| review article | Q7318358 |
| scholarly article | Q13442814 |
| P356 | DOI | 10.1016/S0065-2660(07)00417-8 |
| P698 | PubMed publication ID | 18358330 |
| P50 | author | David G. Cox | Q73023435 |
| P2093 | author name string | Peter Kraft | |
| P2860 | cites work | The Correlation between Relatives on the Supposition of Mendelian Inheritance | Q7727667 |
| Initial sequencing and analysis of the human genome | Q21045365 | ||
| Why most published research findings are false | Q21092395 | ||
| Confounding from cryptic relatedness in case-control association studies | Q21092505 | ||
| Population structure and eigenanalysis | Q21145248 | ||
| The association of a SNP upstream of INSIG2 with body mass index is reproduced in several but not all cohorts | Q21563458 | ||
| Finishing the euchromatic sequence of the human genome | Q22122488 | ||
| Are rare variants responsible for susceptibility to complex diseases? | Q22337172 | ||
| The effects of human population structure on large genetic association studies | Q22337233 | ||
| Assessing the impact of population stratification on genetic association studies | Q22337235 | ||
| How many diseases does it take to map a gene with SNPs? | Q22337263 | ||
| Genetic structure, self-identified race/ethnicity, and confounding in case-control association studies | Q24530649 | ||
| High-resolution whole-genome association study of Parkinson disease. | Q24535861 | ||
| Genome-wide association study of 14,000 cases of seven common diseases and 3,000 shared controls | Q24550675 | ||
| Complement factor H polymorphism in age-related macular degeneration | Q24553334 | ||
| Genome-wide association study identifies novel breast cancer susceptibility loci | Q24645441 | ||
| A common allele on chromosome 9 associated with coronary heart disease | Q24647989 | ||
| A common variant in the FTO gene is associated with body mass index and predisposes to childhood and adult obesity | Q24650037 | ||
| Genome-wide association study identifies new susceptibility loci for Crohn disease and implicates autophagy in disease pathogenesis | Q24656576 | ||
| Genomewide association analysis of coronary artery disease | Q24658344 | ||
| A haplotype map of the human genome | Q24679827 | ||
| The structure of haplotype blocks in the human genome | Q27860500 | ||
| Principal components analysis corrects for stratification in genome-wide association studies | Q27860975 | ||
| A map of human genome sequence variation containing 1.42 million single nucleotide polymorphisms | Q28203288 | ||
| Efficiency and power in genetic association studies | Q28278645 | ||
| A genome-wide association study identifies novel risk loci for type 2 diabetes | Q28287727 | ||
| A genome-wide association study identifies alleles in FGFR2 associated with risk of sporadic postmenopausal breast cancer | Q28303909 | ||
| Novel Crohn disease locus identified by genome-wide association maps to a gene desert on 5p13.1 and modulates expression of PTGER4 | Q28469186 | ||
| Replication of genome-wide association signals in UK samples reveals risk loci for type 2 diabetes | Q28680760 | ||
| A new multipoint method for genome-wide association studies by imputation of genotypes | Q29547209 | ||
| A genome-wide association study of type 2 diabetes in Finns detects multiple susceptibility variants | Q29547210 | ||
| Evaluating coverage of genome-wide association studies | Q46423138 | ||
| Genome-wide tagging for everyone | Q46657606 | ||
| A common genetic variant is associated with adult and childhood obesity | Q46726890 | ||
| Agnosticism and equity in genome-wide association studies | Q46776377 | ||
| Two-stage designs for gene-disease association studies with sample size constraints | Q47408883 | ||
| An efficient Monte Carlo approach to assessing statistical significance in genomic studies. | Q48521093 | ||
| An utter refutation of the "Fundamental Theorem of the HapMap" by Terwilliger and Hiekkalinna. | Q51936239 | ||
| An utter refutation of the "fundamental theorem of the HapMap". | Q51950798 | ||
| Betting odds and genetic associations. | Q52001328 | ||
| Case/pseudocontrol analysis in genetic association studies: A unified framework for detection of genotype and haplotype associations, gene-gene and gene-environment interactions, and parent-of-origin effects. | Q52001434 | ||
| The efficiency of matching in case-control studies of risk-factor interactions. | Q52680409 | ||
| Evaluating and improving power in whole-genome association studies using fixed marker sets. | Q53255191 | ||
| Genome-wide strategies for detecting multiple loci that influence complex diseases. | Q53624440 | ||
| Comment on "A Common Genetic Variant Is Associated with Adult and Childhood Obesity" | Q57249755 | ||
| A candidate gene approach to searching for low-penetrance breast and prostate cancer genes | Q57306192 | ||
| Estimation of Regression Coefficients When Some Regressors are not Always Observed | Q57310639 | ||
| Demonstrating stratification in a European American population | Q57418313 | ||
| Genetic and Environmental Contributions to Atherosclerosis Phenotypes in Men and Women | Q57745943 | ||
| Evidence for Heritability of Abdominal Aortic Calcific Deposits in the Framingham Heart Study | Q57798894 | ||
| Two-Stage Designs for Gene-Disease Association Studies | Q57908316 | ||
| Improving power in genome-wide association studies: weights tip the scale | Q58047087 | ||
| False discovery control with p-value weighting | Q58047091 | ||
| On estimating HLA/disease association with application to a study of aplastic anemia | Q70187094 | ||
| Linkage disequilibrium mapping of complex disease: fantasy or reality? | Q77796076 | ||
| Prospects for whole-genome linkage disequilibrium mapping of common disease genes | Q77883184 | ||
| Population genetics--making sense out of sequence | Q77895910 | ||
| Polygenic susceptibility to breast cancer and implications for prevention | Q78029012 | ||
| A unified approach to the analysis of case-distribution (case-only) studies | Q78174609 | ||
| Polygenic inheritance of breast cancer: Implications for design of association studies | Q79162761 | ||
| Transferability of tag SNPs in genetic association studies in multiple populations | Q79290429 | ||
| Comment on "A common genetic variant is associated with adult and childhood obesity" | Q79546092 | ||
| Merging and emerging cohorts: necessary but not sufficient | Q79579692 | ||
| Exploiting gene-environment interaction to detect genetic associations | Q79731627 | ||
| Multiple prostate cancer risk variants on 8q24 | Q80220532 | ||
| Identifying interacting SNPs using Monte Carlo logic regression | Q80975928 | ||
| In genetic control of disease, does 'race' matter? | Q81060319 | ||
| The need for a systematic approach to complex pathways in molecular epidemiology | Q81521786 | ||
| Aspects of the design and analysis of high-dimensional SNP studies for disease risk estimation | Q82411536 | ||
| Are we ready for genome-wide association studies? | Q83153878 | ||
| Re: Commonly studied single-nucleotide polymorphisms and breast cancer: results from the Breast Cancer Association Consortium | Q94519131 | ||
| Genomic control for association studies | Q29547213 | ||
| Genome-wide association analysis identifies loci for type 2 diabetes and triglyceride levels | Q29547214 | ||
| The future of genetic studies of complex human diseases | Q29547215 | ||
| A genome-wide association scan of nonsynonymous SNPs identifies a susceptibility variant for Crohn disease in ATG16L1 | Q29614873 | ||
| Replicating genotype-phenotype associations | Q29614919 | ||
| Genome-wide association study of prostate cancer identifies a second risk locus at 8q24 | Q29614925 | ||
| Joint analysis is more efficient than replication-based analysis for two-stage genome-wide association studies | Q29614926 | ||
| Population structure, differential bias and genomic control in a large-scale, case-control association study | Q29614945 | ||
| Assessing the probability that a positive report is false: an approach for molecular epidemiology studies | Q29616285 | ||
| On the allelic spectrum of human disease | Q29616298 | ||
| Linkage disequilibrium in humans: models and data | Q30654298 | ||
| Efficient computation of significance levels for multiple associations in large studies of correlated data, including genomewide association studies | Q30946038 | ||
| Genomic screening and replication using the same data set in family-based association testing | Q30991447 | ||
| Two-stage two-locus models in genome-wide association | Q33258470 | ||
| Generalized analysis of molecular variance | Q33281291 | ||
| Association mapping in structured populations | Q33903172 | ||
| Undetected genotyping errors cause apparent overtransmission of common alleles in the transmission/disequilibrium test | Q33904499 | ||
| Cost-effective designs for linkage disequilibrium mapping of complex traits | Q33904708 | ||
| A simple correction for multiple testing for single-nucleotide polymorphisms in linkage disequilibrium with each other | Q33909759 | ||
| Increasing the power and efficiency of disease-marker case-control association studies through use of allele-sharing information | Q33910988 | ||
| Cohort studies for characterizing measured genes. | Q33943341 | ||
| Multifactor-dimensionality reduction reveals high-order interactions among estrogen-metabolism genes in sporadic breast cancer | Q33951229 | ||
| Recent developments in genomewide association scans: a workshop summary and review | Q34021104 | ||
| Accounting for unmeasured population substructure in case-control studies of genetic association using a novel latent-class model | Q34044226 | ||
| Detecting association in a case-control study while correcting for population stratification | Q34105619 | ||
| The genetics and genomics of cancer | Q34180336 | ||
| Additional SNPs and linkage-disequilibrium analyses are necessary for whole-genome association studies in humans | Q34185525 | ||
| Using linkage genome scans to improve power of association in genome scans | Q34398834 | ||
| Evaluating statistical significance in two-stage genomewide association studies | Q34399069 | ||
| Epidemiological methods for studying genes and environmental factors in complex diseases | Q34421625 | ||
| A common genetic variant in the NOS1 regulator NOS1AP modulates cardiac repolarization | Q34519498 | ||
| Genome-wide association studies: theoretical and practical concerns | Q34555195 | ||
| Common variants on chromosomes 2q35 and 16q12 confer susceptibility to estrogen receptor-positive breast cancer | Q34631785 | ||
| Coverage and power in genomewide association studies | Q34658106 | ||
| DNA Pooling: a tool for large-scale association studies | Q34988384 | ||
| No gene is an island: the flip-flop phenomenon | Q35677798 | ||
| A simple and improved correction for population stratification in case-control studies | Q35752599 | ||
| Genetic association studies of complex traits: design and analysis issues | Q36097373 | ||
| Factors affecting statistical power in the detection of genetic association | Q36148601 | ||
| Genetic association studies | Q36267276 | ||
| Systematic review and meta-analysis of the association between complement factor H Y402H polymorphisms and age-related macular degeneration | Q36564570 | ||
| Scanning the horizon: what is the future of genome-wide association studies in accelerating discoveries in cancer etiology and prevention? | Q36794423 | ||
| Comment on "A common genetic variant is associated with adult and childhood obesity" | Q37284957 | ||
| Future directions of research in statistical genetics | Q40734399 | ||
| The problem of multiple inference in studies designed to generate hypotheses | Q41506019 | ||
| Multistage sampling for disease family registries. | Q41741609 | ||
| PBAT: a comprehensive software package for genome-wide association analysis of complex family-based studies. | Q42064007 | ||
| Genomewide association, Parkinson disease, and PARK10 | Q42418561 | ||
| Conflicting results regarding the semaphorin gene (SEMA5A) and the risk for Parkinson disease | Q42418588 | ||
| No evidence for association with Parkinson disease for 13 single-nucleotide polymorphisms identified by whole-genome association screening. | Q42418595 | ||
| A case-control association study of the 12 single-nucleotide polymorphisms implicated in Parkinson disease by a recent genome scan | Q42418600 | ||
| Considerations for genomewide association studies in Parkinson disease | Q42418603 | ||
| A note on permutation tests in multistage association scans. | Q42418628 | ||
| A worldwide survey of haplotype variation and linkage disequilibrium in the human genome | Q42601487 | ||
| Logistic regression protects against population structure in genetic association studies | Q43198448 | ||
| Asymptotic bias and efficiency in case-control studies of candidate genes and gene-environment interactions: basic family designs | Q43765471 | ||
| Selection of controls in case-control studies. II. Types of controls | Q43881538 | ||
| 'Racial' differences in genetic effects for complex diseases | Q44586727 | ||
| A framework for controlling false discovery rates and minimizing the amount of genotyping in the search for disease mutations | Q44901310 | ||
| Optimal two-stage genotyping in population-based association studies | Q44968950 | ||
| Endogenous estrogen, androgen, and progesterone concentrations and breast cancer risk among postmenopausal women | Q45188730 | ||
| Commentary: the concept of 'Mendelian Randomization'. | Q46041775 | ||
| The relative power of family-based and case-control designs for linkage disequilibrium studies of complex human diseases I. DNA pooling | Q46190392 | ||
| Optimal two-stage genotyping designs for genome-wide association scans | Q46304991 | ||
| Genome-wide association study identifies a second prostate cancer susceptibility variant at 8q24. | Q46332214 | ||
| P921 | main subject | genome-wide association study | Q1098876 |
| P304 | page(s) | 465-504 | |
| P577 | publication date | 2008-01-01 | |
| P1433 | published in | Advances in Genetics | Q26842228 |
| P1476 | title | Study designs for genome-wide association studies | |
| P478 | volume | 60 |
| Q34327364 | A critical assessment of the factors affecting reporter gene assays for promoter SNP function: a reassessment of -308 TNF polymorphism function using a novel integrated reporter system |
| Q47954386 | An Enrichment Strategy Yields Seven Novel Single Nucleotide Polymorphisms Associated with Mortality and Altered TH17 Responses Following Blunt Trauma |
| Q33647983 | Bioinformatics challenges for genome-wide association studies |
| Q30234489 | Data-Based Radiation Oncology: Design of Clinical Trials in the Toxicity Biomarkers Era. |
| Q29109926 | Design and analysis issues in gene and environment studies |
| Q38404777 | Ethical aspects of participation in the database of genotypes and phenotypes of the National Center for Biotechnology Information: the Cancer and Leukemia Group B Experience |
| Q29417137 | GWAS of cerebrospinal fluid tau levels identifies risk variants for Alzheimer's disease |
| Q33887427 | Genetic susceptibility to childhood leukaemia |
| Q37390141 | Genetics and sports |
| Q42035322 | Methodological Issues in Multistage Genome-wide Association Studies |
| Q33761835 | Methods for investigating gene-environment interactions in candidate pathway and genome-wide association studies |
| Q37594192 | Multiple linear combination (MLC) regression tests for common variants adapted to linkage disequilibrium structure |
| Q37651413 | Next generation DNA sequencing and the future of genomic medicine |
| Q37654985 | One thousand genomes imputation in the National Cancer Institute Breast and Prostate Cancer Cohort Consortium aggressive prostate cancer genome-wide association study |
| Q30240052 | Optimal design and patient selection for interventional trials using radiogenomic biomarkers: A REQUITE and Radiogenomics consortium statement |
| Q34544433 | Powerful SNP-set analysis for case-control genome-wide association studies |
| Q37319384 | Predicting functional regulatory polymorphisms |
| Q24288830 | Replication in Genome-Wide Association Studies |
| Q35259510 | SNP characteristics predict replication success in association studies |
| Q35805225 | Testing gene-gene interactions in genome wide association studies |
| Q36203536 | The success of pharmacogenomics in moving genetic association studies from bench to bedside: study design and implementation of precision medicine in the post-GWAS era. |
| Q50300835 | [Leukocyte count of puerperal sows]. |
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