Experimental Mapping of the Canine KCNJ2 and KCNJ12 Gene Structures and Functional Analysis of the Canine K(IR)2.2 ion Channel

scientific article published on 30 January 2012

Experimental Mapping of the Canine KCNJ2 and KCNJ12 Gene Structures and Functional Analysis of the Canine K(IR)2.2 ion Channel is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.3389/FPHYS.2012.00009
P932PMC publication ID3277267
P698PubMed publication ID22363290
P5875ResearchGate publication ID221859572

P2093author name stringMarcel A G van der Heyden
Teun P de Boer
Marc A Vos
Marien J C Houtman
Denise R Montagne
Hiroki Takanari
Margot van Eck
Bart G J M Kok
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Kir 2.1 channelopathies: the Andersen-Tawil syndromeQ37715824
Two modes of polyamine block regulating the cardiac inward rectifier K+ current IK1 as revealed by a study of the Kir2.1 channel expressed in a human cell lineQ39385587
Low-affinity spermine block mediating outward currents through Kir2.1 and Kir2.2 inward rectifier potassium channelsQ40104801
A Kir2.1 gain-of-function mutation underlies familial atrial fibrillationQ40416086
Multiple promoter elements interact to control the transcription of the potassium channel gene, KCNJ2.Q41014688
Challenging cardiac electrophysiologyQ41865708
Lysosome mediated Kir2.1 breakdown directly influences inward rectifier current density.Q41941682
Different intracellular polyamine concentrations underlie the difference in the inward rectifier K(+) currents in atria and ventricles of the guinea-pig heartQ45236615
Proarrhythmic electrical remodelling is associated with increased beat-to-beat variability of repolarisationQ48632971
Transgenic upregulation of IK1 in the mouse heart leads to multiple abnormalities of cardiac excitability.Q50797858
P304page(s)9
P577publication date2012-01-30
P1433published inFrontiers in PhysiologyQ2434141
P1476titleExperimental Mapping of the Canine KCNJ2 and KCNJ12 Gene Structures and Functional Analysis of the Canine K(IR)2.2 ion Channel
P478volume3

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Q41033530PA-6 inhibits inward rectifier currents carried by V93I and D172N gain-of-function KIR2.1 channels, but increases channel protein expression
Q37565636Progressive Cl- channel defects reveal disrupted skeletal muscle maturation in R6/2 Huntington's mice