LPS-binding protein enables intestinal epithelial restitution despite LPS exposure

scientific article

LPS-binding protein enables intestinal epithelial restitution despite LPS exposure is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1097/MPG.0B013E31823A895A
P932PMC publication ID3288261
P698PubMed publication ID22002480

P2093author name stringHong Huang
Jianjing Xue
John A Bauer
Peter J Giannone
Juli M Richter
Brandon L Schanbacher
P2860cites workEpithelial restitution and wound healing in inflammatory bowel diseaseQ37058859
LPS-binding protein protects mice from septic shock caused by LPS or gram-negative bacteriaQ37381799
LPS-binding protein circulates in association with apoB-containing lipoproteins and enhances endotoxin-LDL/VLDL interactionQ39906364
Increased expression and function of integrins in enterocytes by endotoxin impairs epithelial restitutionQ40436297
Endotoxin inhibits intestinal epithelial restitution through activation of Rho-GTPase and increased focal adhesionsQ40550768
Poly(ADP-ribose) polymerase-1: a novel therapeutic target in necrotizing enterocolitisQ42004598
Lipopolysaccharide (LPS) signal transduction and clearance. Dual roles for LPS binding protein and membrane CD14.Q46059813
Molecular cloning, characterization, and tissue distribution of rat lipopolysaccharide binding protein. Evidence for extrahepatic expression.Q48080940
A critical role for TLR4 in the pathogenesis of necrotizing enterocolitis by modulating intestinal injury and repair.Q53526875
Concordance of bacterial cultures with endotoxin and interleukin-6 in necrotizing enterocolitis.Q54571445
Lipopolysaccharide (LPS)-binding protein mediates LPS detoxification by chylomicronsQ78826897
High concentrations of lipopolysaccharide-binding protein in serum of patients with severe sepsis or septic shock inhibit the lipopolysaccharide response in human monocytesQ24291979
Structure and function of lipopolysaccharide binding proteinQ24324774
Lipopolysaccharide binding protein and serum amyloid A secretion by human intestinal epithelial cells during the acute phase responseQ28142461
Lipopolysaccharide (LPS)-binding protein inhibits responses to cell-bound LPSQ28204864
Lipopolysaccharide-binding protein is required to combat a murine gram-negative bacterial infectionQ28252006
CD14, a receptor for complexes of lipopolysaccharide (LPS) and LPS binding proteinQ29615189
The roles of bacteria and TLR4 in rat and murine models of necrotizing enterocolitisQ33254555
Mucosal repair in the gastrointestinal tractQ35195644
Toll-like Receptors and Their Signaling MechanismsQ35585033
Treatment and prevention of necrotizing enterocolitisQ35682601
Modulatory effects of sCD14 and LBP on LPS-host cell interactionsQ36263729
The role of lipopolysaccharide-binding protein in modulating the innate immune responseQ36398930
Intestinal immune defences and the inflammatory response in necrotising enterocolitisQ36475697
The role of the intestinal barrier in the pathogenesis of necrotizing enterocolitis.Q36708828
P433issue5
P407language of work or nameEnglishQ1860
P304page(s)639-644
P577publication date2012-05-01
P1433published inJournal of Pediatric Gastroenterology and NutritionQ6295721
P1476titleLPS-binding protein enables intestinal epithelial restitution despite LPS exposure
P478volume54

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cites work (P2860)
Q37122290Cardiolipins Act as a Selective Barrier to Toll-Like Receptor 4 Activation in the Intestine.
Q47642289High-fat, high-fructose, high-cholesterol feeding causes severe NASH and cecal microbiota dysbiosis in juvenile Ossabaw swine.
Q36914228Innate immune signaling in the pathogenesis of necrotizing enterocolitis.
Q35047413Necrotizing enterocolitis in newborns: update in pathophysiology and newly emerging therapeutic strategies
Q91811038The Post-amyloid Era in Alzheimer's Disease: Trust Your Gut Feeling
Q37683363Toll-like receptor 4-mediated endoplasmic reticulum stress in intestinal crypts induces necrotizing enterocolitis.
Q34013912Update in pathogenesis and prospective in treatment of necrotizing enterocolitis.
Q55060330Wogonin suppresses inflammatory response and maintains intestinal barrier function via TLR4-MyD88-TAK1-mediated NF-κB pathway in vitro.

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