scholarly article | Q13442814 |
P2093 | author name string | Christopher C Silliman | |
Daniel R Ambruso | |||
Michael A Ellison | |||
P2860 | cites work | Therapeutic effects of lysophosphatidylcholine in experimental sepsis | Q28238163 |
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Lysophospholipids of different classes mobilize neutrophil secretory vesicles and induce redundant signaling through G2A | Q28300335 | ||
Retraction | Q28302699 | ||
Pharmacologic activity of bepafant (WEB 2170), a new and selective hetrazepinoic antagonist of platelet activating factor | Q28332359 | ||
RETRACTED: Lysophosphatidylcholine as a ligand for the immunoregulatory receptor G2A. | Q34085160 | ||
Mechanisms of leukocyte sequestration in inflamed lungs | Q34265953 | ||
Transfusion-related acute lung injury: a neglected, serious complication of hemotherapy | Q34306589 | ||
The association of biologically active lipids with the development of transfusion-related acute lung injury: a retrospective study | Q34432820 | ||
Tissue injury in inflammation. Oxidants, proteinases, and cationic proteins | Q34562547 | ||
Neutrophil priming: the cellular signals that say 'amber' but not 'green'. | Q34721187 | ||
Lysophosphatidylcholines activate G2A inducing G(αi)₋₁-/G(αq/)₁₁- Ca²(+) flux, G(βγ)-Hck activation and clathrin/β-arrestin-1/GRK6 recruitment in PMNs. | Q35090466 | ||
Identification of lipids that accumulate during the routine storage of prestorage leukoreduced red blood cells and cause acute lung injury. | Q35231541 | ||
Partial characterization of lipids that develop during the routine storage of blood and prime the neutrophil NADPH oxidase. | Q35673608 | ||
Hypotension and acute pulmonary insufficiency following transfusion of autologous red blood cells during surgery: a case report and review of the literature | Q35925724 | ||
Priming of neutrophils for enhanced release of oxygen metabolites by bacterial lipopolysaccharide. Evidence for increased activity of the superoxide-producing enzyme | Q36349700 | ||
The two-event model of transfusion-related acute lung injury | Q36450423 | ||
Lysophospholipid acyltransferases: novel potential regulators of the inflammatory response and target for new drug discovery. | Q37185895 | ||
Plasma and lipids from stored packed red blood cells cause acute lung injury in an animal model. | Q37380265 | ||
The role of the neutrophil in inflammatory diseases of the lung | Q37929923 | ||
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Lysophosphatidylcholine-induced surface redistribution regulates signaling of the murine G protein-coupled receptor G2A. | Q42211562 | ||
Demonstration of reversible priming of human neutrophils using platelet-activating factor | Q42527617 | ||
Transfusion-related acute lung injury: epidemiology and a prospective analysis of etiologic factors | Q44189779 | ||
Lysophosphatidylcholines prime the NADPH oxidase and stimulate multiple neutrophil functions through changes in cytosolic calcium | Q44378760 | ||
HLA class II antibodies in transfusion-related acute lung injury. | Q45957230 | ||
Implementation of a strategy to prevent TRALI in a regional blood centre | Q47686454 | ||
Neutrophil priming: pathophysiological consequences and underlying mechanisms. | Q47772753 | ||
Accumulation of bioactive lipids during storage of blood products is not cell but plasma derived and temperature dependent. | Q50536462 | ||
Generation of neutrophil priming activity by cell-containing blood components treated with pathogen reduction technology and stored in platelet additive solutions. | Q50551784 | ||
Specificities of leucocyte alloantibodies in transfusion-related acute lung injury and results of leucocyte antibody screening of blood donors. | Q50622297 | ||
Transfusion-related acute lung injury caused by human leucocyte antigen class II antibody. | Q51727141 | ||
Diagnostic and pathogenetic considerations in transfusion-related acute lung injury. | Q53770407 | ||
Control of vascular permeability by polymorphonuclear leukocytes in inflammation | Q59077548 | ||
Stored blood components contain agents that prime the neutrophil NADPH oxidase through the platelet-activating-factor receptor | Q67506750 | ||
Neutrophil responses to lipopolysaccharide. Effect of adherence on triggering and priming of the respiratory burst | Q67912766 | ||
Unsaturated fatty acids and lipoxygenase products regulate phagocytic NADPH oxidase activity by a nondetergent mechanism | Q68043195 | ||
Platelet-activating factor primes neutrophil responses to agonists: role in promoting neutrophil-mediated endothelial damage | Q69023417 | ||
Transfusion-related acute lung injury associated with passive transfer of antileukocyte antibodies | Q70989793 | ||
Analysis of the priming activity of lipids generated during routine storage of platelet concentrates | Q71042829 | ||
Diverging signal transduction pathways activated by interleukin 8 (IL-8) and related chemokines in human neutrophils. IL-8 and Gro-alpha differentially stimulate calcium influx through IL-8 receptors A and B | Q71246171 | ||
3-(2-(3-Pyridinyl)thiazolidin-4-oyl)indoles, a novel series of platelet activating factor antagonists | Q72052684 | ||
Severe pulmonary hypersensitivity associated with passive transfusion of a neutrophil-specific antibody | Q72575772 | ||
Compounds biologically similar to platelet activating factor are present in stored blood components | Q72791989 | ||
Plasma and lipids from stored platelets cause acute lung injury in an animal model | Q73288183 | ||
Transfusion-related acute lung injury | Q78057891 | ||
A two-insult in vitro model of PMN-mediated pulmonary endothelial damage: requirements for adherence and chemokine release | Q78396050 | ||
Transfusion-related acute lung injury | Q81079589 | ||
P433 | issue | 22 | |
P921 | main subject | transfusion related acute lung injury | Q1472604 |
acute lung injury | Q4677935 | ||
P304 | page(s) | 3255-3259 | |
P577 | publication date | 2012-01-01 | |
P1433 | published in | Current Pharmaceutical Design | Q5195068 |
P1476 | title | Therapeutic options for transfusion related acute lung injury; the potential of the G2A receptor | |
P478 | volume | 18 |
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