| scholarly article | Q13442814 |
| P2093 | author name string | Lin Xu | |
| Kai Ding | |||
| Feng Wang | |||
| Xiao Shen | |||
| Kun Zhao | |||
| Ren-Min Liu | |||
| Xin-Xin Fan | |||
| P2860 | cites work | Early coagulopathy in trauma patients: an on-scene and hospital admission study. | Q46912713 |
| Endothelial glycocalyx degradation induces endogenous heparinization in patients with severe injury and early traumatic coagulopathy | Q47441825 | ||
| Anti-high mobility group box 1 monoclonal antibody ameliorates brain infarction induced by transient ischemia in rats | Q48106802 | ||
| Definition and drivers of acute traumatic coagulopathy: clinical and experimental investigations. | Q50574747 | ||
| Potential biomarker panel for predicting organ dysfunction and acute coagulopathy in a polytrauma porcine model. | Q54313187 | ||
| HMGB-1 promotes fibrinolysis and reduces neurotoxicity mediated by tissue plasminogen activator | Q60597641 | ||
| Persistent elevation of high mobility group box-1 protein (HMGB1) in patients with severe sepsis and septic shock* | Q60692890 | ||
| A High Admission Syndecan-1 Level, A Marker of Endothelial Glycocalyx Degradation, Is Associated With Inflammation, Protein C Depletion, Fibrinolysis, and Increased Mortality in Trauma Patients | Q61783747 | ||
| Activation of gene expression in human neutrophils by high mobility group box 1 protein | Q73087842 | ||
| High-mobility group box 1 protein promotes development of microvascular thrombosis in rats | Q79613873 | ||
| High mobility group B1 levels in sepsis and Disseminated Intravascular Coagulation | Q85261033 | ||
| Projections of global mortality and burden of disease from 2002 to 2030 | Q21144688 | ||
| HMG-1 as a late mediator of endotoxin lethality in mice | Q28139166 | ||
| Early release of high mobility group box nuclear protein 1 after severe trauma in humans: role of injury severity and tissue hypoperfusion | Q33612700 | ||
| Molecular mechanism and therapeutic modulation of high mobility group box 1 release and action: an updated review | Q33751106 | ||
| Early release of high-mobility group box 1 (HMGB1) from neurons in experimental subarachnoid hemorrhage in vivo and in vitro | Q33936977 | ||
| Inflammatory response to trauma: implications for coagulation and resuscitation | Q34014499 | ||
| Anti-HMGB1 neutralizing antibody ameliorates gut barrier dysfunction and improves survival after hemorrhagic shock | Q35052743 | ||
| Increased HMGB1 expression and release by mononuclear cells following surgical/anesthesia trauma | Q35561332 | ||
| Functional definition and characterization of acute traumatic coagulopathy | Q35571219 | ||
| A cell-based model of coagulation and the role of factor VIIa | Q35617165 | ||
| Early platelet dysfunction: an unrecognized role in the acute coagulopathy of trauma. | Q35948091 | ||
| Cells migrating to sites of tissue damage in response to the danger signal HMGB1 require NF-kappaB activation | Q36119506 | ||
| Alarmins: chemotactic activators of immune responses | Q36162859 | ||
| Early release of soluble receptor for advanced glycation endproducts after severe trauma in humans. | Q36493417 | ||
| Increase in activated protein C mediates acute traumatic coagulopathy in mice | Q36615864 | ||
| HMGB1 induces human lung endothelial cell cytoskeletal rearrangement and barrier disruption | Q36919024 | ||
| Clinical and mechanistic drivers of acute traumatic coagulopathy | Q37127772 | ||
| Early platelet dysfunction in a rodent model of blunt traumatic brain injury reflects the acute traumatic coagulopathy found in humans | Q37580271 | ||
| Trauma alarmins as activators of damage-induced inflammation | Q37996337 | ||
| A standardized technique for performing thromboelastography in rodents. | Q38829684 | ||
| High dose antithrombin III inhibits HMGB1 and improves endotoxin-induced acute lung injury in rats | Q40067011 | ||
| High-mobility group box 1 protein (HMGB1) neutralization ameliorates experimental autoimmune encephalomyelitis | Q41511974 | ||
| Blockade of high-mobility group box-1 ameliorates acute on chronic liver failure in rats. | Q42266454 | ||
| Increased plasma and platelet to red blood cell ratios improves outcome in 466 massively transfused civilian trauma patients | Q43961306 | ||
| Primary fibrinolysis is integral in the pathogenesis of the acute coagulopathy of trauma | Q44405049 | ||
| The incidence and magnitude of fibrinolytic activation in trauma patients | Q44658013 | ||
| HMGB1 increases permeability of the endothelial cell monolayer via RAGE and Src family tyrosine kinase pathways | Q45347650 | ||
| Acute traumatic coagulopathy: initiated by hypoperfusion: modulated through the protein C pathway? | Q46166562 | ||
| High-mobility group box-1 and its receptors contribute to proinflammatory response in the acute phase of spinal cord injury in rats | Q46339809 | ||
| P921 | main subject | systemic inflammation | Q7663815 |
| P1104 | number of pages | 10 | |
| P304 | page(s) | 2561-2570 | |
| P577 | publication date | 2016-07-20 | |
| P1433 | published in | Medical Science Monitor | Q15756323 |
| P1476 | title | Blockade of Extracellular High-Mobility Group Box 1 Attenuates Systemic Inflammation and Coagulation Abnormalities in Rats with Acute Traumatic Coagulopathy | |
| P478 | volume | 22 |
| Q39427034 | Ischemic preconditioning modulates ROS to confer protection in liver ischemia and reperfusion. |
| Q33589533 | Joint Assessment of Donor and Recipient hTERT Gene Polymorphism Provides Additional Information for Early Kidney Transplantation Outcomes. |
| Q47791665 | Mitochondrial dysfunction in rat splenocytes following hemorrhagic shock |
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