| review article | Q7318358 |
| scholarly article | Q13442814 |
| P356 | DOI | 10.1080/10428190210196 |
| P698 | PubMed publication ID | 11908737 |
| P50 | author | Rotraud Wieser | Q57070700 |
| P2093 | author name string | Rotraud Wieser | |
| P2860 | cites work | The evi-1 oncoprotein inhibits c-Jun N-terminal kinase and prevents stress-induced cell death | Q22254262 |
| Molecular genetics of neurofibromatosis type 1 (NF1) | Q24518449 | ||
| Activation of EVI1 gene expression in human acute myelogenous leukemias by translocations spanning 300-400 kilobases on chromosome band 3q26 | Q24561670 | ||
| The oncoprotein Evi-1 represses TGF-beta signalling by inhibiting Smad3 | Q28276517 | ||
| Rescue of the embryonic lethal hematopoietic defect reveals a critical role for GATA-2 in urogenital development | Q28506988 | ||
| An early haematopoietic defect in mice lacking the transcription factor GATA-2 | Q28593072 | ||
| Transcription factor GATA-2 gene is located near 3q21 breakpoints in myeloid leukemia. | Q33907561 | ||
| Differential expression and functional role of GATA-2, NF-E2, and GATA-1 in normal adult hematopoiesis | Q34219129 | ||
| Pluripotent hematopoietic stem cells contain high levels of mRNA for c-kit, GATA-2, p45 NF-E2, and c-myb and low levels or no mRNA for c-fms and the receptors for granulocyte colony-stimulating factor and interleukins 5 and 7. | Q34349127 | ||
| Abnormalities of 3q21 and 3q26 in myeloid malignancy: a United Kingdom Cancer Cytogenetic Group study | Q34369958 | ||
| Consistent intergenic splicing and production of multiple transcripts between AML1 at 21q22 and unrelated genes at 3q26 in (3;21)(q26;q22) translocations | Q35210567 | ||
| Loss of erythropoietin responsiveness in erythroid progenitors due to expression of the Evi-1 myeloid-transforming gene | Q36413795 | ||
| The Evi-1 proto-oncogene encodes a transcriptional repressor activity associated with transformation | Q38348524 | ||
| Acute leukemia with structural rearrangements of chromosome 3. | Q40548678 | ||
| Clinical, haematological and cytogenetic features in 24 patients with structural rearrangements of the Q arm of chromosome 3. | Q40877437 | ||
| Evi-1 ZF1 DNA binding activity and a second distinct transcriptional repressor region are both required for optimal transformation of Rat1 fibroblasts | Q41035934 | ||
| Establishment of a novel human myeloid leukaemia cell line (HNT-34) with t(3;3)(q21;q26), t(9;22)(q34;q11) and the expression of EVI1 gene, P210 and P190 BCR/ABL chimaeric transcripts from a patient with AML after MDS with 3q21q26 syndrome | Q41097117 | ||
| Expression of the Zn finger gene, EVI-1, in acute promyelocytic leukemia | Q41131815 | ||
| The SCL/TAL1 gene: roles in normal and malignant haematopoiesis | Q41538641 | ||
| The search for genetic clues in chronic lymphocytic leukemia | Q41705051 | ||
| The leukemia-associated gene MDS1/EVI1 is a new type of GATA-binding transactivator | Q42803226 | ||
| Leukemia breakpoint region in 3q21 is gene rich | Q48047708 | ||
| Chromosomal abnormality inv(3)(q21q26) associated with multilineage hematopoietic progenitor cells in hematopoietic malignancies | Q51102350 | ||
| Acute leukemia with abnormal thrombopoiesis and inversions of chromosome 3. | Q54352042 | ||
| Clinical correlations of the 3q21;q26 cytogenetic anomaly. A leukemic or myelodysplastic syndrome with preserved or increased platelet production and lack of response to cytotoxic drug therapy | Q70043860 | ||
| Cloning of breakpoints in 3q21 associated with hematologic malignancy | Q72112506 | ||
| Pattern of expression and their clinical implications of the GATA family, stem cell leukemia gene, and EVI1 in leukemia and myelodysplastic syndromes | Q73068648 | ||
| Erythroid defects and increased retrovirally-induced tumor formation in Evi1 transgenic mice | Q73166088 | ||
| Forced expression of the leukemia-associated gene EVI1 in ES cells: a model for myeloid leukemia with 3q26 rearrangements | Q73170962 | ||
| Rapid induction of B-cell lymphomas in mice carrying a human IgH/c-mycYAC | Q73404453 | ||
| EVI1 expression in acute myeloid leukaemia | Q73416593 | ||
| Identification of breakpoint cluster regions at 1p36.3 and 3q21 in hematologic malignancies with t(1;3)(p36;q21) | Q73459793 | ||
| Low curative potential of bone marrow transplantation for highly aggressive acute myelogenous leukemia with inversioin inv (3)(q21q26) or homologous translocation t(3;3) (q21;q26) | Q74244056 | ||
| Mapping of leukaemia-associated breakpoints in chromosome band 3q21 using a newly established PAC contig | Q74260823 | ||
| Expression of the transcription factor Evi-1 in human erythroleukemia cell lines and in leukemias | Q95823971 | ||
| P433 | issue | 1 | |
| P921 | main subject | leukemia | Q29496 |
| P304 | page(s) | 59-65 | |
| P577 | publication date | 2002-01-01 | |
| P1433 | published in | Leukemia & Lymphoma | Q6534493 |
| P1476 | title | Rearrangements of chromosome band 3q21 in myeloid leukemia | |
| P478 | volume | 43 |
| Q38515835 | 3q26/EVI1 rearrangements in myeloid hemopathies: a cytogenetic review |
| Q53517480 | Biology and clinical features of myeloid neoplasms with inv(3)(q21q26) or t(3;3)(q21q26). |
| Q56965502 | Complex chromosomal rearrangements leading to MECOM overexpression are recurrent in myeloid malignancies with various 3q abnormalities |
| Q40099013 | Distinct classes of chromosomal rearrangements create oncogenic ETS gene fusions in prostate cancer |
| Q36585592 | Molecular biology in acute leukemia |
| Q34288682 | Molecular characterization of a t(1;3)(p36;q21) in a patient with MDS. MEL1 is widely expressed in normal tissues, including bone marrow, and it is not overexpressed in the t(1;3) cells. |
| Q44893063 | Molecular heterogeneity in AML/MDS patients with 3q21q26 rearrangements. |
| Q35063883 | New molecular methods for classification, diagnosis and therapy prediction of hematological malignancies |
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