scholarly article | Q13442814 |
P819 | ADS bibcode | 2014PLoSO...9k3691D |
P356 | DOI | 10.1371/JOURNAL.PONE.0113691 |
P932 | PMC publication ID | 4244142 |
P698 | PubMed publication ID | 25423108 |
P5875 | ResearchGate publication ID | 268746862 |
P50 | author | Renato S Aguiar | Q57863458 |
P2093 | author name string | Constance Oliver | |
Eurico Arruda | |||
Luis L P daSilva | |||
Rodrigo O de Castro | |||
Mara E da Silva-Januário | |||
Maria C Jamur | |||
Elaine Z M da Silva | |||
Julianne V de Carvalho | |||
Paola P Silveira | |||
P2860 | cites work | Exosomal-like vesicles are present in human blood plasma | Q24303407 |
Co-localization of HIV-1 Nef with the AP-2 adaptor protein complex correlates with Nef-induced CD4 down-regulation | Q24532862 | ||
HIV-1 Nef disrupts intracellular trafficking of major histocompatibility complex class I, CD4, CD8, and CD28 by distinct pathways that share common elements | Q24602320 | ||
Secretory mechanisms and intercellular transfer of microRNAs in living cells | Q24603413 | ||
ESCRT complexes and the biogenesis of multivesicular bodies | Q24646021 | ||
The use of lead citrate at high pH as an electron-opaque stain in electron microscopy | Q26778439 | ||
HIV entry: a game of hide-and-fuse? | Q26824979 | ||
T-cells play the classics with a different spin | Q27023460 | ||
How HIV-1 Nef hijacks the AP-2 clathrin adaptor to downregulate CD4 | Q27681494 | ||
Receptor downregulation and multivesicular-body sorting | Q28131720 | ||
HIV-1 Nef downregulates MHC-I by a PACS-1- and PI3K-regulated ARF6 endocytic pathway | Q28202962 | ||
Role of LBPA and Alix in multivesicular liposome formation and endosome organization | Q28240220 | ||
ExoCarta: A compendium of exosomal proteins and RNA | Q28260686 | ||
Genomic structure of an attenuated quasi species of HIV-1 from a blood transfusion donor and recipients | Q28284190 | ||
Exosomes and HIV Gag bud from endosome-like domains of the T cell plasma membrane | Q28301729 | ||
Brief report: absence of intact nef sequences in a long-term survivor with nonprogressive HIV-1 infection | Q28304773 | ||
HIV with reduced sensitivity to zidovudine (AZT) isolated during prolonged therapy | Q28339571 | ||
Dynamic interaction of HIV-1 Nef with the clathrin-mediated endocytic pathway at the plasma membrane. | Q50706347 | ||
HIV-1 Nef protein protects infected primary cells against killing by cytotoxic T lymphocytes. | Q54148550 | ||
Massive Secretion by T Cells Is Caused by HIV Nef in Infected Cells and by Nef Transfer to Bystander Cells | Q59667707 | ||
Ibalizumab: an anti-CD4 monoclonal antibody for the treatment of HIV-1 infection. | Q64966141 | ||
HIV-Nef and AIDS pathogenesis: are we barking up the wrong tree? | Q84602577 | ||
HIV Nef is secreted in exosomes and triggers apoptosis in bystander CD4+ T cells | Q37485148 | ||
Kinetics of expression of multiply spliced RNA in early human immunodeficiency virus type 1 infection of lymphocytes and monocytes | Q37528880 | ||
Characterization of a continuous T-cell line susceptible to the cytopathic effects of the acquired immunodeficiency syndrome (AIDS)-associated retrovirus | Q37546388 | ||
Polarized release of T-cell-receptor-enriched microvesicles at the immunological synapse | Q37629486 | ||
ALIX and the multivesicular endosome: ALIX in Wonderland | Q38167245 | ||
The cytoplasmic domain of CD4 is sufficient for its down-regulation from the cell surface by human immunodeficiency virus type 1 Nef. | Q40040583 | ||
Vpr.A3A chimera inhibits HIV replication | Q40040714 | ||
HIV-1 Nef assembles a Src family kinase-ZAP-70/Syk-PI3K cascade to downregulate cell-surface MHC-I. | Q40051245 | ||
Direct measurement of soluble CD4 binding to human immunodeficiency virus type 1 virions: gp120 dissociation and its implications for virus-cell binding and fusion reactions and their neutralization by soluble CD4. | Q40059306 | ||
Interactions between Nef and AIP1 proliferate multivesicular bodies and facilitate egress of HIV-1. | Q40269589 | ||
Nef-induced alteration of the early/recycling endosomal compartment correlates with enhancement of HIV-1 infectivity | Q40486921 | ||
Late endosomal membranes rich in lysobisphosphatidic acid regulate cholesterol transport | Q40918547 | ||
A lipid associated with the antiphospholipid syndrome regulates endosome structure and function | Q41056430 | ||
The simian immunodeficiency virus Nef protein promotes degradation of CD4 in human T cells. | Q41488065 | ||
Exosomes from breast milk inhibit HIV-1 infection of dendritic cells and subsequent viral transfer to CD4+ T cells. | Q42233385 | ||
Induction of HTLV-III/LAV from a nonvirus-producing T-cell line: implications for latency | Q44325918 | ||
The tyrosine binding pocket in the adaptor protein 1 (AP-1) mu1 subunit is necessary for Nef to recruit AP-1 to the major histocompatibility complex class I cytoplasmic tail | Q46855565 | ||
Maternal Pumilio acts together with Nanos in germline development in Drosophila embryos | Q47070377 | ||
The low affinity IgG receptor Fc gamma RIIB contributes to the binding of the mast cell specific antibody, mAb BGD6 | Q28579563 | ||
Membrane vesicles as conveyors of immune responses | Q29547765 | ||
Isolation and characterization of exosomes from cell culture supernatants and biological fluids | Q29615035 | ||
Ceramide triggers budding of exosome vesicles into multivesicular endosomes | Q29616811 | ||
Importance of the nef gene for maintenance of high virus loads and for development of AIDS | Q29618369 | ||
Infection of HTLV-III/LAV in HTLV-I-carrying cells MT-2 and MT-4 and application in a plaque assay | Q29619255 | ||
Association of human immunodeficiency virus Nef protein with actin is myristoylation dependent and influences its subcellular localization. | Q30176459 | ||
Cell-surface expression of CD4 reduces HIV-1 infectivity by blocking Env incorporation in a Nef- and Vpu-inhibitable manner | Q33333829 | ||
HIV-1 Nef targets MHC-I and CD4 for degradation via a final common beta-COP-dependent pathway in T cells | Q33363091 | ||
Soluble CD4 and CD4-mimetic compounds inhibit HIV-1 infection by induction of a short-lived activated state | Q33426352 | ||
High concentrations of recombinant soluble CD4 are required to neutralize primary human immunodeficiency virus type 1 isolates | Q33760671 | ||
Human immunodeficiency virus-1 Nef expression induces intracellular accumulation of multivesicular bodies and major histocompatibility complex class II complexes: potential role of phosphatidylinositol 3-kinase | Q33763110 | ||
Interaction of HIV-1 Nef protein with the host protein Alix promotes lysosomal targeting of CD4 receptor. | Q34283497 | ||
RETRACTED: Pregnancy-associated exosomes and their modulation of T cell signaling | Q34486229 | ||
HIV-1 coreceptors and their inhibitors | Q34506596 | ||
Exosome function: from tumor immunology to pathogen biology | Q34760438 | ||
Exosome release of β-catenin: a novel mechanism that antagonizes Wnt signaling | Q35005587 | ||
ADP ribosylation factor 1 activity is required to recruit AP-1 to the major histocompatibility complex class I (MHC-I) cytoplasmic tail and disrupt MHC-I trafficking in HIV-1-infected primary T cells | Q35531422 | ||
High frequency of defective nef alleles in a long-term survivor with nonprogressive human immunodeficiency virus type 1 infection. | Q35872157 | ||
Sphingolipid-modulated exosome secretion promotes clearance of amyloid-β by microglia | Q35879557 | ||
HIV-1 Nef-induced down-regulation of MHC class I requires AP-1 and clathrin but not PACS-1 and is impeded by AP-2. | Q35949112 | ||
Nef proteins encoded by human and simian immunodeficiency viruses induce the accumulation of endosomes and lysosomes in human T cells | Q35965278 | ||
HIV-1 Nef disrupts MHC-I trafficking by recruiting AP-1 to the MHC-I cytoplasmic tail | Q36322591 | ||
Short-range exosomal transfer of viral RNA from infected cells to plasmacytoid dendritic cells triggers innate immunity | Q36343260 | ||
Downregulation of cell-surface CD4 expression by simian immunodeficiency virus Nef prevents viral super infection | Q36361799 | ||
The human immunodeficiency virus-1 nef gene product: a positive factor for viral infection and replication in primary lymphocytes and macrophages | Q36362652 | ||
Cooperative binding of the class I major histocompatibility complex cytoplasmic domain and human immunodeficiency virus type 1 Nef to the endosomal AP-1 complex via its mu subunit | Q36424069 | ||
Human immunodeficiency virus type 1 Nef: adapting to intracellular trafficking pathways | Q36499676 | ||
Human immunodeficiency virus type 1 Nef-induced down-modulation of CD4 is due to rapid internalization and degradation of surface CD4. | Q36625495 | ||
Two mechanisms of soluble CD4 (sCD4)-mediated inhibition of human immunodeficiency virus type 1 (HIV-1) infectivity and their relation to primary HIV-1 isolates with reduced sensitivity to sCD4. | Q36643604 | ||
Analysis of human immunodeficiency virus type 1 nef gene sequences present in vivo | Q36650992 | ||
HIV-1 Nef binds PACS-2 to assemble a multikinase cascade that triggers major histocompatibility complex class I (MHC-I) down-regulation: analysis using short interfering RNA and knock-out mice. | Q36727308 | ||
HIV-1 Nef: at the crossroads | Q36926697 | ||
A basic patch on alpha-adaptin is required for binding of human immunodeficiency virus type 1 Nef and cooperative assembly of a CD4-Nef-AP-2 complex | Q37110624 | ||
Human immunodeficiency virus type 1 Nef protein targets CD4 to the multivesicular body pathway. | Q37232786 | ||
P275 | copyright license | Creative Commons Attribution 4.0 International | Q20007257 |
P6216 | copyright status | copyrighted | Q50423863 |
P4510 | describes a project that uses | ImageJ | Q1659584 |
P433 | issue | 11 | |
P407 | language of work or name | English | Q1860 |
P304 | page(s) | e113691 | |
P577 | publication date | 2014-11-25 | |
P1433 | published in | PLOS One | Q564954 |
P1476 | title | Nef neutralizes the ability of exosomes from CD4+ T cells to act as decoys during HIV-1 infection | |
P478 | volume | 9 |
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Q27309088 | Exosomes are unlikely involved in intercellular Nef transfer |
Q26765283 | Exosomes in Human Immunodeficiency Virus Type I Pathogenesis: Threat or Opportunity? |
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