Prenatal methylmercury exposure hampers glutathione antioxidant system ontogenesis and causes long-lasting oxidative stress in the mouse brain.

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Prenatal methylmercury exposure hampers glutathione antioxidant system ontogenesis and causes long-lasting oxidative stress in the mouse brain. is …
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scholarly articleQ13442814

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P356DOI10.1016/J.TAAP.2007.10.010
P932PMC publication ID2955629
P698PubMed publication ID18023834
P5875ResearchGate publication ID5823008

P50authorDiogo O SouzaQ39272593
Marcelo FarinaQ42324980
Alcir Luiz DafréQ42731322
Jeferson Luis FrancoQ43117175
Joao Batista da RochaQ47503444
Solange Cristina GarciaQ56333872
Michael AschnerQ64778814
Dejan MilatovicQ84916544
Denise BohrerQ114398573
P2093author name stringAdriana K C Nunes
James Stringari
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SLCO/OATP-like transport of glutathione in FasL-induced apoptosis: glutathione efflux is coupled to an organic anion exchange and is necessary for the progression of the execution phase of apoptosis.Q53610008
Effects of 2,3-dimercapto-1-propanesulfonic acid (DMPS) on methylmercury-induced locomotor deficits and cerebellar toxicity in miceQ58837422
Quantification of F2-isoprostanes as a biomarker of oxidative stressQ59173846
Glutathione reductaseQ70123854
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44 Glutathione peroxidaseQ71070215
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Methylmercury affects multiple subtypes of calcium channels in rat cerebellar granule cellsQ74146022
Mass spectrometric quantification of F2-isoprostanes in biological fluids and tissues as measure of oxidant stressQ77907797
P433issue1
P921main subjectglutathioneQ116907
P304page(s)147-154
P577publication date2007-10-22
P1433published inToxicology and Applied PharmacologyQ2446974
P1476titlePrenatal methylmercury exposure hampers glutathione antioxidant system ontogenesis and causes long-lasting oxidative stress in the mouse brain
P478volume227

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