Fibrinolysis shutdown phenotype masks changes in rodent coagulation in tissue injury versus hemorrhagic shock

scientific article published on 5 June 2015

Fibrinolysis shutdown phenotype masks changes in rodent coagulation in tissue injury versus hemorrhagic shock is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1016/J.SURG.2015.04.008
P8608Fatcat IDrelease_olmdsf5gxves5celbxgeu442oq
P932PMC publication ID4492895
P698PubMed publication ID25979440

P2093author name stringAnirban Banerjee
Christopher C Silliman
Ernest E Moore
Peter J Lawson
Angela Sauaia
Eduardo Gonzalez
Hunter B Moore
Michael P Chapman
Alex P Morton
Fabia Gamboni
Miguel Fragoso
P2860cites workMesenteric lymph diversion abrogates 5-lipoxygenase activation in the kidney following trauma and hemorrhagic shockQ33880916
A principal component analysis of postinjury viscoelastic assays: clotting factor depletion versus fibrinolysisQ34115729
Acute coagulopathy of trauma in the rat.Q34616907
Hyperfibrinolysis, physiologic fibrinolysis, and fibrinolysis shutdown: the spectrum of postinjury fibrinolysis and relevance to antifibrinolytic therapyQ35207771
Hemolysis exacerbates hyperfibrinolysis, whereas platelolysis shuts down fibrinolysis: evolving concepts of the spectrum of fibrinolysis in response to severe injuryQ35216740
Increase in activated protein C mediates acute traumatic coagulopathy in miceQ36615864
Animal models of trauma-induced coagulopathyQ37971198
Hemodilution is not critical in the pathogenesis of the acute coagulopathy of trauma.Q38558714
A standardized technique for performing thromboelastography in rodents.Q38829684
A principal component analysis of coagulation after traumaQ40170564
Advanced Trauma Life Support (ATLS): past, present, future--16th Stone Lecture, American Trauma SocietyQ41103864
Minding the gaps--and the junctions, too.Q43243890
Treatment of severe acute respiratory distress syndrome: a final report on a phase I studyQ43493864
Hyperfibrinolysis is common in out-of-hospital cardiac arrest: results from a prospective observational thromboelastometry studyQ44494788
Acute traumatic coagulopathy: initiated by hypoperfusion: modulated through the protein C pathway?Q46166562
Reproducibility of an animal model simulating complex combat-related injury in a multiple-institution format.Q46580937
Assessment of coagulopathy, endothelial injury, and inflammation after traumatic brain injury and hemorrhage in a porcine modelQ47932863
Generation of Biologically Active Angiostatin Kringle 1-3 by Activated Human NeutrophilsQ58905898
The mode of action of primary bile salts on human plateletsQ70608918
The origin of platelet count and volumeQ70642675
Acute traumatic coagulopathyQ73544166
Acute coagulopathy of trauma: hypoperfusion induces systemic anticoagulation and hyperfibrinolysisQ81248493
Acidosis downregulates platelet haemostatic functions and promotes neutrophil proinflammatory responses mediated by plateletsQ82902286
Fibrinolytic proteins in human bile accelerate lysis of plasma clots and induce breakdown of fibrin sealantsQ84574531
Elevated tissue plasminogen activator and reduced plasminogen activator inhibitor promote hyperfibrinolysis in trauma patientsQ87517095
P433issue2
P407language of work or nameEnglishQ1860
P921main subjectphenotypeQ104053
hemorrhagic shockQ2964243
P304page(s)386-392
P577publication date2015-06-05
P1433published inSurgeryQ15731526
P1476titleFibrinolysis shutdown phenotype masks changes in rodent coagulation in tissue injury versus hemorrhagic shock
P478volume158

Reverse relations

cites work (P2860)
Q36807586Acute Fibrinolysis Shutdown after Injury Occurs Frequently and Increases Mortality: A Multicenter Evaluation of 2,540 Severely Injured Patients
Q41272666Acute mesenteric ischemia: guidelines of the World Society of Emergency Surgery
Q34677754Advances in the understanding of trauma-induced coagulopathy
Q92764318Does Tranexamic Acid Improve Clot Strength in Severely Injured Patients Who Have Elevated Fibrin Degradation Products and Low Fibrinolytic Activity, Measured by Thrombelastography?
Q47251777Fibrinolysis in trauma: a review.
Q52570198Harmful or Physiologic: Diagnosing Fibrinolysis Shutdown in a Trauma Cohort With Rotational Thromboelastometry.
Q47911192Human neutrophil elastase mediates fibrinolysis shutdown through competitive degradation of plasminogen and generation of angiostatin
Q42022691Plasma First Resuscitation Reduces Lactate Acidosis, Enhances Redox Homeostasis, Amino Acid and Purine Catabolism in a Rat Model of Profound Hemorrhagic Shock
Q35646892Postinjury fibrinolysis shutdown: Rationale for selective tranexamic acid
Q45375279Rationale for the selective administration of tranexamic acid to inhibit fibrinolysis in the severely injured patient
Q47880955Targeting resuscitation to normalization of coagulating status: Hyper and hypocoagulability after severe injury are both associated with increased mortality
Q38679752The role of NIGMS P50 sponsored team science in our understanding of multiple organ failure
Q38742341Thrombelastography indicates limitations of animal models of trauma-induced coagulopathy
Q49200827Tissue injury suppresses fibrinolysis after hemorrhagic shock in nonhuman primates (rhesus macaque).
Q40577604Uncritical use of tranexamic acid in trauma patients : Do no further harm!
Q38776292Viscoelastic Tissue Plasminogen Activator Challenge Predicts Massive Transfusion in 15 Minutes
Q36818081Viscoelastic measurements of platelet function, not fibrinogen function, predicts sensitivity to tissue-type plasminogen activator in trauma patients

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