| scholarly article | Q13442814 |
| P50 | author | Cristina Capanni | Q43166626 |
| Vittoria Cenni | Q55728542 | ||
| Elisabetta Mattioli | Q55728550 | ||
| Maria Rosaria D'Apice | Q57069733 | ||
| Marta Columbaro | Q72997886 | ||
| Nadir Mario Maraldi | Q114449242 | ||
| Manfred Wehnert | Q114449243 | ||
| Michela Ortolani | Q114449244 | ||
| Sabino Prencipe | Q114449246 | ||
| Stefano Squarzoni | Q42412676 | ||
| Giovanna Lattanzi | Q42820424 | ||
| P2093 | author name string | Sandra Marmiroli | |
| Giuseppe Novelli | |||
| Milena Fini | |||
| Jessika Bertacchini | |||
| P2860 | cites work | SIRT1 regulates the histone methyl-transferase SUV39H1 during heterochromatin formation | Q24300487 |
| Prelamin A acts to accelerate smooth muscle cell senescence and is a novel biomarker of human vascular aging | Q24311723 | ||
| Ankrd2/ARPP is a novel Akt2 specific substrate and regulates myogenic differentiation upon cellular exposure to H(2)O(2) | Q24311740 | ||
| Reversal of the cellular phenotype in the premature aging disease Hutchinson-Gilford progeria syndrome | Q24539118 | ||
| Mandibuloacral dysplasia is caused by a mutation in LMNA-encoding lamin A/C | Q24612380 | ||
| The processing pathway of prelamin A | Q28253023 | ||
| Increased mammalian lifespan and a segmental and tissue-specific slowing of aging after genetic reduction of mTOR expression | Q28297737 | ||
| Pseudo-DNA damage response in senescent cells | Q28583844 | ||
| BMAL1-dependent regulation of the mTOR signaling pathway delays aging | Q28592920 | ||
| Hyper-mitogenic drive coexists with mitotic incompetence in senescent cells | Q30533934 | ||
| Hydrogen peroxide stimulates nuclear import of the POU homeodomain protein Oct-1 and its repressive effect on the expression of Cdx-2. | Q30985733 | ||
| Prelamin A accumulation and stress conditions induce impaired Oct-1 activity and autophagy in prematurely aged human mesenchymal stem cell | Q33657305 | ||
| Diseases of the nuclear envelope | Q33687043 | ||
| Loss of lamin B1 results in prolongation of S phase and decondensation of chromosome territories | Q33913256 | ||
| Clinical trial of a farnesyltransferase inhibitor in children with Hutchinson-Gilford progeria syndrome | Q34033699 | ||
| Dissecting the role of mTOR complexes in cellular senescence | Q43205990 | ||
| Lamins are rapamycin targets that impact human longevity: a study in centenarians | Q44421915 | ||
| Analysis of cell cycle by flow cytometry | Q44954209 | ||
| Prelamin A is involved in early steps of muscle differentiation. | Q46296157 | ||
| Loss of ZMPSTE24 (FACE-1) causes autosomal recessive restrictive dermopathy and accumulation of Lamin A precursors. | Q46447976 | ||
| Alterations of nuclear envelope and chromatin organization in mandibuloacral dysplasia, a rare form of laminopathy | Q46620985 | ||
| Drugs affecting prelamin A processing: effects on heterochromatin organization | Q46841230 | ||
| Autophagic degradation of nuclear components in mammalian cells. | Q50590333 | ||
| Different prelamin A forms accumulate in human fibroblasts: a study in experimental models and progeria. | Q51730936 | ||
| Lamin A precursor induces barrier-to-autointegration factor nuclear localization. | Q53316358 | ||
| Pre-Lamin A processing is linked to heterochromatin organization. | Q53538268 | ||
| Oct-1 recruitment to the nuclear envelope in adult-onset autosomal dominant leukodystrophy | Q56978882 | ||
| Osteoblasts from a mandibuloacral dysplasia patient induce human blood precursors to differentiate into active osteoclasts | Q56985192 | ||
| Autophagy is defective in collagen VI muscular dystrophies and its reactivation rescues myofiber degeneration | Q57639972 | ||
| Compound Heterozygosity for Mutations inLMNAin a Patient with a Myopathic and Lipodystrophic Mandibuloacral Dysplasia Type A Phenotype | Q58923930 | ||
| Autophagy induction rescues muscular dystrophy | Q84038786 | ||
| Can autophagy promote longevity? | Q34135551 | ||
| Rapamycin reverses cellular phenotypes and enhances mutant protein clearance in Hutchinson-Gilford progeria syndrome cells | Q34196369 | ||
| Dissecting the dynamic turnover of GFP-LC3 in the autolysosome | Q34577387 | ||
| Combined treatment with statins and aminobisphosphonates extends longevity in a mouse model of human premature aging. | Q34790173 | ||
| Investigating the purpose of prelamin A processing | Q35015891 | ||
| Developmental Control of Late Replication and S Phase Length | Q35024446 | ||
| Autophagic degradation of farnesylated prelamin A as a therapeutic approach to lamin-linked progeria. | Q35771419 | ||
| Cell cycle arrest is not yet senescence, which is not just cell cycle arrest: terminology for TOR-driven aging | Q35947377 | ||
| Altered chromatin organization and SUN2 localization in mandibuloacral dysplasia are rescued by drug treatment. | Q36205365 | ||
| Lamin A/C speckles mediate spatial organization of splicing factor compartments and RNA polymerase II transcription | Q36324279 | ||
| Rapamycin extends lifespan and delays tumorigenesis in heterozygous p53+/- mice | Q36455201 | ||
| Rapamycin reverses elevated mTORC1 signaling in lamin A/C-deficient mice, rescues cardiac and skeletal muscle function, and extends survival. | Q36731585 | ||
| Sp1 transcription factor interaction with accumulated prelamin a impairs adipose lineage differentiation in human mesenchymal stem cells: essential role of sp1 in the integrity of lipid vesicles | Q36861511 | ||
| Involvement of prelamin A in laminopathies | Q36921560 | ||
| Fast regulation of AP-1 activity through interaction of lamin A/C, ERK1/2, and c-Fos at the nuclear envelope | Q36972637 | ||
| Targeting autophagy potentiates tyrosine kinase inhibitor-induced cell death in Philadelphia chromosome-positive cells, including primary CML stem cells | Q37170788 | ||
| Rapamycin suppresses brain aging in senescence-accelerated OXYS rats | Q37298898 | ||
| A-type lamins and signaling: the PI 3-kinase/Akt pathway moves forward | Q37500299 | ||
| Cell cycle arrest is not senescence | Q37836231 | ||
| The intricacy of nuclear membrane dynamics during nucleophagy | Q37843669 | ||
| Regulation of nucleotide excision repair by nuclear lamin b1 | Q39118978 | ||
| The protein kinase Akt/PKB regulates both prelamin A degradation and Lmna gene expression | Q39190465 | ||
| Disruption of lamin B1 and lamin B2 processing and localization by farnesyltransferase inhibitors. | Q39726549 | ||
| Lamin A Ser404 is a nuclear target of Akt phosphorylation in C2C12 cells | Q39937276 | ||
| The R527H mutation in LMNA gene causes an increased sensitivity to ionizing radiation | Q39964614 | ||
| Farnesyltransferase inhibitor treatment restores chromosome territory positions and active chromosome dynamics in Hutchinson-Gilford progeria syndrome cells. | Q39983752 | ||
| Rescue of heterochromatin organization in Hutchinson-Gilford progeria by drug treatment | Q42013273 | ||
| Attenuation of TORC1 signaling delays replicative and oncogenic RAS-induced senescence. | Q42178287 | ||
| Premature aging in mice activates a systemic metabolic response involving autophagy induction. | Q42435408 | ||
| The accumulation of un-repairable DNA damage in laminopathy progeria fibroblasts is caused by ROS generation and is prevented by treatment with N-acetyl cysteine | Q42492466 | ||
| Prelamin A processing and functional effects in restrictive dermopathy | Q42648385 | ||
| Prelamin A-mediated recruitment of SUN1 to the nuclear envelope directs nuclear positioning in human muscle. | Q42735850 | ||
| Stabilization of Suv39H1 by SirT1 is part of oxidative stress response and ensures genome protection | Q42811651 | ||
| mTORC2 protein complex-mediated Akt (Protein Kinase B) Serine 473 Phosphorylation is not required for Akt1 activity in human platelets [corrected]. | Q42813304 | ||
| Altered pre-lamin A processing is a common mechanism leading to lipodystrophy. | Q42815664 | ||
| P433 | issue | 9 | |
| P921 | main subject | sirolimus | Q32089 |
| mandibuloacral dysplasia | Q16968886 | ||
| P304 | page(s) | 755-770 | |
| P577 | publication date | 2014-09-01 | |
| P1433 | published in | Aging | Q2845875 |
| P1476 | title | Rapamycin treatment of Mandibuloacral dysplasia cells rescues localization of chromatin-associated proteins and cell cycle dynamics | |
| P478 | volume | 6 |
| Q36557465 | All-trans retinoic acid and rapamycin normalize Hutchinson Gilford progeria fibroblast phenotype. |
| Q92376988 | Altered adipocyte differentiation and unbalanced autophagy in type 2 Familial Partial Lipodystrophy: an in vitro and in vivo study of adipose tissue browning |
| Q56342217 | Altered modulation of lamin A/C-HDAC2 interaction and p21 expression during oxidative stress response in HGPS |
| Q92713440 | Ankrd2 in Mechanotransduction and Oxidative Stress Response in Skeletal Muscle: New Cues for the Pathogenesis of Muscular Laminopathies |
| Q37085852 | Barrier-to-autointegration factor (BAF) involvement in prelamin A-related chromatin organization changes |
| Q52564206 | Elevated TGF β2 serum levels in Emery-Dreifuss muscular dystrophy: implications for myocyte and tenocyte differentiation and fibrogenic processes. |
| Q64242806 | Hutchinson-Gilford Progeria Syndrome-Current Status and Prospects for Gene Therapy Treatment |
| Q38893099 | Modulation of TGFbeta 2 levels by lamin A in U2-OS osteoblast-like cells: understanding the osteolytic process triggered by altered lamins |
| Q28067037 | Potential therapeutic effects of the MTOR inhibitors for preventing ageing and progeria-related disorders |
| Q42582325 | Prelamin A and Oct-1: a puzzle of aging |
| Q61805009 | Statins and Histone Deacetylase Inhibitors Affect Lamin A/C - Histone Deacetylase 2 Interaction in Human Cells |
| Q64256257 | The Cutting Edge: The Role of mTOR Signaling in Laminopathies |
| Q40994525 | The telomeric protein AKTIP interacts with A- and B-type lamins and is involved in regulation of cellular senescence. |
| Q35149607 | Tumor promoter-induced cellular senescence: cell cycle arrest followed by geroconversion |
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