scholarly article | Q13442814 |
P6179 | Dimensions Publication ID | 1008132874 |
P356 | DOI | 10.1186/1471-2121-14-32 |
P932 | PMC publication ID | 3710246 |
P698 | PubMed publication ID | 23834359 |
P5875 | ResearchGate publication ID | 247770476 |
P2093 | author name string | Luis Rodriguez-Menocal | |
Lawrence H Boise | |||
Enrique Cepero | |||
Matthew Brentnall | |||
Rebeka Ladron De Guevara | |||
P2860 | cites work | Ordering the cytochrome c-initiated caspase cascade: hierarchical activation of caspases-2, -3, -6, -7, -8, and -10 in a caspase-9-dependent manner | Q22008727 |
tBID, a membrane-targeted death ligand, oligomerizes BAK to release cytochrome c | Q24290118 | ||
Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade | Q24311006 | ||
Mechanisms of caspase activation and inhibition during apoptosis | Q27919623 | ||
Cell death: critical control points | Q28240722 | ||
Autoactivation of procaspase-9 by Apaf-1-mediated oligomerization | Q28275527 | ||
Proapoptotic BAX and BAK: a requisite gateway to mitochondrial dysfunction and death | Q28363890 | ||
Essential contribution of caspase 3/CPP32 to apoptosis and its associated nuclear changes | Q28506930 | ||
Caspases 3 and 7: key mediators of mitochondrial events of apoptosis | Q28588439 | ||
Cleavage of focal adhesion kinase by caspases during apoptosis | Q28610850 | ||
Bid induces the oligomerization and insertion of Bax into the outer mitochondrial membrane | Q28647586 | ||
Molecular definitions of cell death subroutines: recommendations of the Nomenclature Committee on Cell Death 2012 | Q29616161 | ||
A combinatorial approach defines specificities of members of the caspase family and granzyme B. Functional relationships established for key mediators of apoptosis | Q29617776 | ||
A distinct pathway remodels mitochondrial cristae and mobilizes cytochrome c during apoptosis | Q29620536 | ||
Oxidative phosphorylation at the fin de siècle | Q33538525 | ||
Mitochondrial control of nuclear apoptosis. | Q36366491 | ||
Executioner caspase-3 and caspase-7 are functionally distinct proteases | Q36861736 | ||
Microenvironmental influences of apoptosis in vivo and in vitro | Q37711148 | ||
Rapid production of retroviruses for efficient gene delivery to mammalian cells using 293T cell-based systems | Q39989137 | ||
Caspase-9 and effector caspases have sequential and distinct effects on mitochondria | Q40399437 | ||
Protease activation during apoptosis: death by a thousand cuts? | Q40459802 | ||
Bcl-2 and caspase inhibition cooperate to inhibit tumor necrosis factor-alpha-induced cell death in a Bcl-2 cleavage-independent fashion | Q40947508 | ||
Pro-caspase-3 is a major physiologic target of caspase-8. | Q46014432 | ||
Bcl-xL inhibits cytochrome c release but not mitochondrial depolarization during the activation of multiple death pathways by tumor necrosis factor-alpha | Q74061407 | ||
Superoxide in apoptosis. Mitochondrial generation triggered by cytochrome c loss | Q74495012 | ||
P921 | main subject | apoptotic process | Q14599311 |
P304 | page(s) | 32 | |
P577 | publication date | 2013-07-09 | |
P1433 | published in | BMC Cell Biology | Q2020930 |
P1476 | title | Caspase-9, caspase-3 and caspase-7 have distinct roles during intrinsic apoptosis | |
P478 | volume | 14 |