| P50 | author | Keith S Elmslie | Q56956361 |
| P2093 | author name string | Renuka Ramachandra | |
| | James C Baxter | |
| | Jason R Howard | |
| | Stephanie Y McGrew | |
| P2860 | cites work | Distribution of the tetrodotoxin-resistant sodium channel PN3 in rat sensory neurons in normal and neuropathic conditions. | Q32108845 |
| | Contribution of Na(v)1.8 sodium channels to action potential electrogenesis in DRG neurons | Q34086296 |
| | Roles of tetrodotoxin (TTX)-sensitive Na+ current, TTX-resistant Na+ current, and Ca2+ current in the action potentials of nociceptive sensory neurons. | Q34160850 |
| | A-803467, a potent and selective Nav1.8 sodium channel blocker, attenuates neuropathic and inflammatory pain in the rat. | Q34625901 |
| | Sensory neuron sodium channel Nav1.8 is essential for pain at low temperatures | Q34637485 |
| | muO-conotoxin MrVIB selectively blocks Nav1.8 sensory neuron specific sodium channels and chronic pain behavior without motor deficits | Q35119068 |
| | Tetrodotoxin-resistant voltage-dependent sodium channels in identified muscle afferent neurons | Q36532591 |
| | Multiple sodium channels and their roles in electrogenesis within dorsal root ganglion neurons | Q36678533 |
| | Small interfering RNA-mediated selective knockdown of Na(V)1.8 tetrodotoxin-resistant sodium channel reverses mechanical allodynia in neuropathic rats | Q39338095 |
| | Increased peripheral nerve excitability and local NaV1.8 mRNA up-regulation in painful neuropathy. | Q41899981 |
| | Redistribution of Na(V)1.8 in uninjured axons enables neuropathic pain. | Q44270199 |
| | The intravenous anesthetic propofol inhibits human L-type calcium channels by enhancing voltage-dependent inactivation. | Q48351056 |
| | Synthetic muO-conotoxin MrVIB blocks TTX-resistant sodium channel NaV1.8 and has a long-lasting analgesic activity | Q48692801 |
| | Involvement of the TTX-resistant sodium channel Nav 1.8 in inflammatory and neuropathic, but not post-operative, pain states | Q48714578 |
| | The tetrodotoxin-resistant Na+ channel Nav1.8 is essential for the expression of spontaneous activity in damaged sensory axons of mice. | Q51294477 |
| | The TTX-resistant sodium channel Nav1.8 (SNS/PN3): expression and correlation with membrane properties in rat nociceptive primary afferent neurons. | Q51295679 |
| | alpha-SNS produces the slow TTX-resistant sodium current in large cutaneous afferent DRG neurons. | Q51386667 |
| | Immunolocalization of SNS/PN3 and NaN/SNS2 sodium channels in human pain states | Q73487761 |
| | Nav1.8 expression is not restricted to nociceptors in mouse peripheral nervous system | Q84389261 |
| P433 | issue | 1 | |
| P304 | page(s) | 34-37 | |
| P577 | publication date | 2012-10-12 | |
| P1433 | published in | Channels | Q15764469 |
| P1476 | title | NaV1.8 channels are expressed in large, as well as small, diameter sensory afferent neurons | |
| P478 | volume | 7 | |