scholarly article | Q13442814 |
P2093 | author name string | Thornton W Thompson | |
Malen A Link | |||
Lisa A Pitcher | |||
David J Davido | |||
Heba H Mostafa | |||
Adam M Bayless | |||
Steve D Haenchen | |||
Priscilla A Schaffer | |||
Anna S Kushnir | |||
Emma Loveday | |||
Joshua G Hilliard | |||
P2860 | cites work | PML contributes to a cellular mechanism of repression of herpes simplex virus type 1 infection that is inactivated by ICP0 | Q24298207 |
Differential role of Sp100 isoforms in interferon-mediated repression of herpes simplex virus type 1 immediate-early protein expression | Q24298241 | ||
Viral immediate-early proteins abrogate the modification by SUMO-1 of PML and Sp100 proteins, correlating with nuclear body disruption | Q24527246 | ||
Regulation of herpesvirus macromolecular synthesis. I. Cascade regulation of the synthesis of three groups of viral proteins | Q24630637 | ||
Herpes simplex virus-infected cell protein 0 blocks the silencing of viral DNA by dissociating histone deacetylases from the CoREST-REST complex | Q24675640 | ||
The degradation of promyelocytic leukemia and Sp100 proteins by herpes simplex virus 1 is mediated by the ubiquitin-conjugating enzyme UbcH5a | Q24678629 | ||
Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method | Q25938999 | ||
Characterization of the IE110 gene of herpes simplex virus type 1 | Q28287831 | ||
Posttranslational processing of infected cell proteins 0 and 4 of herpes simplex virus 1 is sequential and reflects the subcellular compartment in which the proteins localize | Q28345181 | ||
A viral ubiquitin ligase has substrate preferential SUMO targeted ubiquitin ligase activity that counteracts intrinsic antiviral defence | Q28477120 | ||
State and role of SRC family kinases in replication of herpes simplex virus 1. | Q30159875 | ||
Herpes simplex virus 1 infected cell protein 0 forms a complex with CIN85 and Cbl and mediates the degradation of EGF receptor from cell surfaces | Q30160269 | ||
Herpes simplex virus type 1 immediate-early protein ICP0 and is isolated RING finger domain act as ubiquitin E3 ligases in vitro | Q39686108 | ||
Regulation of ICP0-null mutant herpes simplex virus type 1 infection by ND10 components ATRX and hDaxx | Q39741152 | ||
Epigenetic modulation of gene expression from quiescent herpes simplex virus genomes | Q39836698 | ||
Replication of ICP0-null mutant herpes simplex virus type 1 is restricted by both PML and Sp100. | Q40030711 | ||
Histone modifications associated with herpes simplex virus type 1 genomes during quiescence and following ICP0-mediated de-repression | Q40033809 | ||
Herpes simplex virus type 1 ICP0 regulates expression of immediate-early, early, and late genes in productively infected cells | Q40065340 | ||
Reciprocal activities between herpes simplex virus type 1 regulatory protein ICP0, a ubiquitin E3 ligase, and ubiquitin-specific protease USP7. | Q40373883 | ||
The immediate-early protein, ICP0, is essential for the resistance of herpes simplex virus to interferon-alpha/beta | Q40745643 | ||
Nuclear domain 10 as preexisting potential replication start sites of herpes simplex virus type-1. | Q41225801 | ||
Trans activation of transcription by herpes virus products: requirement for two HSV-1 immediate-early polypeptides for maximum activity | Q41587905 | ||
Reversal of heterochromatic silencing of quiescent herpes simplex virus type 1 by ICP0. | Q41933341 | ||
Herpes simplex virus type 1 C-terminal variants of the origin binding protein (OBP), OBPC-1 and OBPC-2, cooperatively regulate viral DNA levels in vitro, and OBPC-2 affects mortality in mice | Q42147484 | ||
Defining the SUMO-modified proteome by multiple approaches in Saccharomyces cerevisiae | Q42642323 | ||
The herpes simplex virus type 1 (HSV-1) regulatory protein ICP0 interacts with and Ubiquitinates p53. | Q44509784 | ||
Herpes virus induced proteasome-dependent degradation of the nuclear bodies-associated PML and Sp100 proteins | Q45751210 | ||
Modification of discrete nuclear domains induced by herpes simplex virus type 1 immediate early gene 1 product (ICP0). | Q45777008 | ||
Temperature-sensitive mutants of herpes simplex virus type 1: isolation, complementation and partial characterization | Q45817912 | ||
Isolation and characterization of a herpes simplex virus type 1 mutant containing a deletion within the gene encoding the immediate early polypeptide Vmw110. | Q45835577 | ||
Construction and characterization of herpes simplex virus type 1 mutants with defined lesions in immediate early gene 1. | Q45845827 | ||
Phospho-regulated SUMO interaction modules connect the SUMO system to CK2 signaling | Q46122037 | ||
RNA complementary to a herpesvirus alpha gene mRNA is prominent in latently infected neurons | Q68986775 | ||
The UL13 protein kinase and the infected cell type are determinants of posttranslational modification of ICP0 | Q73661488 | ||
Cellular localization of the herpes simplex virus ICP0 protein dictates its ability to block IRF3-mediated innate immune responses | Q33573566 | ||
The disruption of ND10 during herpes simplex virus infection correlates with the Vmw110- and proteasome-dependent loss of several PML isoforms | Q33783492 | ||
Herpes simplex virus ICP0 mutants are hypersensitive to interferon | Q33798242 | ||
Optimized viral dose and transient immunosuppression enable herpes simplex virus ICP0-null mutants To establish wild-type levels of latency in vivo | Q33806643 | ||
ICP0 is required for efficient reactivation of herpes simplex virus type 1 from neuronal latency | Q33838198 | ||
Herpes simplex virus type 1 corneal infection results in periocular disease by zosteriform spread | Q33851697 | ||
Herpes simplex virus 1-infected cell protein 0 contains two E3 ubiquitin ligase sites specific for different E2 ubiquitin-conjugating enzymes | Q33894274 | ||
Herpes simplex virus phosphoproteins. I. Phosphate cycles on and off some viral polypeptides and can alter their affinity for DNA. | Q33911883 | ||
B7 costimulation molecules encoded by replication-defective, vhs-deficient HSV-1 improve vaccine-induced protection against corneal disease | Q33985984 | ||
Point mutations in herpes simplex virus type 1 oriL, but not in oriS, reduce pathogenesis during acute infection of mice and impair reactivation from latency | Q34233084 | ||
The cyclin-dependent kinase inhibitor roscovitine inhibits the transactivating activity and alters the posttranslational modification of herpes simplex virus type 1 ICP0. | Q34330457 | ||
Alpha/Beta interferon and gamma interferon synergize to inhibit the replication of herpes simplex virus type 1. | Q34356255 | ||
Herpes simplex virus ICP0 promotes both histone removal and acetylation on viral DNA during lytic infection | Q34848465 | ||
ICP0 antagonizes Stat 1-dependent repression of herpes simplex virus: implications for the regulation of viral latency | Q35013167 | ||
The herpes simplex virus immediate-early protein ICP0 affects transcription from the viral genome and infected-cell survival in the absence of ICP4 and ICP27 | Q35886888 | ||
Interferons regulate the phenotype of wild-type and mutant herpes simplex viruses in vivo | Q36367805 | ||
The herpes simplex virus type 1 regulatory protein ICP0 enhances virus replication during acute infection and reactivation from latency. | Q36655446 | ||
Herpes simplex virus 1 has multiple mechanisms for blocking virus-induced interferon production | Q36684310 | ||
Immediate-early regulatory gene mutants define different stages in the establishment and reactivation of herpes simplex virus latency | Q36780729 | ||
Quantitative polymerase chain reaction analysis of herpes simplex virus DNA in ganglia of mice infected with replication-incompetent mutants | Q36782886 | ||
Herpes simplex virus type 1 ICP0 plays a critical role in the de novo synthesis of infectious virus following transfection of viral DNA. | Q36831483 | ||
Deletion mutants in the gene encoding the herpes simplex virus type 1 immediate-early protein ICP0 exhibit impaired growth in cell culture | Q36885629 | ||
Characterization of herpes simplex virus 1 alpha proteins 0, 4, and 27 with monoclonal antibodies | Q36894567 | ||
Herpes simplex virus type 1 ICP0 phosphorylation mutants impair the E3 ubiquitin ligase activity of ICP0 in a cell type-dependent manner | Q36949684 | ||
The Differential Requirement for Cyclin-Dependent Kinase Activities Distinguishes Two Functions of Herpes Simplex Virus Type 1 ICP0 | Q37060135 | ||
The infected cell protein 0 of herpes simplex virus 1 dynamically interacts with proteasomes, binds and activates the cdc34 E2 ubiquitin-conjugating enzyme, and possesses in vitro E3 ubiquitin ligase activity. | Q37116670 | ||
The role of cdc2 in the expression of herpes simplex virus genes | Q37269688 | ||
Re-evaluating natural resistance to herpes simplex virus type 1. | Q37492787 | ||
Identification of immediate early genes from herpes simplex virus that transactivate the virus thymidine kinase gene | Q37527308 | ||
Role of the VP16-binding domain of vhs in viral growth, host shutoff activity, and pathogenesis | Q37683002 | ||
Phosphorylation site mutations affect herpes simplex virus type 1 ICP0 function | Q37743081 | ||
Specification of SUMO1- and SUMO2-interacting motifs | Q38315174 | ||
Activities of ICP0 involved in the reversal of silencing of quiescent herpes simplex virus 1. | Q39575218 | ||
P433 | issue | 23 | |
P407 | language of work or name | English | Q1860 |
P304 | page(s) | 12631-12637 | |
P577 | publication date | 2011-09-21 | |
P1433 | published in | Journal of Virology | Q1251128 |
P1476 | title | Herpes simplex virus 1 ICP0 phosphorylation site mutants are attenuated for viral replication and impaired for explant-induced reactivation | |
P478 | volume | 85 |
Q36306971 | A quantitative assay to monitor HSV-1 ICP0 ubiquitin ligase activity in vitro |
Q64108733 | Attenuated infectious laryngotracheitis virus vaccines differ in their capacity to establish latency in the trigeminal ganglia of specific pathogen free chickens following eye drop inoculation |
Q33883877 | HSV-1 ICP0: An E3 Ubiquitin Ligase That Counteracts Host Intrinsic and Innate Immunity |
Q47555886 | Herpes Simplex Virus 1 Mutant with Point Mutations in UL39 Is Impaired for Acute Viral Replication in Mice, Establishment of Latency, and Explant-Induced Reactivation |
Q37336866 | Herpes simplex virus 1 ICP22 but not US 1.5 is required for efficient acute replication in mice and VICE domain formation |
Q39796290 | Herpes simplex virus 1 upregulates p35, alters CDK-5 localization, and stimulates CDK-5 kinase activity during acute infection in neurons |
Q26766356 | Infected cell protein 0 functional domains and their coordination in herpes simplex virus replication |
Q36607356 | N-terminal phosphorylation sites of herpes simplex virus 1 ICP0 differentially regulate its activities and enhance viral replication |
Q94951027 | The HSV-1 ubiquitin ligase ICP0: modifying the cellular proteome to promote infection |
Q37336832 | Two overlapping regions within the N-terminal half of the herpes simplex virus 1 E3 ubiquitin ligase ICP0 facilitate the degradation and dissociation of PML and dissociation of Sp100 from ND10. |
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