| scholarly article | Q13442814 |
| P819 | ADS bibcode | 2014PNAS..11117809Y |
| P356 | DOI | 10.1073/PNAS.1414004111 |
| P8608 | Fatcat ID | release_fbf42nayindhbbm5xovmnedrs4 |
| P932 | PMC publication ID | 4273402 |
| P698 | PubMed publication ID | 25453086 |
| P5875 | ResearchGate publication ID | 269178597 |
| P50 | author | Jozsef Gal | Q57653594 |
| P2093 | author name string | Jing Chen | |
| Haining Zhu | |||
| Liuqing Yang | |||
| P2860 | cites work | The RIP1/RIP3 necrosome forms a functional amyloid signaling complex required for programmed necrosis | Q24293754 |
| Fusion of the dominant negative transcription regulator CHOP with a novel gene FUS by translocation t(12;16) in malignant liposarcoma | Q24304506 | ||
| Fusion of CHOP to a novel RNA-binding protein in human myxoid liposarcoma | Q24309359 | ||
| MAVS forms functional prion-like aggregates to activate and propagate antiviral innate immune response | Q24316320 | ||
| TLS-ERG leukemia fusion protein inhibits RNA splicing mediated by serine-arginine proteins | Q24554236 | ||
| Stress granule assembly is mediated by prion-like aggregation of TIA-1 | Q24559953 | ||
| Mutations in prion-like domains in hnRNPA2B1 and hnRNPA1 cause multisystem proteinopathy and ALS | Q24629495 | ||
| ALS-associated fused in sarcoma (FUS) mutations disrupt Transportin-mediated nuclear import | Q24630100 | ||
| Induced ncRNAs allosterically modify RNA-binding proteins in cis to inhibit transcription | Q24646744 | ||
| Refined crystal structure of DsRed, a red fluorescent protein from coral, at 2.0-A resolution | Q27629931 | ||
| Structural rearrangements near the chromophore influence the maturation speed and brightness of DsRed variants | Q27648898 | ||
| Edc3p and a glutamine/asparagine-rich domain of Lsm4p function in processing body assembly in Saccharomyces cerevisiae | Q27930894 | ||
| Mutations in the FUS/TLS gene on chromosome 16 cause familial amyotrophic lateral sclerosis | Q28236796 | ||
| Mutations in FUS, an RNA processing protein, cause familial amyotrophic lateral sclerosis type 6 | Q28236805 | ||
| TLS/FUS fusion domain of TLS/FUS-erg chimeric protein resulting from the t(16;21) chromosomal translocation in human myeloid leukemia functions as a transcriptional activation domain | Q28241486 | ||
| TDP-43 mutations in familial and sporadic amyotrophic lateral sclerosis | Q28270779 | ||
| Stress granules: the Tao of RNA triage | Q29615263 | ||
| Blessings in disguise: biological benefits of prion-like mechanisms | Q30428155 | ||
| FUS binds the CTD of RNA polymerase II and regulates its phosphorylation at Ser2. | Q30456334 | ||
| RNA seeds higher-order assembly of FUS protein | Q30457170 | ||
| FUS is sequestered in nuclear aggregates in ALS patient fibroblasts | Q30458567 | ||
| Phosphorylation-regulated binding of RNA polymerase II to fibrous polymers of low-complexity domains | Q30577537 | ||
| The in vivo minigene approach to analyze tissue-specific splicing | Q33883223 | ||
| Molecular determinants and genetic modifiers of aggregation and toxicity for the ALS disease protein FUS/TLS | Q33889532 | ||
| Aplysia CPEB can form prion-like multimers in sensory neurons that contribute to long-term facilitation. | Q34097850 | ||
| The tip of the iceberg: RNA-binding proteins with prion-like domains in neurodegenerative disease. | Q34263380 | ||
| Mutant FUS proteins that cause amyotrophic lateral sclerosis incorporate into stress granules | Q34308339 | ||
| Nuclear localization sequence of FUS and induction of stress granules by ALS mutants. | Q34384744 | ||
| The RNA-binding protein fused in sarcoma (FUS) functions downstream of poly(ADP-ribose) polymerase (PARP) in response to DNA damage | Q34801900 | ||
| Compromised paraspeckle formation as a pathogenic factor in FUSopathies | Q35066217 | ||
| A yeast functional screen predicts new candidate ALS disease genes | Q35641365 | ||
| Transcriptional activation by TAL1 and FUS-CHOP proteins expressed in acute malignancies as a result of chromosomal abnormalities. | Q35669591 | ||
| Divergent roles of ALS-linked proteins FUS/TLS and TDP-43 intersect in processing long pre-mRNAs | Q36650975 | ||
| Stress granules as crucibles of ALS pathogenesis. | Q36804082 | ||
| Genome wide array analysis indicates that an amyotrophic lateral sclerosis mutation of FUS causes an early increase of CAMK2N2 in vitro. | Q36920326 | ||
| Genome-wide profiling of salt fractions maps physical properties of chromatin | Q37142037 | ||
| ALS-associated mutation FUS-R521C causes DNA damage and RNA splicing defects | Q37609639 | ||
| FUsed in sarcoma is a novel regulator of manganese superoxide dismutase gene transcription. | Q37617619 | ||
| TDP-43 and FUS/TLS: emerging roles in RNA processing and neurodegeneration | Q37733168 | ||
| Intranuclear aggregation of mutant FUS/TLS as a molecular pathomechanism of amyotrophic lateral sclerosis | Q39054098 | ||
| The self-interaction of native TDP-43 C terminus inhibits its degradation and contributes to early proteinopathies. | Q39369902 | ||
| Interaction of FUS and HDAC1 regulates DNA damage response and repair in neurons. | Q41469956 | ||
| P433 | issue | 50 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | self-assembly | Q910150 |
| protein homooligomerization | Q14860815 | ||
| FUS RNA binding protein | Q21118098 | ||
| P304 | page(s) | 17809-17814 | |
| P577 | publication date | 2014-12-01 | |
| P1433 | published in | Proceedings of the National Academy of Sciences of the United States of America | Q1146531 |
| P1476 | title | Self-assembled FUS binds active chromatin and regulates gene transcription | |
| P478 | volume | 111 |
| Q93222043 | ALS mutations of FUS suppress protein translation and disrupt the regulation of nonsense-mediated decay |
| Q35944221 | Altered mRNP granule dynamics in FTLD pathogenesis. |
| Q33834492 | An obligatory role for neurotensin in high-fat-diet-induced obesity |
| Q41277725 | Clinical and experimental studies of a novel P525R FUS mutation in amyotrophic lateral sclerosis |
| Q90428731 | Conditional depletion of Fus in oligodendrocytes leads to motor hyperactivity and increased myelin deposition associated with Akt and cholesterol activation |
| Q47351811 | Detergent Insoluble Proteins and Inclusion Body-Like Structures Immunoreactive for PRKDC/DNA-PK/DNA-PKcs, FTL, NNT, and AIFM1 in the Amygdala of Cognitively Impaired Elderly Persons. |
| Q49911437 | Dynamic Cycling of t-SNARE Acylation Regulates Platelet Exocytosis |
| Q47603767 | Extracellular vesicles released by cardiomyocytes in a doxorubicin-induced cardiac injury mouse model contain protein biomarkers of early cardiac injury |
| Q64101978 | FUS (fused in sarcoma) is a component of the cellular response to topoisomerase I-induced DNA breakage and transcriptional stress |
| Q57029061 | FUS Negatively Regulates Kaposi's Sarcoma-Associated Herpesvirus Gene Expression |
| Q52369373 | FUS Regulates Activity of MicroRNA-Mediated Gene Silencing. |
| Q37330498 | FUS interacts with nuclear matrix-associated protein SAFB1 as well as Matrin3 to regulate splicing and ligand-mediated transcription |
| Q36147578 | Human metapneumovirus Induces Reorganization of the Actin Cytoskeleton for Direct Cell-to-Cell Spread |
| Q39661461 | Hyperamylinemia Increases IL-1β Synthesis in the Heart via Peroxidative Sarcolemmal Injury |
| Q37037394 | Intraneuronal Amylin Deposition, Peroxidative Membrane Injury and Increased IL-1β Synthesis in Brains of Alzheimer's Disease Patients with Type-2 Diabetes and in Diabetic HIP Rats |
| Q100693811 | LINC01106 drives colorectal cancer growth and stemness through a positive feedback loop to regulate the Gli family factors |
| Q28084859 | Legal but lethal: functional protein aggregation at the verge of toxicity |
| Q102053796 | Molecular structure and interactions within amyloid-like fibrils formed by a low-complexity protein sequence from FUS |
| Q55038139 | Nuclear egress of TDP-43 and FUS occurs independently of Exportin-1/CRM1. |
| Q48411710 | O-GlcNAc glycosylation stoichiometry of the FET protein family: only EWS is glycosylated with a high stoichiometry |
| Q60045951 | Phosphorylation of nuclear Tau is modulated by distinct cellular pathways |
| Q42378053 | Phosphorylation of the FUS low-complexity domain disrupts phase separation, aggregation, and toxicity. |
| Q38789192 | Physiological functions and pathobiology of TDP-43 and FUS/TLS proteins |
| Q41633788 | Proteomic analysis of FUS interacting proteins provides insights into FUS function and its role in ALS. |
| Q38680217 | Quantitative computational models of molecular self-assembly in systems biology |
| Q89628567 | RNA Droplets |
| Q33683022 | RRM domain of ALS/FTD-causing FUS characteristic of irreversible unfolding spontaneously self-assembles into amyloid fibrils |
| Q90862545 | Regulation of zebrafish dorsoventral patterning by phase separation of RNA-binding protein Rbm14 |
| Q36172715 | Residue-by-Residue View of In Vitro FUS Granules that Bind the C-Terminal Domain of RNA Polymerase II |
| Q35678009 | Subcellular localization and RNAs determine FUS architecture in different cellular compartments. |
| Q36386975 | Sulfiredoxin Promotes Colorectal Cancer Cell Invasion and Metastasis through a Novel Mechanism of Enhancing EGFR Signaling |
| Q28553354 | Systematic Proteomic Identification of the Heat Shock Proteins (Hsp) that Interact with Estrogen Receptor Alpha (ERα) and Biochemical Characterization of the ERα-Hsp70 Interaction |
| Q47584390 | TAF15b, involved in the autonomous pathway for flowering, represses transcription of FLOWERING LOCUS C. |
| Q36336278 | TDP-43 and FUS en route from the nucleus to the cytoplasm |
| Q93034876 | The Lyme disease spirochete's BpuR DNA/RNA-binding protein is differentially expressed during the mammal-tick infectious cycle, which affects translation of the SodA superoxide dismutase |
| Q47708939 | The hnRNP RALY regulates transcription and cell proliferation by modulating the expression of specific factors including the proliferation marker E2F1. |
| Q57793297 | The hnRNP raly regulates PRMT1 expression and interacts with the ALS-linked protein FUS: implication for reciprocal cellular localization |
| Q58806144 | The prionlike domain of FUS is multiphosphorylated following DNA damage without altering nuclear localization |
| Q37474001 | Two familial ALS proteins function in prevention/repair of transcription-associated DNA damage |
| Q55550307 | Yeast as a Model to Unravel Mechanisms Behind FUS Toxicity in Amyotrophic Lateral Sclerosis. |
| Q58700147 | genetically interacts with the ALS-associated orthologue and mediates its toxicity |
| Q93032933 | hnRNPDL Phase Separation Is Regulated by Alternative Splicing and Disease-Causing Mutations Accelerate Its Aggregation |
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