Inhibition of iPLA2 β and of stretch-activated channels by doxorubicin alters dystrophic muscle function.

scientific article published on August 2013

Inhibition of iPLA2 β and of stretch-activated channels by doxorubicin alters dystrophic muscle function. is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1111/BPH.12188
P932PMC publication ID3724110
P698PubMed publication ID23849042
P5875ResearchGate publication ID249317945

P50authorHesham M IsmailQ59533115
Urs RueggQ42668553
P2093author name stringR Perozzo
L Scapozza
O M Dorchies
M K Strosova
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Calcium influx through calcium leak channels is responsible for the elevated levels of calcium-dependent proteolysis in dystrophic myotubesQ41726707
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Clinical concentrations of doxorubicin inhibit activity of myocardial membrane-associated, calcium-independent phospholipase A(2).Q43612280
P433issue7
P407language of work or nameEnglishQ1860
P921main subjectdoxorubicinQ18936
P304page(s)1537-1550
P577publication date2013-08-01
P1433published inBritish Journal of PharmacologyQ919631
P1476titleInhibition of iPLA2 β and of stretch-activated channels by doxorubicin alters dystrophic muscle function
P478volume169

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cites work (P2860)
Q92027009A Narrative Review of Cancer-Related Fatigue (CRF) and Its Possible Pathogenesis
Q34444163Diapocynin, a dimer of the NADPH oxidase inhibitor apocynin, reduces ROS production and prevents force loss in eccentrically contracting dystrophic muscle
Q38257886Dysregulation of calcium homeostasis in muscular dystrophies.
Q60950279Low Sucrose, Omega-3 Enriched Diet Has Region-Specific Effects on Neuroinflammation and Synaptic Function Markers in a Mouse Model of Doxorubicin-Based Chemotherapy
Q38831900Mitochondria: Inadvertent targets in chemotherapy-induced skeletal muscle toxicity and wasting?

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