scholarly article | Q13442814 |
P6179 | Dimensions Publication ID | 1030862914 |
P356 | DOI | 10.1203/PDR.0B013E318211C917 |
P932 | PMC publication ID | 3092484 |
P698 | PubMed publication ID | 21270677 |
P5875 | ResearchGate publication ID | 49788662 |
P2093 | author name string | Yang Yu | |
Irfan Rahman | |||
Isaac K Sundar | |||
Vedang A Londhe | |||
Zubair H Aghai | |||
Benjamin Lopez | |||
Tiffany M Maisonet | |||
P2860 | cites work | Azithromycin treatment alters gene expression in inflammatory, lipid metabolism, and cell cycle pathways in well-differentiated human airway epithelia | Q33463373 |
Essential function of histone deacetylase 1 in proliferation control and CDK inhibitor repression | Q34088777 | ||
Pathology of new bronchopulmonary dysplasia | Q35095615 | ||
Azithromycin in the extremely low birth weight infant for the prevention of bronchopulmonary dysplasia: a pilot study | Q35850824 | ||
Lung vascular development: implications for the pathogenesis of bronchopulmonary dysplasia | Q36041092 | ||
Oxidative stress and redox regulation of lung inflammation in COPD. | Q36525624 | ||
Coping with stress: multiple ways to activate p53. | Q36745375 | ||
Differential responses in the lungs of newborn mouse pups exposed to 85% or >95% oxygen | Q37119181 | ||
Hyperoxia-induced premature senescence requires p53 and pRb, but not mitochondrial matrix ROS. | Q37123940 | ||
Histone deacetylase 2 is phosphorylated, ubiquitinated, and degraded by cigarette smoke | Q37139659 | ||
Manipulation of gene expression by oxygen: a primer from bedside to bench | Q37238463 | ||
Histone deacetylase HDAC1/HDAC2-controlled embryonic development and cell differentiation | Q37468782 | ||
Pitfalls, problems, and progress in bronchopulmonary dysplasia | Q37502269 | ||
Current perspectives on role of chromatin modifications and deacetylases in lung inflammation in COPD. | Q37610467 | ||
Disruption of p21 attenuates lung inflammation induced by cigarette smoke, LPS, and fMLP in mice. | Q39587342 | ||
Apoptosis in neonatal murine lung exposed to hyperoxia | Q40785655 | ||
Induction of p21WAF/CIP1 during hyperoxia | Q41063743 | ||
The number of alveoli in the terminal respiratory unit of man during late intrauterine life and childhood | Q41546755 | ||
High-mobility group box-1 protein in tracheal aspirates from premature infants: relationship with bronchopulmonary dysplasia and steroid therapy | Q43943397 | ||
Expression and activity of histone deacetylases in human asthmatic airways | Q44086893 | ||
The Effect of Neonatal Hyperoxia on the Lung of p21Waf1/Cip1/Sdi1-Deficient Mice | Q44649039 | ||
Cigarette smoke-mediated inflammatory and oxidative responses are strain-dependent in mice. | Q46674627 | ||
Azithromycin protects against hyperoxic lung injury in neonatal rats. | Q46929230 | ||
Functional and pathological effects of prolonged hyperoxia in neonatal mice. | Q47747208 | ||
Oxygen toxicity in mouse lung: pathways to cell death. | Q52532112 | ||
Decreased histone deacetylase activity in chronic obstructive pulmonary disease. | Q54664094 | ||
Exposure to hyperoxia induces p53 expression in mouse lung epithelium | Q74111149 | ||
Accumulation of p21(Cip1/WAF1) during hyperoxic lung injury in mice | Q77526392 | ||
Depletion of histone deacetylase protein: a common consequence of inflammatory cytokine signaling? | Q79436613 | ||
Azithromycin suppresses activation of nuclear factor-kappa B and synthesis of pro-inflammatory cytokines in tracheal aspirate cells from premature infants | Q80714985 | ||
Pulmonary expression of vascular endothelial growth factor (VEGF) and alveolar septation in a newborn rat model exposed to acute hypoxia and recovered under conditions of air or hyperoxia | Q83159686 | ||
Anti-inflammatory activity of azithromycin attenuates the effects of lipopolysaccharide administration in mice | Q83335529 | ||
CXCR2/CXCR2 ligand biological axis impairs alveologenesis during dsRNA-induced lung inflammation in mice | Q94460108 | ||
P433 | issue | 5 Pt 1 | |
P407 | language of work or name | English | Q1860 |
P304 | page(s) | 371-377 | |
P577 | publication date | 2011-05-01 | |
P1433 | published in | Pediatric Research | Q7159215 |
P1476 | title | Hyperoxia impairs alveolar formation and induces senescence through decreased histone deacetylase activity and up-regulation of p21 in neonatal mouse lung | |
P478 | volume | 69 |
Q27021767 | Beyond the genome: epigenetic mechanisms in lung remodeling |
Q38673032 | Bridging Lung Development with Chronic Obstructive Pulmonary Disease. Relevance of Developmental Pathways in Chronic Obstructive Pulmonary Disease Pathogenesis |
Q37619613 | Caffeine induces alveolar apoptosis in the hyperoxia-exposed developing mouse lung |
Q30317732 | Cellular senescence in normal and premature lung aging |
Q91743238 | Cellular senescence in the lung across the age spectrum |
Q33756577 | Chronic lung disease in the preterm infant. Lessons learned from animal models |
Q44488466 | Delay in rat lung alveolarization after the combined exposure of maternal hyperglycemia and postnatal hyperoxia |
Q26741052 | Early injury of the neonatal lung contributes to premature lung aging: a hypothesis |
Q35824490 | Effect of Hyperoxia on Retinoid Metabolism and Retinoid Receptor Expression in the Lungs of Newborn Mice |
Q99604380 | Epigenetic response to hyperoxia in the neonatal lung is sexually dimorphic |
Q38603475 | Expression profiling of genes regulated by sphingosine kinase1 signaling in a murine model of hyperoxia induced neonatal bronchopulmonary dysplasia |
Q33613229 | Genome-wide association mapping of acute lung injury in neonatal inbred mice |
Q28579280 | Hyperoxia arrests alveolar development through suppression of histone deacetylases in neonatal rats |
Q90216474 | Hyperoxia-induced Cellular Senescence in Fetal Airway Smooth Muscle Cells |
Q41127217 | Hyperoxia-induced methylation decreases RUNX3 in a newborn rat model of bronchopulmonary dysplasia |
Q35747991 | Impaired pulmonary vascular development in bronchopulmonary dysplasia |
Q28578094 | Lipopolysaccharide induces up-regulation of TGF-α through HDAC2 in a rat model of bronchopulmonary dysplasia |
Q64104455 | Live imaging of alveologenesis in precision-cut lung slices reveals dynamic epithelial cell behaviour |
Q47669220 | Long-Term Effects of Neonatal Hyperoxia in Adult Mice. |
Q38396850 | Lung consequences in adults born prematurely |
Q40894973 | Lung consequences in adults born prematurely. |
Q50689147 | Lung development alterations in newborn mice after recovery from exposure to sublethal hyperoxia. |
Q37507601 | Lung development: orchestrating the generation and regeneration of a complex organ |
Q38584746 | Lung endoderm morphogenesis: gasping for form and function |
Q51734245 | Neonatal exposure to hyperoxia leads to persistent disturbances in pulmonary histone signatures associated with NOS3 and STAT3 in a mouse model. |
Q30573333 | Neurodevelopmental impairment following neonatal hyperoxia in the mouse |
Q38309155 | New insights into lung development and diseases: the role of microRNAs. |
Q38043545 | Oxidative stress and chromatin remodeling in chronic obstructive pulmonary disease and smoking-related diseases |
Q38171333 | Oxygen regulates molecular mechanisms of cancer progression and metastasis |
Q92662037 | Positive Roles of Resveratrol in Early Development of Testicular Germ Cells against Maternal Restraint Stress in Mice |
Q33622213 | Postnatal inflammation in the pathogenesis of bronchopulmonary dysplasia |
Q101226483 | Preconditioning the immature lung with enhanced Nrf2 activity protects against oxidant-induced hypoalveolarization in mice |
Q38030311 | Role of histone deacetylase 2 in epigenetics and cellular senescence: implications in lung inflammaging and COPD |
Q40976331 | Sex-specific differences in neonatal hyperoxic lung injury |
Q34350843 | Suppressed expression of T-box transcription factors is involved in senescence in chronic obstructive pulmonary disease |
Q64933865 | The etiologic origins for chronic obstructive pulmonary disease. |
Q37419183 | Transcriptional responses of neonatal mouse lung to hyperoxia by Nrf2 status |
Q34584445 | Zfp148 deficiency causes lung maturation defects and lethality in newborn mice that are rescued by deletion of p53 or antioxidant treatment |
Q53061941 | [17β‑estradiol suppresses hyperoxia‑induced apoptosis of oligodendrocyte precursor cells through paired‑immunoglobulin‑like receptor B]. |
Search more.