Disturbed flow: p53 SUMOylation in the turnover of endothelial cells

scientific article

Disturbed flow: p53 SUMOylation in the turnover of endothelial cells is …
instance of (P31):
review articleQ7318358
scholarly articleQ13442814

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P356DOI10.1083/JCB.201104140
P932PMC publication ID3105545
P698PubMed publication ID21624951
P5875ResearchGate publication ID51177031

P50authorHanjoong JoQ40201403
P2093author name stringNoah Alberts-Grill
Wakako Takabe
P2860cites workp53 has a direct apoptogenic role at the mitochondriaQ24298888
Apoptosis, cancer and the p53 tumour suppressor geneQ28611432
Cell signaling by reactive nitrogen and oxygen species in atherosclerosisQ34006311
Effects of disturbed flow on vascular endothelium: pathophysiological basis and clinical perspectivesQ34159834
Discovery of novel mechanosensitive genes in vivo using mouse carotid artery endothelium exposed to disturbed flowQ34282296
p53-mediated inhibition of angiogenesis through up-regulation of a collagen prolyl hydroxylaseQ34558437
Endothelial protein kinase C isoform identity and differential activity of PKCzeta in an athero-susceptible region of porcine aortaQ34670556
PKCζ mediates disturbed flow-induced endothelial apoptosis via p53 SUMOylationQ35018220
Molecular mechanism of endothelial growth arrest by laminar shear stressQ35205740
Protein kinase Czeta-dependent LKB1 serine 428 phosphorylation increases LKB1 nucleus export and apoptosis in endothelial cellsQ36727225
DNA damage, p53, apoptosis and vascular diseaseQ36768762
Hemodynamics influences vascular peroxynitrite formation: Implication for low-density lipoprotein apo-B-100 nitrationQ36922288
Sustained activation of XBP1 splicing leads to endothelial apoptosis and atherosclerosis development in response to disturbed flowQ37176759
Partial carotid ligation is a model of acutely induced disturbed flow, leading to rapid endothelial dysfunction and atherosclerosisQ37406316
p53 Impairs Endothelial Function by Transcriptionally Repressing Kruppel-Like Factor 2Q42090504
Bone morphogenic protein 4 produced in endothelial cells by oscillatory shear stress induces monocyte adhesion by stimulating reactive oxygen species production from a nox1-based NADPH oxidaseQ45075488
Increased p53 gene dosage reduces neointimal thickening induced by mechanical injury but has no effect on native atherosclerosisQ57562752
The absence of p53 accelerates atherosclerosis by increasing cell proliferation in vivoQ74623272
Co-expression of p53 and MDM2 in human atherosclerosis: implications for the regulation of cellularity of atherosclerotic lesionsQ77407109
P433issue5
P407language of work or nameEnglishQ1860
P921main subjectendotheliumQ111140
protein sumoylationQ3503705
P1104number of pages3
P304page(s)805-807
P577publication date2011-05-01
P1433published inJournal of Cell BiologyQ1524550
P1476titleDisturbed flow: p53 SUMOylation in the turnover of endothelial cells
P478volume193

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cites work (P2860)
Q38125279Molecular biology of atherosclerosis
Q27300991Nanoscale Patterning of Extracellular Matrix Alters Endothelial Function under Shear Stress
Q64355502miR-214 is Stretch-Sensitive in Aortic Valve and Inhibits Aortic Valve Calcification

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