Mutual rescues between two dominant negative mutations in cardiac troponin I and cardiac troponin T

scientific article

Mutual rescues between two dominant negative mutations in cardiac troponin I and cardiac troponin T is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1074/JBC.M110.137844
P932PMC publication ID2934648
P698PubMed publication ID20551314

P2093author name stringJ-P Jin
Bin Wei
Jimin Gao
Xu-Pei Huang
P2860cites workStructure of the core domain of human cardiac troponin in the Ca(2+)-saturated formQ24306847
Troponin I: inhibitor or facilitatorQ28140058
The highly conserved COOH terminus of troponin I forms a Ca2+-modulated allosteric domain in the troponin complexQ28186310
Calcium, thin filaments, and the integrative biology of cardiac contractilityQ28234995
To investigate protein evolution by detecting suppressed epitope structuresQ28241204
Ca(2+)-regulated structural changes in troponinQ28241337
Myofilament incorporation determines the stoichiometry of troponin I in transgenic expression and the rescue of a null mutationQ28244574
Selective deletion of the NH2-terminal variable region of cardiac troponin T in ischemia reperfusion by myofibril-associated mu-calpain cleavageQ28264199
Isoform diversity, regulation, and functional adaptation of troponin and calponinQ28270406
Troponin T: genetics, properties and functionQ28282871
Restricted N-terminal truncation of cardiac troponin T: a novel mechanism for functional adaptation to energetic crisisQ28283976
Localization of the two tropomyosin-binding sites of troponin TQ28284494
Alternative RNA splicing-generated cardiac troponin T isoform switching: a non-heart-restricted genetic programming synchronized in developing cardiac and skeletal musclesQ28288140
Removal of the N-terminal extension of cardiac troponin I as a functional compensation for impaired myocardial beta-adrenergic signalingQ28295123
Proteolytic N-terminal truncation of cardiac troponin I enhances ventricular diastolic functionQ28299362
Cardiac troponin I gene knockout: a mouse model of myocardial troponin I deficiencyQ28594884
Regulation of contraction in striated muscleQ28610114
Myosin crossbridge activation of cardiac thin filaments: implications for myocardial function in health and diseaseQ35785986
Specific myosin heavy chain mutations suppress troponin I defects in Drosophila musclesQ36288309
Sarcomeric proteins and familial hypertrophic cardiomyopathy: linking mutations in structural proteins to complex cardiovascular phenotypesQ36369925
Tropomyosin and troponin cooperativity on the thin filamentQ36728169
Exon skipping in cardiac troponin T of turkeys with inherited dilated cardiomyopathyQ38291314
Thin filament-mediated regulation of cardiac contractionQ41117735
Cardiac troponin T variants produced by aberrant splicing of multiple exons in animals with high instances of dilated cardiomyopathyQ44177990
An R111C polymorphism in wild turkey cardiac troponin I accompanying the dilated cardiomyopathy-related abnormal splicing variant of cardiac troponin T with potentially compensatory effectsQ44737279
Influence of substrate supply on cardiac efficiency, as measured by pressure-volume analysis in ex vivo mouse heartsQ46375363
Conformational modulation of slow skeletal muscle troponin T by an NH(2)-terminal metal-binding extension.Q52165026
P4510describes a project that usesImageJQ1659584
P433issue36
P407language of work or nameEnglishQ1860
P1104number of pages11
P304page(s)27806-27816
P577publication date2010-06-15
P1433published inJournal of Biological ChemistryQ867727
P1476titleMutual rescues between two dominant negative mutations in cardiac troponin I and cardiac troponin T
P478volume285