scholarly article | Q13442814 |
P50 | author | Jerome I Rotter | Q59813011 |
Nicholette D Palmer | Q88464225 | ||
Kent D Taylor | Q89467237 | ||
Donald W Bowden | Q92206005 | ||
Carl D. Langefeld | Q102208371 | ||
Michael Bryer-Ash | Q114385367 | ||
P2860 | cites work | Genetic epidemiology of insulin resistance and visceral adiposity. The IRAS Family Study design and methods | Q44396015 |
Minimal model-based insulin sensitivity has greater heritability and a different genetic basis than homeostasis model assessment or fasting insulin | Q44433950 | ||
Haplotype structure and genotype-phenotype correlations of the sulfonylurea receptor and the islet ATP-sensitive potassium channel gene region | Q44865544 | ||
Permanent neonatal diabetes due to mutations in KCNJ11 encoding Kir6.2: patient characteristics and initial response to sulfonylurea therapy | Q45077978 | ||
Candidate gene association analysis for a quantitative trait, using parent-offspring trios. | Q45935639 | ||
Common variants in the ATP-sensitive K+ channel genes KCNJ11 (Kir6.2) and ABCC8 (SUR1) in relation to glucose intolerance: population-based studies and meta-analyses | Q46447198 | ||
Kir6.2 mutations are a common cause of permanent neonatal diabetes in a large cohort of French patients | Q47306165 | ||
MINMOD: a computer program to calculate insulin sensitivity and pancreatic responsivity from the frequently sampled intravenous glucose tolerance test. | Q54421540 | ||
Quantitative traits associated with the Type 2 diabetes susceptibility allele in Kir6.2 | Q57112357 | ||
Reconstitution of IKATP: an inward rectifier subunit plus the sulfonylurea receptor | Q24304448 | ||
Activating mutations in the gene encoding the ATP-sensitive potassium-channel subunit Kir6.2 and permanent neonatal diabetes | Q24338339 | ||
PedCheck: a program for identification of genotype incompatibilities in linkage analysis | Q24539016 | ||
Activating mutations in the ABCC8 gene in neonatal diabetes mellitus | Q27863930 | ||
Molecular biology of adenosine triphosphate-sensitive potassium channels | Q28141603 | ||
Adenosine 5'-triphosphate-sensitive potassium channels | Q28307662 | ||
Multipoint quantitative-trait linkage analysis in general pedigrees | Q29614967 | ||
Accurate assessment of beta-cell function: the hyperbolic correction | Q34508795 | ||
The evolution of beta-cell dysfunction and insulin resistance in type 2 diabetes | Q34655580 | ||
Neonatal and very-early-onset diabetes mellitus | Q35687894 | ||
Roles of KATP channels as metabolic sensors in acute metabolic changes | Q36135755 | ||
Abnormalities of pancreatic islets by targeted expression of a dominant-negative KATP channel | Q36632145 | ||
Assessment of insulin sensitivity in vivo | Q39816379 | ||
Promiscuous coupling between the sulphonylurea receptor and inwardly rectifying potassium channels | Q41229259 | ||
Quantitative estimation of insulin sensitivity | Q41600825 | ||
Amino acid polymorphisms in the ATP-regulatable inward rectifier Kir6.2 and their relationships to glucose- and tolbutamide-induced insulin secretion, the insulin sensitivity index, and NIDDM. | Q42551184 | ||
Reduced sample number for calculation of insulin sensitivity and glucose effectiveness from the minimal model. Suitability for use in population studies | Q43632818 | ||
K(IR)6.2 polymorphism predisposes to type 2 diabetes by inducing overactivity of pancreatic beta-cell ATP-sensitive K(+) channels | Q43901662 | ||
Variations in insulin secretion in carriers of the E23K variant in the KIR6.2 subunit of the ATP-sensitive K(+) channel in the beta-cell | Q44155606 | ||
The E23K variant of Kir6.2 associates with impaired post-OGTT serum insulin response and increased risk of type 2 diabetes | Q44286437 | ||
P433 | issue | 12 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | preproinsulin | Q7240673 |
P304 | page(s) | 4979-4983 | |
P577 | publication date | 2008-09-16 | |
P1433 | published in | The Journal of Clinical Endocrinology and Metabolism | Q3186902 |
P1476 | title | Association of the Kir6.2 E23K variant with reduced acute insulin response in African-Americans | |
P478 | volume | 93 |
Q28649579 | Genetic variations in magnesium-related ion channels may affect diabetes risk among African American and Hispanic American women |
Q37267062 | Kir6.2 variant E23K increases ATP-sensitive K+ channel activity and is associated with impaired insulin release and enhanced insulin sensitivity in adults with normal glucose tolerance |
Q33604496 | TCF7L2 variant rs7903146 affects the risk of type 2 diabetes by modulating incretin action. |
Q39783056 | The ATP-sensitive K(+) channel ABCC8 S1369A type 2 diabetes risk variant increases MgATPase activity |
Q37942030 | The molecular genetics of sulfonylurea receptors in the pathogenesis and treatment of insulin secretory disorders and type 2 diabetes |
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