Causal connectivity alterations of cortical-subcortical circuit anchored on reduced hemodynamic response brain regions in first-episode drug-naïve major depressive disorder

scientific article published on 25 February 2016

Causal connectivity alterations of cortical-subcortical circuit anchored on reduced hemodynamic response brain regions in first-episode drug-naïve major depressive disorder is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1038/SREP21861
P932PMC publication ID4766513
P698PubMed publication ID26911651
P5875ResearchGate publication ID297791671

P2093author name stringXueli Sun
Huafu Chen
Qing Gao
Ke Zou
Zongling He
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The neural correlates of reward-related processing in major depressive disorder: a meta-analysis of functional magnetic resonance imaging studiesQ39384358
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Retrieving the Hemodynamic Response Function in resting state fMRI: Methodology and applicationQ40137269
Association of resting-state network dysfunction with their dynamics of inter-network interactions in depressionQ41628589
Unidirectionally affected causal connectivity of cortico-limbic-cerebellar circuit by structural deficits in drug-naive major depressive disorderQ41753433
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Reduced neuronal size and glial cell density in area 9 of the dorsolateral prefrontal cortex in subjects with major depressive disorder.Q47191236
Diminished caudate and superior temporal gyrus responses to effort-based decision making in patients with first-episode major depressive disorderQ47630597
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Cerebral and cerebellar gray matter reduction in first-episode patients with major depressive disorder: a voxel-based morphometry studyQ48199617
Alterations in effective connectivity anchored on the insula in major depressive disorderQ48213726
Altered cerebellar-cerebral resting-state functional connectivity reliably identifies major depressive disorderQ48258360
Aberrant topology of striatum's connectivity is associated with the number of episodes in depressionQ48408787
Functional connectivity bias of the orbitofrontal cortex in drug-free patients with major depressionQ48439647
Abnormal regional spontaneous neural activity in first-episode, treatment-naive patients with late-life depression: a resting-state fMRI studyQ48440326
Point-Process Deconvolution of fMRI BOLD Signal Reveals Effective Connectivity Alterations in Chronic Pain PatientsQ48497736
Identifying major depression using whole-brain functional connectivity: a multivariate pattern analysisQ48614919
P275copyright licenseCreative Commons Attribution 4.0 InternationalQ20007257
P6216copyright statuscopyrightedQ50423863
P407language of work or nameEnglishQ1860
P921main subjecthemodynamicsQ1642137
P304page(s)21861
P577publication date2016-02-25
P1433published inScientific ReportsQ2261792
P1476titleCausal connectivity alterations of cortical-subcortical circuit anchored on reduced hemodynamic response brain regions in first-episode drug-naïve major depressive disorder
P478volume6

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cites work (P2860)
Q39331327Acupuncture treatment modulates the corticostriatal reward circuitry in major depressive disorder.
Q52366060Brain structure alterations in depression: Psychoradiological evidence.
Q33608113Cortico-Striatal-Thalamic Loop Circuits of the Orbitofrontal Cortex: Promising Therapeutic Targets in Psychiatric Illness
Q37444148Environmental factors linked to depression vulnerability are associated with altered cerebellar resting-state synchronization.
Q39264760Extraversion and neuroticism related to the resting-state effective connectivity of amygdala
Q64097679Neural cell adhesion molecule Negr1 deficiency in mouse results in structural brain endophenotypes and behavioral deviations related to psychiatric disorders
Q89597763Orbitofrontal cortex activity and connectivity predict future depression symptoms in adolescence
Q92153497Profound and reproducible patterns of reduced regional gray matter characterize major depressive disorder
Q89338439Regional alterations of cerebral [18F]FDG metabolism in the chronic unpredictable mild stress- and the repeated corticosterone depression model in rats
Q57799526The brain's hemodynamic response function rapidly changes under acute psychosocial stress in association with genetic and endocrine stress response markers

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