| P2093 | author name string | Peter J Hollenbeck | |
| | Alex Liu | |
| | Swathi Devireddy | |
| | Taylor Lampe | |
| P2860 | cites work | PINK1 is selectively stabilized on impaired mitochondria to activate Parkin | Q21145802 |
| | Parkinson's disease-associated kinase PINK1 regulates Miro protein level and axonal transport of mitochondria | Q21563375 |
| | PARIS (ZNF746) repression of PGC-1α contributes to neurodegeneration in Parkinson's disease | Q24294809 |
| | PINK1 and Parkin target Miro for phosphorylation and degradation to arrest mitochondrial motility | Q24296955 |
| | The axonal transport of mitochondria | Q27007553 |
| | The pathways of mitophagy for quality control and clearance of mitochondria | Q27009206 |
| | Mechanisms of mitophagy | Q27310261 |
| | The yeast complex I equivalent NADH dehydrogenase rescues pink1 mutants | Q27335187 |
| | Disruption of fast axonal transport is a pathogenic mechanism for intraneuronal amyloid beta | Q28239322 |
| | TANGO: a generic tool for high-throughput 3D image analysis for studying nuclear organization | Q28364897 |
| | Inhibition of fast axonal transport by pathogenic SOD1 involves activation of p38 MAP kinase | Q28533850 |
| | Loss of PINK1 causes mitochondrial functional defects and increased sensitivity to oxidative stress | Q28592727 |
| | PINK1 loss-of-function mutations affect mitochondrial complex I activity via NdufA10 ubiquinone uncoupling | Q28593164 |
| | A guided tour into subcellular colocalization analysis in light microscopy | Q29547199 |
| | Drosophila pink1 is required for mitochondrial function and interacts genetically with parkin | Q29547423 |
| | A pyramid approach to subpixel registration based on intensity | Q29614713 |
| | Drosophila parkin requires PINK1 for mitochondrial translocation and ubiquitinates mitofusin | Q29615625 |
| | The PINK1/Parkin pathway regulates mitochondrial morphology | Q29615641 |
| | Mitochondrial dynamics--fusion, fission, movement, and mitophagy--in neurodegenerative diseases | Q29615646 |
| | Mitochondrial dysfunction in Drosophila PINK1 mutants is complemented by parkin | Q29615684 |
| | PINK1 stabilized by mitochondrial depolarization recruits Parkin to damaged mitochondria and activates latent Parkin for mitophagy | Q29616005 |
| | Synaptic mitochondria are critical for mobilization of reserve pool vesicles at Drosophila neuromuscular junctions | Q29617065 |
| | Mitophagy of damaged mitochondria occurs locally in distal neuronal axons and requires PINK1 and Parkin | Q30273805 |
| | Autophagosomes initiate distally and mature during transport toward the cell soma in primary neurons | Q30425491 |
| | Kinesin-1 and Dynein are the primary motors for fast transport of mitochondria in Drosophila motor axons | Q30477062 |
| | Pathogenic huntingtin inhibits fast axonal transport by activating JNK3 and phosphorylating kinesin | Q30490028 |
| | Bioenergetics of neurons inhibit the translocation response of Parkin following rapid mitochondrial depolarization. | Q30498087 |
| | Transcellular degradation of axonal mitochondria. | Q30583027 |
| | The mitochondrial fusion-promoting factor mitofusin is a substrate of the PINK1/parkin pathway | Q33552320 |
| | Mitofusin 2 is necessary for transport of axonal mitochondria and interacts with the Miro/Milton complex | Q33779586 |
| | Evidence that myosin activity opposes microtubule-based axonal transport of mitochondria | Q33998992 |
| | Defects in mitochondrial axonal transport and membrane potential without increased reactive oxygen species production in a Drosophila model of Friedreich ataxia | Q34143936 |
| | Altered axonal mitochondrial transport in the pathogenesis of Charcot-Marie-Tooth disease from mitofusin 2 mutations | Q34600251 |
| | Mitochondrial movement and positioning in axons: the role of growth factor signaling | Q35131142 |
| | Spatial parkin translocation and degradation of damaged mitochondria via mitophagy in live cortical neurons | Q35855944 |
| | Silent information regulator 2 (Sir2) and Forkhead box O (FOXO) complement mitochondrial dysfunction and dopaminergic neuron loss in Drosophila PTEN-induced kinase 1 (PINK1) null mutant | Q35921597 |
| | Parkinson's disease mutations in PINK1 result in decreased Complex I activity and deficient synaptic function | Q36042047 |
| | Mitochondria and neurotransmission: evacuating the synapse | Q36213274 |
| | The axonal transport of mitochondria. | Q36321480 |
| | Mitochondrial trafficking and morphology in healthy and injured neurons | Q36692390 |
| | The PINK1-Parkin pathway promotes both mitophagy and selective respiratory chain turnover in vivo | Q36782133 |
| | Mitochondrial biogenesis in the axons of vertebrate peripheral neurons | Q36883274 |
| | TRAP1 rescues PINK1 loss-of-function phenotypes | Q36950677 |
| | Mitochondrial membrane potential in axons increases with local nerve growth factor or semaphorin signaling | Q37003099 |
| | Bcl-x L increases mitochondrial fission, fusion, and biomass in neurons | Q37202353 |
| | Mitochondrial quality control: a matter of life and death for neurons | Q37987023 |
| | Effects of imaging conditions on mitochondrial transport and length in larval motor axons of Drosophila | Q40068903 |
| | Measurement of mitochondrial membrane potential using fluorescent rhodamine derivatives | Q40223888 |
| | Drosophila Trap1 protects against mitochondrial dysfunction in a PINK1/parkin model of Parkinson's disease. | Q41892915 |
| | Effects of the uncoupling agents FCCP and CCCP on the saltatory movements of cytoplasmic organelles | Q42816519 |
| | Drosophila Miro is required for both anterograde and retrograde axonal mitochondrial transport. | Q43099615 |
| | Glutamate decreases mitochondrial size and movement in primary forebrain neurons. | Q44566043 |
| | Principles of the mitochondrial fusion and fission cycle in neurons | Q44593503 |
| | Axonal mitochondrial transport and potential are correlated | Q44901240 |
| | Analysis of mitochondrial traffic in Drosophila | Q46811563 |
| | Nerve growth factor signaling regulates motility and docking of axonal mitochondria. | Q51642532 |
| P4510 | describes a project that uses | ImageJ | Q1659584 |
| P433 | issue | 25 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | quality control | Q827792 |
| P304 | page(s) | 9391-9401 | |
| P577 | publication date | 2015-06-01 | |
| P1433 | published in | Journal of Neuroscience | Q1709864 |
| P1476 | title | The Organization of Mitochondrial Quality Control and Life Cycle in the Nervous System In Vivo in the Absence of PINK1. | |
| P478 | volume | 35 | |