scholarly article | Q13442814 |
P50 | author | Roberto Romero | Q42734915 |
Edi Vaisbuch | Q47805338 | ||
P2093 | author name string | S K Kim | |
O Erez | |||
N G Than | |||
Y Sorokin | |||
J P Kusanovic | |||
F Gotsch | |||
P Mittal | |||
S Mazaki-Tovi | |||
S S Hassan | |||
L Yeo | |||
T Chaiworapongsa | |||
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Soluble endoglin contributes to the pathogenesis of preeclampsia | Q28244053 | ||
Hypertension produced by placental ischemia in pregnant rats is associated with increased soluble endoglin expression | Q28573546 | ||
Excess placental soluble fms-like tyrosine kinase 1 (sFlt1) may contribute to endothelial dysfunction, hypertension, and proteinuria in preeclampsia | Q29615916 | ||
Circulatory soluble endoglin and its predictive value for preeclampsia in second-trimester pregnancies with abnormal uterine perfusion | Q33316902 | ||
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Identification of patients at risk for early onset and/or severe preeclampsia with the use of uterine artery Doppler velocimetry and placental growth factor | Q36360704 | ||
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The two stage model of preeclampsia: variations on the theme | Q37187589 | ||
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The frequency and severity of placental findings in women with preeclampsia are gestational age dependent | Q47414750 | ||
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Morphometric placental villous and vascular abnormalities in early- and late-onset pre-eclampsia with and without fetal growth restriction. | Q50734891 | ||
Alteration of serum soluble endoglin levels after the onset of preeclampsia is more pronounced in women with early-onset. | Q51865183 | ||
Unexplained fetal death: another anti-angiogenic state. | Q51909063 | ||
Normal pregnancy and preeclampsia both produce inflammatory changes in peripheral blood leukocytes akin to those of sepsis. | Q52237839 | ||
Soluble endoglin and other circulating antiangiogenic factors in preeclampsia. | Q53600812 | ||
Doppler velocimetry studies of the uterine artery in the early prediction of pre-eclampsia and intra-uterine growth retardation. | Q54283587 | ||
Placental stress and pre-eclampsia: a revised view. | Q54774592 | ||
A high uterine artery pulsatility index reflects a defective development of placental bed spiral arteries in pregnancies complicated by hypertension and fetal growth retardation | Q56444585 | ||
Placental angiogenic growth factors and uterine artery Doppler findings for characterization of different subsets in preeclampsia and in isolated intrauterine growth restriction | Q57316953 | ||
Excess syncytiotrophoblast microparticle shedding is a feature of early-onset pre-eclampsia, but not normotensive intrauterine growth restriction | Q58623928 | ||
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Plasma soluble vascular endothelial growth factor receptor-1 concentration is elevated prior to the clinical diagnosis of pre-eclampsia | Q58778301 | ||
The maternal plasma soluble vascular endothelial growth factor receptor-1 concentration is elevated in SGA and the magnitude of the increase relates to Doppler abnormalities in the maternal and fetal circulation | Q61845344 | ||
Early and Late Preeclampsia | Q61853974 | ||
Recombinant vascular endothelial growth factor 121 attenuates hypertension and improves kidney damage in a rat model of preeclampsia | Q64377002 | ||
Doppler flow measurements and histomorphology of the placental bed in uteroplacental insufficiency | Q68013154 | ||
Normal values of Pulsatility Index from fetal vessels: a cross-sectional study on 1556 healthy fetuses | Q68533648 | ||
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A United States national reference for fetal growth | Q70895060 | ||
Doppler ultrasound screening as part of routine antenatal scanning: prediction of pre-eclampsia and intrauterine growth retardation | Q72634270 | ||
Apoptosis cascade progresses during turnover of human trophoblast: analysis of villous cytotrophoblast and syncytial fragments in vitro | Q73314016 | ||
Risk factors associated with preeclampsia in healthy nulliparous women. The Calcium for Preeclampsia Prevention (CPEP) Study Group | Q73937666 | ||
Activation of leukocytes during the uteroplacental passage in preeclampsia | Q77513220 | ||
Hypoxia is responsible for soluble vascular endothelial growth factor receptor-1 (VEGFR-1) but not for soluble endoglin induction in villous trophoblast | Q81086992 | ||
Angiotensin II type 1 receptor agonistic antibodies reflect fundamental alterations in the uteroplacental vasculature | Q81423834 | ||
First-trimester serum levels of soluble endoglin and soluble fms-like tyrosine kinase-1 as first-trimester markers for late-onset preeclampsia | Q81931104 | ||
Maternal endothelial function and serum concentrations of placental growth factor and soluble endoglin in women with abnormal placentation | Q82054283 | ||
Maternal serum concentration of soluble fms-like tyrosine kinase 1 and vascular endothelial growth factor in women with abnormal uterine artery Doppler and in those with fetal growth restriction | Q83212134 | ||
P433 | issue | 2 | |
P921 | main subject | pre-eclampsia | Q61335 |
P304 | page(s) | 155-162 | |
P577 | publication date | 2010-02-01 | |
P1433 | published in | Ultrasound in Obstetrics and Gynecology | Q1817048 |
P1476 | title | Plasma soluble endoglin concentration in pre-eclampsia is associated with an increased impedance to flow in the maternal and fetal circulations | |
P478 | volume | 35 |
Q47162116 | Altered angiogenesis as a common mechanism underlying preterm birth, small for gestational age, and stillbirth in women living with HIV. |
Q35104545 | Angiogenic factors and uterine Doppler velocimetry in early- and late-onset preeclampsia |
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Q61844895 | ELABELA plasma concentrations are increased in women with late-onset preeclampsia |
Q35547367 | Endocan, a putative endothelial cell marker, is elevated in preeclampsia, decreased in acute pyelonephritis, and unchanged in other obstetrical syndromes |
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Q35362708 | Full-length human placental sFlt-1-e15a isoform induces distinct maternal phenotypes of preeclampsia in mice |
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Q58795465 | Integrated Systems Biology Approach Identifies Novel Maternal and Placental Pathways of Preeclampsia |
Q36107337 | Late-onset preeclampsia is associated with an imbalance of angiogenic and anti-angiogenic factors in patients with and without placental lesions consistent with maternal underperfusion |
Q36062460 | Maternal plasma concentrations of angiogenic/anti-angiogenic factors are of prognostic value in patients presenting to the obstetrical triage area with the suspicion of preeclampsia |
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Q33650241 | Soluble endoglin, transforming growth factor-Beta 1 and soluble tumor necrosis factor alpha receptors in different clinical manifestations of preeclampsia |
Q36015615 | The "Great Obstetrical Syndromes" are associated with disorders of deep placentation |
Q64116833 | The Role of Placental Growth Factor, Soluble Endoglin, and Uterine Artery Diastolic Notch to Predict the Early Onset of Preeclampsia |
Q34150192 | The anti-aging factor α-klotho during human pregnancy and its expression in pregnancies complicated by small-for-gestational-age neonates and/or preeclampsia |
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Q50102668 | The profiles of soluble adhesion molecules in the "great obstetrical syndromes". |
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