsy: where do the seizures really begin? | Q30435240 | ||
K(+)-channel openers suppress epileptiform activities induced by 4-aminopyridine in cultured rat hippocampal neurons | Q46201828 | ||
Anticonvulsant effects of the BK-channel antagonist paxilline | Q46215516 | ||
1-Ethyl-2-benzimidazolinone (EBIO) suppresses epileptiform activity in in vitro hippocampus. | Q46579717 | ||
Hyperpolarizing potentials in guinea pig hippocampal CA3 neurons. | Q46863145 | ||
Memory processing and apamin induce immediate early gene expression in mouse brain | Q48309378 | ||
Inhibition of apamin-sensitive calcium dependent potassium channels facilitate the induction of long-term potentiation in the CA1 region of rat hippocampus in vitro. | Q48375923 | ||
Calcium-activated afterhyperpolarizations regulate synchronization and timing of epileptiform bursts in hippocampal CA3 pyramidal neurons | Q48421576 | ||
Abnormal mGluR2/3 expression in the perforant path termination zones and mossy fibers of chronically epileptic rats. | Q48490338 | ||
Control of the repetitive discharge of rat CA 1 pyramidal neurones in vitro | Q48628862 | ||
Effect of apamin, a toxin that inhibits Ca(2+)-dependent K+ channels, on learning and memory processes | Q48707301 | ||
Voltage-clamp analysis of the potentiation of the slow Ca2+-activated K+ current in hippocampal pyramidal neurons. | Q51404156 | ||
Differential distribution of three Ca(2+)-activated K(+) channel subunits, SK1, SK2, and SK3, in the adult rat central nervous system. | Q53911073 | ||
Control of epileptiform burst rate by CA3 hippocampal cell afterhyperpolarizations in vitro | Q69663857 | ||
Epileptiform burst afterhyperolarization: calcium-dependent potassium potential in hippocampal CA1 pyramidal cells | Q71416535 | ||
Synthesis, molecular modeling, and pharmacological testing of bis-quinolinium cyclophanes: potent, non-peptidic blockers of the apamin-sensitive Ca(2+)-activated K(+) channel | Q73435021 | ||
Relationship between large conductance calcium-activated potassium channel and bursting activity | Q73571701 | ||
In vivo characterisation of the small-conductance KCa (SK) channel activator 1-ethyl-2-benzimidazolinone (1-EBIO) as a potential anticonvulsant | Q80152918 | ||
Cyclicity of spontaneous recurrent seizures in pilocarpine model of temporal lobe epilepsy in rat | Q80176957 | ||
Reversal of age-related alterations in synaptic plasticity by blockade of L-type Ca2+ channels. | Q30471958 | ||
Mislocalization of h channel subunits underlies h channelopathy in temporal lobe epilepsy | Q30485454 | ||
Down-regulation of BK channel expression in the pilocarpine model of temporal lobe epilepsy | Q33320921 | ||
Molecular and cellular basis of small--and intermediate-conductance, calcium-activated potassium channel function in the brain | Q33560844 | ||
GCP II (NAALADase) inhibition suppresses mossy fiber-CA3 synaptic neurotransmission by a presynaptic mechanism | Q33606875 | ||
Small conductance Ca2+-activated K+ channel knock-out mice reveal the identity of calcium-dependent afterhyperpolarization currents | Q33701513 | ||
Excitation and inhibition in temporal lobe epilepsy: a close encounter. | Q33748554 | ||
Temporal lobe epilepsy surgery: definition of candidacy | Q33929608 | ||
What is GABAergic inhibition? How is it modified in epilepsy? | Q34041405 | ||
Localization of brain-derived neurotrophic factor to distinct terminals of mossy fiber axons implies regulation of both excitation and feedforward inhibition of CA3 pyramidal cells | Q34314205 | ||
"Epileptic neurons" in temporal lobe epilepsy | Q34604299 | ||
Temporal lobe epilepsy -current wisdom | Q34961163 | ||
Small conductance Ca2+-activated K+ channels as targets of CNS drug development | Q35793863 | ||
Impaired and repaired inhibitory circuits in the epileptic human hippocampus | Q36146236 | ||
Clinical aspects of temporal/limbic epilepsy and their relationships to intractability. | Q36355818 | ||
Deficit of Kcnma1 mRNA expression in the dentate gyrus of epileptic rats. | Q37000100 | ||
Differential changes in mGlu2 and mGlu3 gene expression following pilocarpine-induced status epilepticus: a comparative real-time PCR analysis | Q37102095 | ||
Mechanism of increased BK channel activation from a channel mutation that causes epilepsy. | Q37124257 | ||
Synergistic roles of GABAA receptors and SK channels in regulating thalamocortical oscillations | Q37265945 | ||
Molecular and cellular basis of epileptogenesis in symptomatic epilepsy | Q37286638 | ||
Hippocampal epileptogenesis in animal models of mesial temporal lobe epilepsy with hippocampal sclerosis: the importance of the "latent period" and other concepts | Q37352151 | ||
Functions and modulation of neuronal SK channels | Q37572459 | ||
Activation of SK channels inhibits epileptiform bursting in hippocampal CA3 neurons. | Q40342294 | ||
Specific enhancement of SK channel activity selectively potentiates the afterhyperpolarizing current I(AHP) and modulates the firing properties of hippocampal pyramidal neurons. | Q40359725 | ||
The pilocarpine model of epilepsy | Q40463975 | ||
How long does it take for partial epilepsy to become intractable? | Q40608293 | ||
The pathophysiology of human mesial temporal lobe epilepsy | Q40615836 | ||
Small conductance Ca2+-activated K+ channels and calmodulin: cell surface expression and gating. | Q40649619 | ||
Pathogenesis of mesial temporal sclerosis. | Q40772151 | ||
Pharmacological characterisation of the human small conductance calcium-activated potassium channel hSK3 reveals sensitivity to tricyclic antidepressants and antipsychotic phenothiazines | Q40804141 | ||
Excitation and inhibition in epilepsy | Q41156248 | ||
Mechanisms of seizure-induced 'transcriptional channelopathy' of hyperpolarization-activated cyclic nucleotide gated (HCN) channels | Q42148855 | ||
Seizures produced by pilocarpine in mice: a behavioral, electroencephalographic and morphological analysis | Q42250713 | ||
Comparative immunohistochemical distribution of three small-conductance Ca2+-activated potassium channel subunits, SK1, SK2, and SK3 in mouse brain | Q42464133 | ||
Upregulation of a T-type Ca2+ channel causes a long-lasting modification of neuronal firing mode after status epilepticus. | Q43972759 | ||
Regional differences in distribution and functional expression of small-conductance Ca2+-activated K+ channels in rat brain. | Q44213481 | ||
The small conductance Ca2+-activated K+ channel SK3 is localized in nerve terminals of excitatory synapses of cultured mouse hippocampal neurons | Q44326165 | ||
SKCa channels mediate the medium but not the slow calcium-activated afterhyperpolarization in cortical neurons. | Q44835120 | ||
A novel mechanism for the facilitation of theta-induced long-term potentiation by brain-derived neurotrophic factor. | Q44919593 | ||
Acquired dendritic channelopathy in temporal lobe epilepsy | Q44992557 | ||
Protein kinase CK2 is coassembled with small conductance Ca(2+)-activated K+ channels and regulates channel gating | Q45055667 | ||
Circuit mechanisms of seizures in the pilocarpine model of chronic epilepsy: cell loss and mossy fiber sprouting. | Q45999480 | ||
Transcriptional upregulation of Cav3.2 mediates epileptogenesis in the pilocarpine model of epilepsy. | Q46082573 | ||
P407 | language of work or name | English | Q1860 |
P1104 | number of pages | 13 | |
P304 | page(s) | 187-199 | |
P577 | publication date | 2010-06-08 | |
P1433 | published in | Brain Research | Q4955782 |
P1476 | title | Altered expression and function of small-conductance (SK) Ca(2+)-activated K+ channels in pilocarpine-treated epileptic rats | |
P478 | volume | 1348 |
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Q64084199 | Epilepsy-Induced Reduction in HCN Channel Expression Contributes to an Increased Excitability in Dorsal, But Not Ventral, Hippocampal CA1 Neurons |
Q28080655 | Homeostasis or channelopathy? Acquired cell type-specific ion channel changes in temporal lobe epilepsy and their antiepileptic potential |
Q45805851 | In vivo treatment with the casein kinase 2 inhibitor 4,5,6,7- tetrabromotriazole augments the slow afterhyperpolarizing potential and prevents acute epileptiform activity |
Q99609172 | Low-frequency Stimulation Decreases Hyperexcitability Through Adenosine A1 Receptors in the Hippocampus of Kindled Rats |
Q37495452 | Overexpression of KCNN3 results in sudden cardiac death |
Q28833834 | Pharmacological gating modulation of small- and intermediate-conductance Ca(2+)-activated K(+) channels (KCa2.x and KCa3.1) |
Q38824328 | Potassium Channels in Epilepsy. |
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Q34639033 | Rapamycin reverses status epilepticus-induced memory deficits and dendritic damage |
Q47674308 | SK channels participate in the formation of after burst hyperpolarization and partly inhibit the burst strength of epileptic ictal discharges |
Q91829563 | Switching between persistent firing and depolarization block in individual rat CA1 pyramidal neurons |
Q93013709 | Transcriptional Regulation of Channelopathies in Genetic and Acquired Epilepsies |
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