Macrophages, reactive nitrogen species, and lung injury

scientific article published on August 2010

Macrophages, reactive nitrogen species, and lung injury is …
instance of (P31):
review articleQ7318358
scholarly articleQ13442814

External links are
P356DOI10.1111/J.1749-6632.2010.05607.X
P932PMC publication ID3797654
P698PubMed publication ID20716284

P50authorAndrew J. GowQ30511946
P2093author name stringJeffrey D Laskin
Debra L Laskin
Vasanthi R Sunil
Ladan Fakhrzadeh
Angela Groves
P2860cites workSoluble metals in residual oil fly ash alter innate and adaptive pulmonary immune responses to bacterial infection in ratsQ23914723
Molecular genetics of the caveolin gene family: implications for human cancers, diabetes, Alzheimer disease, and muscular dystrophyQ24539692
Acute ozone-induced differential gene expression profiles in rat lungQ24811714
Caveolae compartmentalization of heme oxygenase-1 in endothelial cellsQ28575667
Macrophage activation and polarizationQ29616353
S-nitrosylation of surfactant protein-D controls inflammatory functionQ33384274
The phagocytes: neutrophils and monocytesQ34013216
Role of macrophages and inflammatory mediators in chemically induced toxicityQ34179549
The Role of TNF and Its Family Members in Inflammation and Cancer: Lessons from Gene DeletionQ35560573
The biology of caveolae: lessons from caveolin knockout mice and implications for human diseaseQ35679599
Basic pathogenetic mechanisms in silicosis: current understandingQ36034136
Reactive nitrogen species in the respiratory tractQ36389957
Regulation of caveolin-1 expression, nitric oxide production and tissue injury by tumor necrosis factor-alpha following ozone inhalationQ36426283
Macrophage activation by endogenous danger signalsQ37045743
Ozone exposure and mortality: an empiric bayes metaregression analysisQ39720089
Inhibition of macrophages with gadolinium chloride abrogates ozone-induced pulmonary injury and inflammatory mediator productionQ41666401
Caveolins, a family of scaffolding proteins for organizing "preassembled signaling complexes" at the plasma membraneQ41715384
Macrophages and inflammatory mediators in chemical toxicity: a battle of forcesQ42134547
Enhanced production of interleukin-1, tumor necrosis factor-alpha, and fibronectin by rat lung phagocytes following inhalation of a pulmonary irritantQ42284256
Inhibition of ozone-induced nitric oxide synthase expression in the lung by endotoxinQ42556075
Deficiency in inducible nitric oxide synthase protects mice from ozone-induced lung inflammation and tissue injuryQ43936965
Upregulation of phosphoinositide 3-kinase and protein kinase B in alveolar macrophages following ozone inhalation. Role of NF-kappaB and STAT-1 in ozone-induced nitric oxide production and toxicityQ44089720
Superoxide dismutase-overexpressing mice are resistant to ozone-induced tissue injury and increases in nitric oxide and tumor necrosis factor-alphaQ44509563
Ozone-induced production of nitric oxide and TNF-alpha and tissue injury are dependent on NF-kappaB p50.Q44830075
Differential regulation of TNF-R1 signaling: lipid raft dependency of p42mapk/erk2 activation, but not NF-kappaB activationQ44928990
Derivation and characterization of murine alternatively activated (M2) macrophagesQ46060747
Nitrosative stress in the bronchial mucosa of severe chronic obstructive pulmonary diseaseQ46794696
Macrophage activation switching: an asset for the resolution of inflammationQ46811059
In situ assessment of oxidant and nitrogenic stress in bleomycin pulmonary fibrosisQ46819511
The cyclin D1 gene is transcriptionally repressed by caveolin-1.Q52169522
P407language of work or nameEnglishQ1860
P921main subjectmacrophageQ184204
pulmonary injuryQ7259546
P304page(s)60-65
P577publication date2010-08-01
P1433published inAnnals of the New York Academy of SciencesQ2431664
P1476titleMacrophages, reactive nitrogen species, and lung injury
P478volume1203

Reverse relations

cites work (P2860)
Q50632076Acute secondhand smoke-induced pulmonary inflammation is diminished in RAGE knockout mice.
Q34396267Antenatal exposure of maternal secondhand smoke (SHS) increases fetal lung expression of RAGE and induces RAGE-mediated pulmonary inflammation.
Q39392448Anti-TNFα therapy in inflammatory lung diseases
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Q36218777Autocatalytic nitration of prostaglandin endoperoxide synthase-2 by nitrite inhibits prostanoid formation in rat alveolar macrophages
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Q26861392Oxidative stress-induced autophagy: role in pulmonary toxicity
Q42698507Ozone Inhalation Provokes Glucocorticoid-Dependent and -Independent Effects on Inflammatory and Metabolic Pathways
Q38991830Primary alveolar macrophages exposed to diesel particulate matter increase RAGE expression and activate RAGE signaling
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Q37330783Role of NOS2 in pulmonary injury and repair in response to bleomycin
Q36297772Sex differences in the expression of lung inflammatory mediators in response to ozone
Q28387372The fungal T-2 toxin alters the activation of primary macrophages induced by TLR-agonists resulting in a decrease of the inflammatory response in the pig
Q88477988Transgenic up-regulation of Claudin-6 decreases fine diesel particulate matter (DPM)-induced pulmonary inflammation

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