| scholarly article | Q13442814 |
| P50 | author | Spencer Proctor | Q47890729 |
| Russell Terry | Q60369691 | ||
| P2093 | author name string | Erika Smith | |
| Petra Rocic | |||
| Rashmi Jadhav | |||
| Rebecca Hutcheson | |||
| Robert Barrington | |||
| Brenda Hutcheson | |||
| Jennifer Chaplin | |||
| P2860 | cites work | MicroRNA-21 exhibits antiangiogenic function by targeting RhoB expression in endothelial cells | Q21135545 |
| Endothelial cell dysfunction and the vascular complications associated with type 2 diabetes: assessing the health of the endothelium | Q28219326 | ||
| MicroRNAs and ischemic heart disease: towards a better comprehension of pathogenesis, new diagnostic tools and new therapeutic targets | Q28248253 | ||
| Redox-sensitive Akt and Src regulate coronary collateral growth in metabolic syndrome | Q28389912 | ||
| Coronary collateral growth--back to the future | Q28658320 | ||
| Neutrophil cell surface receptors and their intracellular signal transduction pathways. | Q30009577 | ||
| Spatial and temporal coordination of bone marrow-derived cell activity during arteriogenesis: regulation of the endogenous response and therapeutic implications | Q30430026 | ||
| miR-21 normalizes vascular smooth muscle proliferation and improves coronary collateral growth in metabolic syndrome | Q34072676 | ||
| Restoration of coronary endothelial function in obese Zucker rats by a low-carbohydrate diet | Q34601284 | ||
| Nitric oxide and cell signaling pathways in mitochondrial-dependent apoptosis | Q34658879 | ||
| MMPs 2 and 9 are essential for coronary collateral growth and are prominently regulated by p38 MAPK. | Q35529307 | ||
| Angiotensin type I receptor blockade in conjunction with enhanced Akt activation restores coronary collateral growth in the metabolic syndrome | Q35767686 | ||
| Silencing of microRNA families by seed-targeting tiny LNAs | Q36522844 | ||
| MicroRNA-145 restores contractile vascular smooth muscle phenotype and coronary collateral growth in the metabolic syndrome | Q36773823 | ||
| Bcl-2 family members regulate anoxia-induced cell death | Q36879207 | ||
| Osteopontin: a multifunctional molecule regulating chronic inflammation and vascular disease | Q36918536 | ||
| Mechanisms of arteriogenesis | Q37237370 | ||
| Wall thickness of coronary vessels varies transmurally in the LV but not the RV: implications for local stress distribution | Q37298352 | ||
| Role of MCP-1 in tumor necrosis factor-alpha-induced endothelial dysfunction in type 2 diabetic mice | Q37406302 | ||
| Nitric oxide promotes endothelial cell survival signaling through S-nitrosylation and activation of dynamin-2. | Q40179416 | ||
| Analysis of the alpha4beta1 integrin-osteopontin interaction | Q40868329 | ||
| Altered PYK2 phosphorylation by ANG II in hypertensive vascular smooth muscle | Q42515619 | ||
| Impaired coronary collateral growth in the metabolic syndrome is in part mediated by matrix metalloproteinase 12-dependent production of endostatin and angiostatin | Q42571891 | ||
| Restoration of coronary collateral growth in the Zucker obese rat: impact of VEGF and ecSOD. | Q45866264 | ||
| The mechanistic basis for the disparate effects of angiotensin II on coronary collateral growth | Q46930209 | ||
| Fibrinolysis and atherogenesis in the JCR:LA-cp rat in relation to insulin and triglyceride concentrations in blood. | Q50903243 | ||
| Reactive oxygen species-selective regulation of aortic inflammatory gene expression in Type 2 diabetes | Q59279106 | ||
| Translational evidence of endothelial damage in obese individuals: inflammatory and prothrombotic responses | Q59421710 | ||
| Down-regulation by antisense oligonucleotides establishes a role for the proline-rich tyrosine kinase PYK2 in angiotensin ii-induced signaling in vascular smooth muscle | Q73656443 | ||
| Cardiovascular disease in the JCR:LA-cp rat | Q77582051 | ||
| Acute phase response, inflammation and metabolic syndrome biomarkers of Libby asbestos exposure | Q83514676 | ||
| P433 | issue | 11 | |
| P921 | main subject | metabolic syndrome | Q657193 |
| P304 | page(s) | H1323-35 | |
| P577 | publication date | 2015-04-03 | |
| P1433 | published in | American Journal of Physiology Heart and Circulatory Physiology | Q3193662 |
| P1476 | title | miR-21-mediated decreased neutrophil apoptosis is a determinant of impaired coronary collateral growth in metabolic syndrome | |
| P478 | volume | 308 |
| Q40608876 | Effects of ghrelin on the apoptosis of human neutrophils in vitro |
| Q51239324 | Elevated 20-HETE impairs coronary collateral growth in metabolic syndrome via endothelial dysfunction. |
| Q50081720 | Elevated 20-HETE in metabolic syndrome regulates arterial stiffness and systolic hypertension via MMP12 activation |
| Q90884479 | Identification of key protein-coding genes and lncRNAs in spontaneous neutrophil apoptosis |
| Q35904086 | Impaired coronary collateral growth: miR-shaken neutrophils caught in the act. |
| Q28066227 | Mechanisms of Comorbidities Associated With the Metabolic Syndrome: Insights from the JCR:LA-cp Corpulent Rat Strain |
| Q37010626 | Profiling of differentially expressed microRNAs in arrhythmogenic right ventricular cardiomyopathy |
| Q35973580 | Sulforaphane induces apoptosis and inhibits invasion in U251MG glioblastoma cells |
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