scholarly article | Q13442814 |
P2093 | author name string | Jian-Jun Wen | |
Monisha Dhiman | |||
Nisha Jain Garg | |||
Elbert B Whorton | |||
P2860 | cites work | Oxidative modification of mitochondrial respiratory complexes in response to the stress of Trypanosoma cruzi infection | Q28293406 |
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Oxygen toxicity, oxygen radicals, transition metals and disease | Q29615096 | ||
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Status of myocardial antioxidants in ischemia-reperfusion injury | Q34017481 | ||
Chemokines, inflammation and Trypanosoma cruzi infection. | Q34660986 | ||
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Phenyl-alpha-tert-butyl nitrone reverses mitochondrial decay in acute Chagas' disease. | Q35569849 | ||
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Bioluminescent assay of bacterial intracellular AMP, ADP, and ATP with the use of a coimmobilized three-enzyme reagent (adenylate kinase, pyruvate kinase, and firefly luciferase). | Q36728664 | ||
Role of ubiquinone in the mitochondrial generation of hydrogen peroxide | Q40005741 | ||
Gene expression analysis in mitochondria from chagasic mice: alterations in specific metabolic pathways | Q40564183 | ||
Hemoproteins affect H(2)O(2) removal from rat tissues | Q43581333 | ||
Profiling gene transcription reveals a deficiency of mitochondrial oxidative phosphorylation in Trypanosoma cruzi-infected murine hearts: implications in chagasic myocarditis development | Q43893923 | ||
Effect of glutathione depletion on sites and topology of superoxide and hydrogen peroxide production in mitochondria. | Q44628561 | ||
Oxidative damage during chagasic cardiomyopathy development: role of mitochondrial oxidant release and inefficient antioxidant defense | Q45140382 | ||
Relationship between oxidative stress and systolic dysfunction in patients with hypertrophic cardiomyopathy | Q45283111 | ||
Impaired mitochondrial respiratory chain and bioenergetics during chagasic cardiomyopathy development | Q47345530 | ||
Tissue protection against oxidative stress. | Q52299052 | ||
Microarray analysis of changes in gene expression in a murine model of chronic chagasic cardiomyopathy. | Q53652238 | ||
Heart FoF1-ATPase changes during the acute phase of Trypanosoma cruzi infection in rats | Q58843255 | ||
Increased oxidative stress is correlated with mitochondrial dysfunction in chagasic patients | Q58845248 | ||
Relationship between Trypanosoma cruzi and human chagasic megaesophagus: blood and tissue parasitism | Q58846357 | ||
Chagas' heart disease | Q58846630 | ||
Oxidizing intermediates in the reaction of ferrous EDTA with hydrogen peroxide. Reactions with organic molecules and ferrocytochrome c | Q70137905 | ||
Free hydroxyl radicals are formed on reaction between the neutrophil-derived species superoxide anion and hypochlorous acid | Q72565213 | ||
Mitochondrial oxidative stress in heart failure: "oxygen wastage" revisited | Q73427273 | ||
P433 | issue | 10-11 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | Trypanosoma cruzi | Q150162 |
mitochondrion | Q39572 | ||
P5008 | on focus list of Wikimedia project | ScienceSource | Q55439927 |
P304 | page(s) | 1201-1209 | |
P577 | publication date | 2008-07-16 | |
P1433 | published in | Microbes and Infection | Q15760242 |
P1476 | title | Tissue-specific oxidative imbalance and mitochondrial dysfunction during Trypanosoma cruzi infection in mice | |
P478 | volume | 10 |
Q58648633 | Q58648633 |
Q39602017 | Antagonistic effect of atorvastatin on high fat diet induced survival during acute Chagas disease |
Q89481213 | Benefits of Ascorbic Acid in Association with Low-Dose Benznidazole in Treatment of Chagas Disease |
Q34124960 | Cardiac-oxidized antigens are targets of immune recognition by antibodies and potential molecular determinants in chagas disease pathogenesis |
Q33773325 | Carvedilol enhances the antioxidant effect of vitamins E and C in chronic Chagas heart disease |
Q35745413 | Chagas heart disease: report on recent developments |
Q35937562 | Changes in Proteome Profile of Peripheral Blood Mononuclear Cells in Chronic Chagas Disease |
Q37821802 | Chronic Chagas' heart disease: a disease on its way to becoming a worldwide health problem: epidemiology, etiopathology, treatment, pathogenesis and laboratory medicine |
Q36429150 | Delivery of antigenic candidates by a DNA/MVA heterologous approach elicits effector CD8(+)T cell mediated immunity against Trypanosoma cruzi |
Q36737035 | Developments in the management of Chagas cardiomyopathy |
Q50533595 | Discovery of Candidate Biomarkers. |
Q38986531 | Early Trypanosoma cruzi infection reprograms human epithelial cells. |
Q40429340 | Gene Expression Profiling and Functional Characterization of Macrophages in Response to Circulatory Microparticles Produced during Trypanosoma cruzi Infection and Chagas Disease. |
Q35056325 | Generation and characterization of a novel kidney-specific manganese superoxide dismutase knockout mouse |
Q84980450 | High levels of oxidative stress in rats infected with Blastocystis hominis |
Q90077254 | Immunity and vaccine development efforts against Trypanosoma cruzi |
Q37190915 | Increased myeloperoxidase activity and protein nitration are indicators of inflammation in patients with Chagas' disease |
Q40354504 | Inhibition of NFE2L2-Antioxidant Response Element Pathway by Mitochondrial Reactive Oxygen Species Contributes to Development of Cardiomyopathy and Left Ventricular Dysfunction in Chagas Disease. |
Q34952422 | Innate immune responses and antioxidant/oxidant imbalance are major determinants of human Chagas disease |
Q35218544 | Markers of oxidative stress in adipose tissue during Trypanosoma cruzi infection |
Q33649904 | Mitochondrial complex III defects contribute to inefficient respiration and ATP synthesis in the myocardium of Trypanosoma cruzi-infected mice |
Q41360365 | Mitochondrial dysfunction in skeletal muscle during experimental Chagas disease |
Q37328752 | MnSODtg mice control myocardial inflammatory and oxidative stress and remodeling responses elicited in chronic Chagas disease |
Q47120125 | Modulation of host central carbon metabolism and in situ glucose uptake by intracellular Trypanosoma cruzi amastigotes. |
Q35230292 | NADPH oxidase inhibition ameliorates Trypanosoma cruzi-induced myocarditis during Chagas disease |
Q55416701 | PARP1 depletion improves mitochondrial and heart function in Chagas disease: Effects on POLG dependent mtDNA maintenance. |
Q88988590 | Pathogenesis of Chronic Chagas Disease: Macrophages, Mitochondria, and Oxidative Stress |
Q57789637 | Pathology and Pathogenesis of Chagas Heart Disease |
Q37467729 | Perspectives on Trypanosoma cruzi-induced heart disease (Chagas disease). |
Q33921202 | Phenyl-alpha-tert-butyl-nitrone and benzonidazole treatment controlled the mitochondrial oxidative stress and evolution of cardiomyopathy in chronic chagasic Rats |
Q92063996 | Potential Role of Antioxidants as Adjunctive Therapy in Chagas Disease |
Q34112124 | Proteome expression and carbonylation changes during Trypanosoma cruzi infection and Chagas disease in rats. |
Q55286677 | ROS and Trypanosoma cruzi: Fuel to infection, poison to the heart. |
Q92542179 | Redox Balance Keepers and Possible Cell Functions Managed by Redox Homeostasis in Trypanosoma cruzi |
Q37223016 | S-Nitrosylation Proteome Profile of Peripheral Blood Mononuclear Cells in Human Heart Failure |
Q36169626 | SIRT1-PGC1α-NFκB Pathway of Oxidative and Inflammatory Stress during Trypanosoma cruzi Infection: Benefits of SIRT1-Targeted Therapy in Improving Heart Function in Chagas Disease |
Q34250328 | Serum proteomic signature of human chagasic patients for the identification of novel potential protein biomarkers of disease. |
Q37713220 | Serum-mediated activation of macrophages reflects TcVac2 vaccine efficacy against Chagas disease |
Q35663668 | Therapeutic Efficacy of a Subunit Vaccine in Controlling Chronic Trypanosoma cruzi Infection and Chagas Disease Is Enhanced by Glutathione Peroxidase Over-Expression |
Q64972901 | Trypanosoma cruzi Infection Induces Cellular Stress Response and Senescence-Like Phenotype in Murine Fibroblasts. |
Q37086096 | Trypanosoma cruzi antioxidant enzymes as virulence factors in Chagas disease |
Q37399631 | Trypanosoma cruzi infection disturbs mitochondrial membrane potential and ROS production rate in cardiomyocytes. |
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